Hi, I never went to Jaeger but the dialysis centre in Bayreuth. No tests were done there but my blood looked like it was full of clots. I know that Jaeger has ways of testing but not sure how it works.
Well in addition to the you know what, it seems to be that they are created secondarily to the core illness processes. So things like post-exertional malaise and ans dysfunction upregulate clotting pathways that then cause the microclots to form.
From the best of my knowledge, it seems the micoclots do keep occurring but the main factors that drive them are crashing (PEM episodes) and the flight/fight state of the nervous system. Both seem to upregulate the clotting pathways. I learnt that from an interview of Dr. Asad Khan. Therefore, hypothetically, never crashing and brain retraining might help reduce clotting pathways. On the flip side, I had a good period of three months prior to my HELP apheresis treatment in Germany and my blood was still thick as anything. I wonder if the microclot situation is particularly bad if the body can clear them itself or not. Check out my video on the drug treatment also for the microclots as that seems to clear them over a 3-4 month period, though personally I wouldn't take anticoagulants.
Did you experience fatigue in legs specifically after your foot injury or entire body in general without legs standing out as a primary concern compared to the body?,did you see any of the typical hypervigilance symptoms becoming severe alongside the fatigue and pain? Did you felt that symptoms are at their worst when you just wake up and start to tone down a slowly as the day goes by? These are some way I'd assume to determine if one has automatic imbalance or serious secondary issue,they call it cfs with s standing for syndrome which means they don't know why it happens but it would be very ignorant to assume that there could be only one unknown cause that would incapacitate a person in such a way it would better be called chronic fatigue syndromes at this point
I follow and agree with the work of Profs Scheibenbogen and Wirth which describe ME/CFS as an illness of global hypoperfusion, or an illness in which blood is not perfusing into the body adequately. On top of that, like Wirth and Schiebenbogen, I would regard ANS dysfunction as going on and - to my mind - it is likely the root cause BUT the various secondary dysfunctions may then become so stubborn and imbedded that they might need their own treatment. Microclotting is one of the secondary dysfunctions, not the root cause.
Well, an ESR of less than 3 would suggest thick blood and microclotting pathology. There are plenty of other pathologies that contribute to POTS and ME/CFS - low blood volume, autoantibodies, mitochondrial issues, etc. Not everyone has all pathologies equally. I have a good friend with ME/CFS who has no signs of microclots at all but for me my microclots were through the roof.
You're doing some great work on this channel, thanks!
Thanks very much, I much appreciate that.
Was this sufficient basis for Jaeger to confirm you had microclots or did they do additional testing? If so, what markers were they looking at?
Hi, I never went to Jaeger but the dialysis centre in Bayreuth. No tests were done there but my blood looked like it was full of clots. I know that Jaeger has ways of testing but not sure how it works.
Do we know why these clots form (prior to the recent you know what that also causes clotting)?
Well in addition to the you know what, it seems to be that they are created secondarily to the core illness processes. So things like post-exertional malaise and ans dysfunction upregulate clotting pathways that then cause the microclots to form.
So do the microclots keep occurring? I suffer from long COVID and can’t find any details on if/when the body stops producing microclotting
From the best of my knowledge, it seems the micoclots do keep occurring but the main factors that drive them are crashing (PEM episodes) and the flight/fight state of the nervous system. Both seem to upregulate the clotting pathways. I learnt that from an interview of Dr. Asad Khan. Therefore, hypothetically, never crashing and brain retraining might help reduce clotting pathways. On the flip side, I had a good period of three months prior to my HELP apheresis treatment in Germany and my blood was still thick as anything. I wonder if the microclot situation is particularly bad if the body can clear them itself or not. Check out my video on the drug treatment also for the microclots as that seems to clear them over a 3-4 month period, though personally I wouldn't take anticoagulants.
@@me-cfs-strategiesforhealing Isn't brain retraining a scum?
Did you experience fatigue in legs specifically after your foot injury or entire body in general without legs standing out as a primary concern compared to the body?,did you see any of the typical hypervigilance symptoms becoming severe alongside the fatigue and pain? Did you felt that symptoms are at their worst when you just wake up and start to tone down a slowly as the day goes by? These are some way I'd assume to determine if one has automatic imbalance or serious secondary issue,they call it cfs with s standing for syndrome which means they don't know why it happens but it would be very ignorant to assume that there could be only one unknown cause that would incapacitate a person in such a way it would better be called chronic fatigue syndromes at this point
I follow and agree with the work of Profs Scheibenbogen and Wirth which describe ME/CFS as an illness of global hypoperfusion, or an illness in which blood is not perfusing into the body adequately. On top of that, like Wirth and Schiebenbogen, I would regard ANS dysfunction as going on and - to my mind - it is likely the root cause BUT the various secondary dysfunctions may then become so stubborn and imbedded that they might need their own treatment. Microclotting is one of the secondary dysfunctions, not the root cause.
What is your D dimer level?
Hi, never tested though my understanding is that D-Dimer won't indicate microclotting.
ESR Normal 😅 like 8
An ESR of 8 would seem to rule out microclotting.
@@me-cfs-strategiesforhealing Really? Then why do I have POTS and constant fatigue?😅😭😭.
Well, an ESR of less than 3 would suggest thick blood and microclotting pathology. There are plenty of other pathologies that contribute to POTS and ME/CFS - low blood volume, autoantibodies, mitochondrial issues, etc. Not everyone has all pathologies equally. I have a good friend with ME/CFS who has no signs of microclots at all but for me my microclots were through the roof.