Sympathomimetics (Adrenergic Agonists)-Epinephrine, Norepinephrine - Alpha & Beta Receptors
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- Опубликовано: 29 авг 2024
- Sympathomimetics (Adrenergic Agonists)-Catecholamines (Epinephrine, Norepinephrine, Dopamine), Alpha & Beta Receptors…Alpha adrenergic receptors (alpha 1, alpha 2), beta adrenergic (beta 1, beta 2, beta 3). receptors. Sympathetic autonomic nervous system (SANS). Alpha agonists, alpha antagonists (alpha blockers), beta agonists, beta antagonists (beta blockers). Autonomic pharmacology, Asthma treatment, Respiratory pharmacology, cardiovascular pharmacology, Hypertension pharmacology.
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if your voice was like Khan or Ninja, you would've been the #1 medical resource to every nation. vocal plastic surgery is still available
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I need to watch this! Got POTS which seems to be linked to deregulation of the autonomic nervous system and one of the treatments is beta blockers
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السلام عليكم ورحمة الله وبركاته دكتور ممكن حضرتك تدينا نصائح عن المذاكره في طب خصوصا سنه أولى 😢
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Could you please create a video on the topic of medicinal chemistry? Thank you.
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In a sepsis you have distributive shock . wow It is a well done explanation. if I want to read more about sympathetic and parasympathetic system, what book would you recommend? or perhaps a paper?
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Can u please make video on pharmacognosy
I just remembered, when i was a kid, i used to get injured and one time i got hit in the head which caused bleeding and back then old folks whould manage that by compressing the wound site with guess what, wet coffee ground 😳
And i can't help it by noticing thanks to this video that caffeine actually acts as a vasodilator (so not of a significant way of stopping bleeding) but i still wonder where did they got the idea from and can it be, as opposed what i just mentioned, beneficial in some other way in treating wound bleed? Nevertheless, it keeps boggling my mind how drugs can act differently based on their dosage and route of administration (nothing in this life is straightforward, NOTHING!)
I never wanted or bothered to learn this, uworld is telling me to learn it. but I still don't want to ewwwwwwwww igeewwwwww. Thank you sir. :)
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1:00:31
if the venous return drops, while contractility increases, can we be sure CO increases? without a good preload, isnt that CO's gonna drop in accordance?
As per my knowledge, Higher Contraction gives more output . As For normal it's about 70ml Remaining in Ventricle after Ventricular Contraction , but if Contraction increases this value of Remaining 70 ml will be less in Ventricle and CO will be More.
So in This Venous return is less but the Higher contraction will compensate to give higher CO
Shock in Sepsis is distributive.
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it sounds that your idol is dr Nagi
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if your voice was like Khan or Ninja, you would've been the #1 medical resource to every nation. vocal plastic surgery is still available.