Yeah, I’m sure he knows that but it would still be interesting to have it in there , plus triglycerides. Cos most GP’s don’t know that LDL s are not an indicator of heart health and are actually neuro protective. At least my GP practice didn’t know until I mentioned it. No idea if they took any notice from me.
I think the larger point is being missed with these studies. It’s not about lab panel associations that seem to be isolated here and there. Everything has a great deal to do with each other. Diabetes and cardiovascular disease are the same disease. As an eye doctor, I take the view that it’s happening in the eyes it’s happening elsewhere in the body too. The eyes and vision is where everything directly collides: vascular, neurological, striated muscle, smooth muscle, autonomic function, endocrine, immune, vestibular, auditory. Even colon polyps can have signs in the eye (Gardner’s syndrome). Look at the studies in regards to niacin and mitochondrial function. There is no krebs cycles without niacin. In fact niacin restores settling-3 pathway function in depletion. In the depletion of sertuin-3 pathway, the mitochondria recruit neighboring cardiocytes to begin and innate immune system response(a fancy way of saying localized inflammation). So why do these patients also have the same thing in the retina, except with neural tissue. In severe non-proliferative diabetic retinopathy, cotton wool spit show up, they are explained as poor oxygenation of the retinal tissue causing localized inflammation. I only partly agree with this hypothesis. I would venture to say this is poor metabolism in general including oxygen metabolism. One field of eye care known as syntonics, often scoffed at by some optometrists and ophthalmologists, has long wavelength la listen for retinal hemorrhages. Unbeknownst to these docs, research in mitochondria, melatonin and melanin research with long wavelength and how they interact with these components show promising improvements in their functionality. That is, red light is energizing to mitochondria and melanin. The possible mechanism that could vindicate these syntonic practitioners is that long wavelengths improve metabolism eliminating the need for the increase vascular permeability to aid in metabolic function thus retinal problem in diabetics begin to improve. This of course assumes the philosophy that our bodies are smart and know what they are doing by deliberately increasing the permeability of vasculature to aid in a waning metabolic function in which these are mistakenly called hemorrhages by the novice practitioners, but it is referred to as permeability in the literature. We should most definitely expect that every metabolically active tissue will then have this problem if it is in the eyes: heart, kidneys, gastrointestinal linings, brain (microvessel disease anyone?). I could say more about the physical science of niacin and how it activates differently in the presence of UV light, however no one seems to have applied that clinically but is food for thought. Similar things can be said about the B1 (thiamine) and the literature studies in regards to its protective effect against mitochondrial fission in muscular injury (including myo infarction). We know that Thiamine exists biologically as a diphosphate yet this is not what’s in supplements. Phosphorous is needed for B1 absorption and B1 is essential in recycling Phosphorous back to ATP, again no krebs cycle without thiamine. Mitochondrial health depends upon B1 and other B vitamins. This is why diabetics are known to be deficient in B vitamins. Sad to say supplementation is probably a lost cause due to unknown interactions between other vitamin cofactors and mineral cofactors that are required for proper restoration. I do believe there is a good piece to the puzzle though. So what does this have to do with insulin. Insulin is an indicator of metabolic disease. Insulin is needed, it does need to rise some after eating. But, what is triggering insulin to rise? Literature does indeed have suggestion about mitochondrial signaling in beta cells. This is an underlying metabolic issue. Late professor Dr Elliot Forrest at State University of New York Optometry wrote a book called “Stress and Vision” in which he uses the metabolic and endocrine basis for functional visual problems. The same principles apply for all chronic disease in my opinion. 1)first the stress(sympathetic), then then (parasympathetic) rest response as slow acting. If the stress is either remaining or perceived to have remained, a long acting parasympathetic response will entail until either termination of life or permanent restructuring occurs. This model explains myopia progression quite well as we see an increase in accommodation and esophoria as a stress response both parasympathetic responses until a permanent nearsightedness results and now suddenly the patient is exophoric again in order to permanently deal with the problem. Here a summary. 1)a stressor. When not removed 2)parasympathic, when not relieved 3)retoning of neurology, when exhausted 4)rebaselining of endocrine function or permanent structural change. Why can it we solve heart disease? Most heart disease, hypertension, diabetes and triglyceride problems are basically all the same with differing nuances from time to time. They got the model wrong…entirely wrong. Dr. Forrest is correct. A model conforming to his system adapted from vision to the above disease would be 1)metabolic stress due to poor nutrient availability, toxic substances and lack of quality/ abundance poor quality radiant energy. 2)metabolic stress results increasing localized vascular permeability and inflammation for cleaning up of dying tissue. The heart must keep pumping 3)relieve metabolic requirements of the heart by increasing peripheral resistance causing higher blood pressure (the retoning of the autonomic nervous system) via the ANS controlled smooth muscle surrounding vascular. 4)relieve strain on ANS by a deliberate, permanent structural change of deposition of plaque to permanently increase peripheral resistance. Why the association with insulin, because the mitochondrial signaling needs the insulin in order to drive the metabolic process in most essential organs and down regulate the receptors for the not-as-essential tissues. Therefore the insulin and the plaque are therefore both attempts by the body to solve the problem with the metabolic deficiencies while trying to preserve life. Solutions: One of the most important ones would be to quit all bread products in store (including breaded). Mill your own whole grains fresh at home and make your own bread. Out of 40-50 known major dietary nutrients, wheat is known to have 40. Listen to Sue Becker. ruclips.net/user/liveecxL66i-ne0?si=RLYMrwegpGH0eB4H ruclips.net/p/PLfWXNPCkO0yWnKpKQOQslaVx3TRCOAOQ1&si=wIQMmSxvhtbg5vQA www.breadbeckers.com/store/pc/Getting-Started-c185.htm
This....was....the..best comment. I not only nodded in agreement the entire time, as a nurse and a patient, and some one walking my father through all of this 'end result' catastrophe (he took all their meds did all their tests, followed all their everything....still ended up needing LAD stent, and now on the jetway for CABG), but printed this out. My optometrist said the SAME thing, and encouraged me to continue my HBOT. I can't even express how wonderful a qualitative commentary this was and so SO sad that no one else "liked" it.....LAZY (sorry, just too blunt for what this generation can handle but I won't stop for them). The summary was brilliant because it was dead on true, with a type of concise I can't do. Yes! They got the model wrong...for every disease, brain to toe, entire lifespan, womb to tomb.Excellent education and I'm so sad this was not appreciated but to let you know, I DO! Thank you!
@Jennifer-gr7hn I think it's more of a case of it being hard to understand for the layperson without a medical background, although I understood most of it. You used 3 acronyms in your post. Can you please tell me what they stand for?
Yes, he is extremely knowledgeable and has a good understanding of the material. If he has any fault, it would be making it palatable for the rest of us who do not have his level of knowledge and skill.
Don't forget chronically elevated cortisol. "High levels of cortisol, the stress hormone, are linked to increased arterial plaque buildup, meaning chronic stress can contribute to the development of atherosclerosis (plaque buildup in arteries) by promoting inflammation and impairing the normal anti-inflammatory function of cortisol, ultimately leading to potential cardiovascular issues."
It's what I believe led to a lot of our family diabetes....we ate clean by our cultural traditional food, grew a lot of our own stuff, a lot of omega 3, healthy meats, fish, eggs, leafy greens, oils, EVOO,etc...but a lot of stress, also mthfr mutations. The stress is constant cortisol. I thank you for bringing this up because I was about it. I am SO sick of people shallowly and narrowly thinking it's "diet and exercise" issues alone. If it was, none of us here would have the syndromes, meanwhile some mordibly obese, fast "food" eating, sedentary people who don't, would.
@@brandonyoung4910 as well as the hard work as there is no quick fix or herb to work THROUGH trauma, ptsd, that leads to working in stressful jobs, being in stressful relationships, etc.
Much like the study discussed in the video, I don't like "linked", "associated", etc. I want experimental data, for specific groups. Even cortisol as a "stress hormone" is somewhat misleading, chronic stress increases cortisol, to my understanding, the reverse is not true, and does not mean there aren't other reasons to increase cortisol production. Insulin does seem pretty closely tied to blood sugar however. edit: easy points to make about cortisol on different populations: high cortisol breaks down lean tissue in normal populations, fasting increases cortisol chronically over the duration of the fast, yet it does not result in significant loss of lean tissue when the subject is active. Same is true of people on keto diets which mimic fasting.
Chronically elevated insulin increases blood sugar and that leads to higher insulin. Insulin is what is important, everything else is connected to insulin...
I'd bet a dollar that LDL ended up not having a statistically significant relationship with plaque formation. I've often heard the size of your LDL particles has a greater correlation to CVD risk than the actual number of LDL particles in your blood. Apparently, smaller LDL particles can more easily penetrate the endothelium. However, there are also theories that it is lp(a) or even foam cells that cause the plaque formations, not LDL. I find it quite humorous that we as a species have no idea the specific mechanism for plaque formation. We can only come up with theories. I wish some big pharma money could be used to study it, but we all know "a patient cured is a customer lost."
LDL particles can't penetrate the endothelium. What happens at sites of plaque formation (arterial scabs) is that circulating endothelium progenitor cells create a new layer of endothelium on top of the plaque. Giving the illusion that plaque penetrates the lining of blood vessels.
This was what someone said in r/Cholersterol when I asked recently. That triglycerides, cac, LDL doesn't really tell enough and there are other ways of actually checking it. I can't recall the specifics, but large LDL particles was among one of the causes mentioned.
I've got CVD on both sides of family. Dad had 6 arteries bypassed at age 39 and died at 58. Full brother died of heart attack at 37. He was just at the top range of a healthy bmi and was riding his motorcycle with paddles at the dunes the weekend before and snowboarding a couple times the week before that..... I got tested afterward and had medium high Lp(a) even though overall cholesterol was 178 I believe. LDL and apoB form were also a little on the high side even with that overall decent number. 6 years after that test in 2022 I had CT angiography done at a Providence hospital and read by a well respected cardiologist with many years experience. He was dumbfounded to find that there wasn't any thickening at all. The CAC score was 0 then and 0 when I did one the 6 years prior after my brother died. I also did a CMIT that came back with no abnormalities. I started learning about nutrition, herbs, et cetera back when I was ten years old, and my dad was having surgery. Polyphenols are one of the main things I swear by..... I've done plenty of things wrong, but a lot of really good things at the same time. E.g. I would binge drink with friends a couple nights and then spend the next couple making green juices with a Gold Star juicer; I've been downing handfuls of supplements at a time since middle school age..... things of that sort. I think there's a lot more to the story of heart disease. Namely, the inflammation and immune system aspects that can be kept under control even if some things don't look so good. Oh, did I mention I smoked a pack+ of cigarettes daily from 1992 until 2003 as well 😊
it's NOT just the sugar! It's not even the sugar! it's about oils that clog the liver, then making it harder for the pancrease to deal WITH sugar into which everything boils down.
@@Jennifer-gr7hn insulin resistance is a far bigger issue. People are ingesting way more sugar than we have ever consumed throughout human history regardless of oil being present in the foods they eat. High oil consumption isn’t great, but the sugar found in all foods is a recent (last 50 years or so) phenomenon that has resulted in the highest rates of obesity and metabolic diseases, which is leading to early death.
Is there value in distinguishing between type of oil and sugar? Is it possible to do so, given the almost exclusive use of “vegetable” / seed oils in conjunction with carbohydrates in “food”?
Found this on a Physionics Video, hope this person was giving accurate information. >>Two years ago, a 2D echocardiogram revealed coronary calcification, which was preventing one heart valve from closing. My doctor indicated that no surgery was immediately warranted, and the issue would be monitored. I sought some way to address the calcification, and eventually learned about Berberine possibly being able to dissolve the calcification. I started taking Berberine as a daily supplement about a year ago. Last month, a follow up ultrasound of my heart found all valves to be functioning normally, and there were no signs of coronary calcification! I am so relieved and glad that it worked. Meanwhile, my cardiologist asked for the second time, "What's Berberine?" Go figure.
If serum (blood) insulin levels are higher in a diabetic getting insulin injections as part of standard treatment, than a diabetic using a low-carb diet approach, or than even a non-diabetic, aren't insulin injections likely increasing a person's risk of vulnerable plaque components under the guise of "managing diabetes?" So part and parcel of standard "treating" diabetes is the doctor-induced increase of negative heart/stroke events?
Immediate thoughts: that's a lot of patients on antihypertensive medication, and the association with insulin is there. What are the side effects of antihypertensive medication? (it's a pity they lump all blood pressure medication together) Especially given that the reason for high blood pressure is high resistance, and thus lowering blood pressure may reduce blood, ie. oxygen and nutrition, availability. Diuretics may be an exception to that, but those would reduce minerals and possibly increase burden on kidneys, especially seeing how kidneys generally increase renin production in response to low sodium (and possibly other minerals), and renin increases BP. Kidneys are also part of the calcium absorption cycle, and loop back to this discussion that way too. Also, any relative risk less than 100% in epidemiology can generally be ignored outright.
The other component that’s completely over looked is definitely the degree of inflammation in the blood vessel linings! As you mentioned, higher serum insulin can cause a build up of white blood cells and necrotic tissue to develop within the lining of the artery linings. Having said that, i definitely think that monitoring all inflammatory markers such as highly sensitive C-reactive protein, tumor necrosis factor, insulin like factors are a must in this study!!! That is why you don’t always observe a significant amount of plaque buildup if the amount of insulin does not induce inflammation caused by what you mentioned towards the end of your presentation. And I’m sure you’re quite aware that insulin has multiple physiological functions and effects upon the body as well such as inducing fat storage, increasing cortisol levels as well in addition to allowing for glucose permeability through cellular membranes if the receptors are working properly. Also induces other metabolic processes such as the increase in MTOR or mammalian target of Rapamyosin which stimulates growth in addition to signaling for glucagon production as well through a set of biofeedback mechanisms.
Thanks for info, I work in a hospital serving food ,diabetes is so common, you should read the book newly written Post Diabetic,don't have book near me.
Biomed engineer in coronary just to let you know where I am coming from, not going to reference but recent findings in my field using intravascular Imaging has found that high shear rates are also associated with vulnerable plaque, like waves of ocean eroding rock, damaging... you know the rest
It is very interesting to learn about the heart conditions ,2 of my uncles died at 45 ,the other 55 dropped dead 2 of mums cousins @47 and 49 also 3 uncles at 70.
eeeeeexcellent point because fungi is HUGE - VERY big problem, and then the oxalates that come with that.....yes yes and YES.. So thankful you brought that up. People are so narrow!
I wonder who paid for the study to be done🤔. It seems to me there is good reason Type 2 diabetes strokes and Heart attacks have become more prevalent in these past 8 decades.
It all comes down to mitochondrial function. If its not working correctly then glucose oxidation is impaired. When cant use glucose correctly, insulin goes up. Now you have higher levels of glucose from it not being matabolized and higher levels of insulin as an attempt to force glucose oxidation. This signals cortisol, oxidative stress and inflammation which over time cause all helath related issues. I think everything comes back to mitochondrial function.
It’s important to understand that avoiding carbohydrate does not improve fasting insulin. In fact up regulating glucagon and cortisol can increase insulin resistance, fasting blood sugar, and insulin
Weird, i am almost totally carb free and have been for 3 years. My fasting insulin is 3. If your glucose intake is low, so goes your insulin levels. That's why it reverses T2D. Maybe I read your post wrong?
@@DaveIrish66 in the short run, yes......but if you resumed any carbs, you'd go right back to it...THAT is not then a cure. Food is just one part of diabetes. It's stress, not being breastfed, infections, injections, sleep habits, ptsd, heavy metals, fungi build up, eeeetc. Soooo much more is going on with the mitochondria
@@Jennifer-gr7hn if carbs are a poison to you, then resuming ingesting poison will bring back the symptoms of poisoning that said, even after resuming carbs (slowly) he would have lowered insulin response for quite some time
Great video. Keep of this graduate level education you provide! Everyone needs to understand how to separate the money hungry cardiology machine causes and treatments from the real science. Your videos help us all understand how to do that for our own health wellbeing. Sort of like being able to separate raisins from rabbit poop. They can look a lot a like until you pop them in your mouth.
10:46 It is confusing: Glucose below and higher than 7 mmol/L are associated with increased risk? Does it have to mean prediabetes (5,6-6,9 mmol/L) and diabetes (7 mmol/L), but not less than 5,6 mmol/L which is normal, right?
Therefore, it may be the secondary effects of insulin levels that may lead to plaque development rather than the primary effects of insulin itself. What the insulin molecule affects other physiological processes which are secondary effects that may be the culprit!
Glucose is acidic & excess glucose in the blood stream causes the blood pH to become acidic. This acidic environment damages the inner lining of the blood vessels causing atherosclerosis. Gluconeogenesis provides the exact dose & correct type of glucose as required. All glucose consumed is excess & is removed from the blood stream by being processed into fat. Causing obesity. Fructose cannot be metabolized by our cells nor processed into fat via insulin. It can only be processed by the liver, causing non-alcoholic fatty liver disease & causes the blood sugar levels to remain elevated for extended periods. Producing insulin & adrenaline to process excess glucose depletes one of essential vitamins & minerals. Causing nutrient deficient disorders such as type 2 diabetes. Atherosclerosis, type 2 diabetes & obesity are symptoms of the same metabolic disorders caused by sugar & all food digested as sugar. This includes all grains, fruit & high glycemic vegetable matter. The cure is to simply remove the cause. Seed & vegetable oils are the worse substances one can consume to destroy their liver.
Sorry but you’re wrong on all points besides the seed oils thing at the end. Check the Jay Feldman wellness channel for a better picture of how fructose is metabolized and why we don’t want to rely on gluconeogenesis nor be a “fat burner”
@@aspiresk8boarding He's another fool preaching the nonsensical Lipid Hypothesis & a high carb/sugar diet. IF you don't wake up & keep believing such nonsense, you will learn the truth in the hardest way.
@@_Hal9000 unsustainable and managing symptoms. It won't fix the fungi and metal overload, it won't cure the ptsd an childhood trauma, it won't deal with the blue light the Nitric oxide deficiencies, eeeetc. Oversimpificaiton and not a fix in the long run
@@Jennifer-gr7hn Actually it kinda is easy. How did the human body just survive all the time.... Low carb, keeps the mood stable and the brain more focused. Brain wants ketons more than carbs. This helps too with immune issues. Stable mood helps with the coping from trauma or living in general, less stress better sleep. But indeed, the way to get there can be hard, since sugar is very addictive. If no gene defect is present the NO is produced in the cell from Arginin which is in a lot of foods. Blue light... broo. Just put the phone down... good mood = less need for distraction. The nordic phenotype human used carbs in summer and autumn to fatten itself for winter. We can do something similar to hibernation.
I did calcium ct for 3 yrs. Had high blood sugars. One artery did reduce when I got my blood sugar down. The other one increased. This is why I think I have increased calcium showing up in one coronary artery. Would love to know if this is healing or getting worse.
But is serum insulin a true cause or just a marker for other things e.g. diabetes? IE people with high serum insulin are probably diabetic or at least moving in that direction
Looking at the table going from group with low insulin values to the highest insulin values interplak Hemorrhage went from 32% to 35% with no error bars..... so basically the same. I see NO RESULT here. AND calcification score actually went down in the higher insulin level group. ?!?
While the study is on the cutting edge, one study isn't sufficient to base anything on. Let's maintain perspective. What are you suggesting is actionable resulting from this study?
Mike, did you delete your video about Trglycerides/HDL Ratio video? I can't find the long form video, but there is still a short from that video. If you discovered something new, you'd need to delete that short too
My dad is getting a cardiac bypass next month. It’s got me wondering about my cardiovascular status. One day when I get health coverage, I’m gonna get that stuff checked out.
Wanna know your cardiovascular status? Get a CAC score. It will tell you where you stand with calcium buildup in the coronary arteries. The test is about $100 and at many imaging centers you don't need a doctor's order to get it. Easy-peasy. I just paid $99 for mine.
@@swamphawk6227 very limited 'advice,' and also, don't wait for coverage. Get to a functional medicine practitioner, spend the money (if you have an iPhone, eat out, drive a car...it's worth the sacrifice), and get checked out. I will pray for your dad. Mine is also having that (maybe) next month at all but thanks to his nurse daughter thinker guardian angel, I had to save his life from it twice now as the doctors weren't looking at HIM but at labs and I said "he doesn't look good, and he's still flippin bleeding from the cardiac Cath site, and his kidneys are now compromised from the lack of IV fluids you gave him! His liver and kidneys are messed up and it may not be worth it but we'll see after some detoxing I have him on, glutathione, and baths, saunas...to see if he's better able to withstand the bypass. VERY risky surgery. Tell me about it, medical ptsd for me as a nurse and patient and I'm scared for him but stay strong for him. I cry when I get home. My gut doesn't feel good about it....
Just assume that you do have cardiovascular problems and change your lifestyle accordingly. Better diet, more exercise, more sleep, less stress. No need to wait for health coverage.
Is this "vulnerable plaque" even plaque at all, or actually blood clots, which some argue are actually the real cause of plaque in the first place. Bearing in mind that real plaque is behind the artery wall and cant be dislodged to cause problems downstream.
The best and only scientific method is to do a Glucose tolerance test with insulin measurements. This is confirmed in the book by Professor Joseph Kraft, and known as the Kraft Test. Anything other than that is just not good enough…. Patients are therefore being ‘short changed’ by the medical profession, when they have an Hba1c, or a fasting glucose level to assess their health…
@ I worked in Pharma for 12 years. It was a straight business at that time - 1990 - 2002. The company I worked for was ethical. No buying of prescriptions and they followed AMA guidelines. Things have changed and the pendulum must come back for the sake of everyone’s health and well-being.
@@Jennifer-gr7hn you can also add a$$h0le to your ‘extraordinary’ academic CV. You made a lot of assumptions about me that are not anywhere near the truth. Get a life.
So if we have a higher fasted insulin number if we can get it down it will reduce the damage/plaque in theory potentially? What are those median numbers to aim for? Mine was in low teens so can't be the units of measure showing on this study. I know I need to get it down. I've heard others say 5 is what to aim for?
On glucose meter like diabetics use, glucose should run below 100 about an hour and a half after eating. A1C should be no higher than about 5.5. Dr. Paul Mason states that A1C is not accurate in lots of cases where the person does not eat much carbs because the blood cells don’t turn over as fast as normal. Mike should comment on this.
Thanks for the video. Very interesting. Just fyi, ischemic is pronounced /ɪˈskiː.mɪk/ not /I' she mik/ The sch makes a hard k sound like in ski. Thanks again!
LDL is not an issue. It’s just used to sell statins.
Yeah, I’m sure he knows that but it would still be interesting to have it in there , plus triglycerides. Cos most GP’s don’t know that LDL s are not an indicator of heart health and are actually neuro protective. At least my GP practice didn’t know until I mentioned it. No idea if they took any notice from me.
I think the larger point is being missed with these studies. It’s not about lab panel associations that seem to be isolated here and there. Everything has a great deal to do with each other.
Diabetes and cardiovascular disease are the same disease. As an eye doctor, I take the view that it’s happening in the eyes it’s happening elsewhere in the body too. The eyes and vision is where everything directly collides: vascular, neurological, striated muscle, smooth muscle, autonomic function, endocrine, immune, vestibular, auditory. Even colon polyps can have signs in the eye (Gardner’s syndrome).
Look at the studies in regards to niacin and mitochondrial function. There is no krebs cycles without niacin. In fact niacin restores settling-3 pathway function in depletion. In the depletion of sertuin-3 pathway, the mitochondria recruit neighboring cardiocytes to begin and innate immune system response(a fancy way of saying localized inflammation). So why do these patients also have the same thing in the retina, except with neural tissue. In severe non-proliferative diabetic retinopathy, cotton wool spit show up, they are explained as poor oxygenation of the retinal tissue causing localized inflammation. I only partly agree with this hypothesis. I would venture to say this is poor metabolism in general including oxygen metabolism. One field of eye care known as syntonics, often scoffed at by some optometrists and ophthalmologists, has long wavelength la listen for retinal hemorrhages. Unbeknownst to these docs, research in mitochondria, melatonin and melanin research with long wavelength and how they interact with these components show promising improvements in their functionality. That is, red light is energizing to mitochondria and melanin. The possible mechanism that could vindicate these syntonic practitioners is that long wavelengths improve metabolism eliminating the need for the increase vascular permeability to aid in metabolic function thus retinal problem in diabetics begin to improve. This of course assumes the philosophy that our bodies are smart and know what they are doing by deliberately increasing the permeability of vasculature to aid in a waning metabolic function in which these are mistakenly called hemorrhages by the novice practitioners, but it is referred to as permeability in the literature. We should most definitely expect that every metabolically active tissue will then have this problem if it is in the eyes: heart, kidneys, gastrointestinal linings, brain (microvessel disease anyone?). I could say more about the physical science of niacin and how it activates differently in the presence of UV light, however no one seems to have applied that clinically but is food for thought.
Similar things can be said about the B1 (thiamine) and the literature studies in regards to its protective effect against mitochondrial fission in muscular injury (including myo infarction). We know that Thiamine exists biologically as a diphosphate yet this is not what’s in supplements. Phosphorous is needed for B1 absorption and B1 is essential in recycling Phosphorous back to ATP, again no krebs cycle without thiamine. Mitochondrial health depends upon B1 and other B vitamins. This is why diabetics are known to be deficient in B vitamins. Sad to say supplementation is probably a lost cause due to unknown interactions between other vitamin cofactors and mineral cofactors that are required for proper restoration. I do believe there is a good piece to the puzzle though.
So what does this have to do with insulin. Insulin is an indicator of metabolic disease. Insulin is needed, it does need to rise some after eating. But, what is triggering insulin to rise? Literature does indeed have suggestion about mitochondrial signaling in beta cells. This is an underlying metabolic issue.
Late professor Dr Elliot Forrest at State University of New York Optometry wrote a book called “Stress and Vision” in which he uses the metabolic and endocrine basis for functional visual problems. The same principles apply for all chronic disease in my opinion. 1)first the stress(sympathetic), then then (parasympathetic) rest response as slow acting. If the stress is either remaining or perceived to have remained, a long acting parasympathetic response will entail until either termination of life or permanent restructuring occurs. This model explains myopia progression quite well as we see an increase in accommodation and esophoria as a stress response both parasympathetic responses until a permanent nearsightedness results and now suddenly the patient is exophoric again in order to permanently deal with the problem.
Here a summary. 1)a stressor. When not removed 2)parasympathic, when not relieved 3)retoning of neurology, when exhausted 4)rebaselining of endocrine function or permanent structural change.
Why can it we solve heart disease? Most heart disease, hypertension, diabetes and triglyceride problems are basically all the same with differing nuances from time to time. They got the model wrong…entirely wrong. Dr. Forrest is correct. A model conforming to his system adapted from vision to the above disease would be 1)metabolic stress due to poor nutrient availability, toxic substances and lack of quality/ abundance poor quality radiant energy. 2)metabolic stress results increasing localized vascular permeability and inflammation for cleaning up of dying tissue. The heart must keep pumping 3)relieve metabolic requirements of the heart by increasing peripheral resistance causing higher blood pressure (the retoning of the autonomic nervous system) via the ANS controlled smooth muscle surrounding vascular. 4)relieve strain on ANS by a deliberate, permanent structural change of deposition of plaque to permanently increase peripheral resistance.
Why the association with insulin, because the mitochondrial signaling needs the insulin in order to drive the metabolic process in most essential organs and down regulate the receptors for the not-as-essential tissues.
Therefore the insulin and the plaque are therefore both attempts by the body to solve the problem with the metabolic deficiencies while trying to preserve life.
Solutions:
One of the most important ones would be to quit all bread products in store (including breaded). Mill your own whole grains fresh at home and make your own bread. Out of 40-50 known major dietary nutrients, wheat is known to have 40. Listen to Sue Becker.
ruclips.net/user/liveecxL66i-ne0?si=RLYMrwegpGH0eB4H
ruclips.net/p/PLfWXNPCkO0yWnKpKQOQslaVx3TRCOAOQ1&si=wIQMmSxvhtbg5vQA
www.breadbeckers.com/store/pc/Getting-Started-c185.htm
This....was....the..best comment. I not only nodded in agreement the entire time, as a nurse and a patient, and some one walking my father through all of this 'end result' catastrophe (he took all their meds did all their tests, followed all their everything....still ended up needing LAD stent, and now on the jetway for CABG), but printed this out. My optometrist said the SAME thing, and encouraged me to continue my HBOT. I can't even express how wonderful a qualitative commentary this was and so SO sad that no one else "liked" it.....LAZY (sorry, just too blunt for what this generation can handle but I won't stop for them). The summary was brilliant because it was dead on true, with a type of concise I can't do. Yes! They got the model wrong...for every disease, brain to toe, entire lifespan, womb to tomb.Excellent education and I'm so sad this was not appreciated but to let you know, I DO! Thank you!
Absolutely great comment. Hope to see more of this. Shame YT does not let viewers copy the text
@@Jennifer-gr7hnYup! Me too
@Jennifer-gr7hn I think it's more of a case of it being hard to understand for the layperson without a medical background, although I understood most of it. You used 3 acronyms in your post. Can you please tell me what they stand for?
Thanks ❤❤❤❤❤ and solutions please
THIS GUY PRESENTS THE BEST INFO ANYWHERE. BAR NONE
I’ve been saying this for literally 5 years now
Yes, he is extremely knowledgeable and has a good understanding of the material. If he has any fault, it would be making it palatable for the rest of us who do not have his level of knowledge and skill.
@@jamboy4308 That and slowing down and trying to breathe properly through his partially blocked nose....
LDL surely was included in the trial but the data was omitted in the report ... because the result most likely was unfavorable to statins.
The concept of health in "The 23 Former Doctor Truths" book completely explains this. I wish I read it sooner.
Go away bot and take your pathetic fake likes with you.
i ve been looking for this book . where can u get this book may i ask ?
Don't forget chronically elevated cortisol. "High levels of cortisol, the stress hormone, are linked to increased arterial plaque buildup, meaning chronic stress can contribute to the development of atherosclerosis (plaque buildup in arteries) by promoting inflammation and impairing the normal anti-inflammatory function of cortisol, ultimately leading to potential cardiovascular issues."
Shoutout to ashwagandha
It's what I believe led to a lot of our family diabetes....we ate clean by our cultural traditional food, grew a lot of our own stuff, a lot of omega 3, healthy meats, fish, eggs, leafy greens, oils, EVOO,etc...but a lot of stress, also mthfr mutations. The stress is constant cortisol. I thank you for bringing this up because I was about it. I am SO sick of people shallowly and narrowly thinking it's "diet and exercise" issues alone. If it was, none of us here would have the syndromes, meanwhile some mordibly obese, fast "food" eating, sedentary people who don't, would.
@@brandonyoung4910 as well as the hard work as there is no quick fix or herb to work THROUGH trauma, ptsd, that leads to working in stressful jobs, being in stressful relationships, etc.
Much like the study discussed in the video, I don't like "linked", "associated", etc. I want experimental data, for specific groups. Even cortisol as a "stress hormone" is somewhat misleading, chronic stress increases cortisol, to my understanding, the reverse is not true, and does not mean there aren't other reasons to increase cortisol production. Insulin does seem pretty closely tied to blood sugar however.
edit: easy points to make about cortisol on different populations: high cortisol breaks down lean tissue in normal populations, fasting increases cortisol chronically over the duration of the fast, yet it does not result in significant loss of lean tissue when the subject is active. Same is true of people on keto diets which mimic fasting.
Chronically elevated insulin increases blood sugar and that leads to higher insulin.
Insulin is what is important, everything else is connected to insulin...
You have to ask if Nattokinase, Serrapeptase, Lumbrokinase, aged black garlic, berberine are safe to apply to this sort of plaque.
I'd bet a dollar that LDL ended up not having a statistically significant relationship with plaque formation. I've often heard the size of your LDL particles has a greater correlation to CVD risk than the actual number of LDL particles in your blood. Apparently, smaller LDL particles can more easily penetrate the endothelium. However, there are also theories that it is lp(a) or even foam cells that cause the plaque formations, not LDL.
I find it quite humorous that we as a species have no idea the specific mechanism for plaque formation. We can only come up with theories. I wish some big pharma money could be used to study it, but we all know "a patient cured is a customer lost."
LDL particles can't penetrate the endothelium. What happens at sites of plaque formation (arterial scabs) is that circulating endothelium progenitor cells create a new layer of endothelium on top of the plaque. Giving the illusion that plaque penetrates the lining of blood vessels.
HDL particle sizes are smaller than the various forms of LDL particles.
My Lpa is 450:( I'm focusing on keeping my blood sugar and insulin levels low.
This was what someone said in r/Cholersterol when I asked recently. That triglycerides, cac, LDL doesn't really tell enough and there are other ways of actually checking it. I can't recall the specifics, but large LDL particles was among one of the causes mentioned.
I've got CVD on both sides of family. Dad had 6 arteries bypassed at age 39 and died at 58. Full brother died of heart attack at 37. He was just at the top range of a healthy bmi and was riding his motorcycle with paddles at the dunes the weekend before and snowboarding a couple times the week before that..... I got tested afterward and had medium high Lp(a) even though overall cholesterol was 178 I believe. LDL and apoB form were also a little on the high side even with that overall decent number. 6 years after that test in 2022 I had CT angiography done at a Providence hospital and read by a well respected cardiologist with many years experience. He was dumbfounded to find that there wasn't any thickening at all. The CAC score was 0 then and 0 when I did one the 6 years prior after my brother died. I also did a CMIT that came back with no abnormalities. I started learning about nutrition, herbs, et cetera back when I was ten years old, and my dad was having surgery. Polyphenols are one of the main things I swear by..... I've done plenty of things wrong, but a lot of really good things at the same time. E.g. I would binge drink with friends a couple nights and then spend the next couple making green juices with a Gold Star juicer; I've been downing handfuls of supplements at a time since middle school age..... things of that sort. I think there's a lot more to the story of heart disease. Namely, the inflammation and immune system aspects that can be kept under control even if some things don't look so good. Oh, did I mention I smoked a pack+ of cigarettes daily from 1992 until 2003 as well 😊
Very interesting, thanks!
sugar in foods should be regulated as cigarette does
This is such an underrated statement. I couldn’t agree more with you.
it's NOT just the sugar! It's not even the sugar! it's about oils that clog the liver, then making it harder for the pancrease to deal WITH sugar into which everything boils down.
@@Jennifer-gr7hn insulin resistance is a far bigger issue. People are ingesting way more sugar than we have ever consumed throughout human history regardless of oil being present in the foods they eat. High oil consumption isn’t great, but the sugar found in all foods is a recent (last 50 years or so) phenomenon that has resulted in the highest rates of obesity and metabolic diseases, which is leading to early death.
Is there value in distinguishing between type of oil and sugar? Is it possible to do so, given the almost exclusive use of “vegetable” / seed oils in conjunction with carbohydrates in “food”?
Found this on a Physionics Video, hope this person was giving accurate information.
>>Two years ago, a 2D echocardiogram revealed coronary calcification, which was preventing one heart valve from closing. My doctor indicated that no surgery was immediately warranted, and the issue would be monitored. I sought some way to address the calcification, and eventually learned about Berberine possibly being able to dissolve the calcification.
I started taking Berberine as a daily supplement about a year ago.
Last month, a follow up ultrasound of my heart found all valves to be functioning normally, and there were no signs of coronary calcification! I am so relieved and glad that it worked. Meanwhile, my cardiologist asked for the second time, "What's Berberine?"
Go figure.
Try vitamin k2 also maybe
Keep healthy
@josephhooper8655 Absolutely, and find Maurice Daher in here.
If serum (blood) insulin levels are higher in a diabetic getting insulin injections as part of standard treatment, than a diabetic using a low-carb diet approach, or than even a non-diabetic, aren't insulin injections likely increasing a person's risk of vulnerable plaque components under the guise of "managing diabetes?" So part and parcel of standard "treating" diabetes is the doctor-induced increase of negative heart/stroke events?
Thanks for your information this kind of thing interests me a lot 👍
I don’t see triglycerides here either LDL can be calculated from total cholesterol, HDL and a part of triglycerides. Strange
Immediate thoughts: that's a lot of patients on antihypertensive medication, and the association with insulin is there. What are the side effects of antihypertensive medication? (it's a pity they lump all blood pressure medication together) Especially given that the reason for high blood pressure is high resistance, and thus lowering blood pressure may reduce blood, ie. oxygen and nutrition, availability. Diuretics may be an exception to that, but those would reduce minerals and possibly increase burden on kidneys, especially seeing how kidneys generally increase renin production in response to low sodium (and possibly other minerals), and renin increases BP. Kidneys are also part of the calcium absorption cycle, and loop back to this discussion that way too.
Also, any relative risk less than 100% in epidemiology can generally be ignored outright.
They must have removed LDL because of the findings. The industry is deeply corrupt
The other component that’s completely over looked is definitely the degree of inflammation in the blood vessel linings! As you mentioned, higher serum insulin can cause a build up of white blood cells and necrotic tissue to develop within the lining of the artery linings. Having said that, i definitely think that monitoring all inflammatory markers such as highly sensitive C-reactive protein, tumor necrosis factor, insulin like factors are a must in this study!!! That is why you don’t always observe a significant amount of plaque buildup if the amount of insulin does not induce inflammation caused by what you mentioned towards the end of your presentation. And I’m sure you’re quite aware that insulin has multiple physiological functions and effects upon the body as well such as inducing fat storage, increasing cortisol levels as well in addition to allowing for glucose permeability through cellular membranes if the receptors are working properly. Also induces other metabolic processes such as the increase in MTOR or mammalian target of Rapamyosin which stimulates growth in addition to signaling for glucagon production as well through a set of biofeedback mechanisms.
Thanks for info, I work in a hospital serving food ,diabetes is so common, you should read the book newly written Post Diabetic,don't have book near me.
Thank you for your Great work, is there a link available to this study.
Ian
Can you tell us the actual insulin levels? What's the rande for high level? Thanks
👍 give 👍 a 👍 like 👍 for 👍 Mike! 👍
Biomed engineer in coronary just to let you know where I am coming from, not going to reference but recent findings in my field using intravascular Imaging has found that high shear rates are also associated with vulnerable plaque, like waves of ocean eroding rock, damaging... you know the rest
So good! Thank you !
It is very interesting to learn about the heart conditions ,2 of my uncles died at 45 ,the other 55 dropped dead 2 of mums cousins @47 and 49 also 3 uncles at 70.
I think you answered your own question about WHY LDL wasn't included. Cause, like Angel Keys, they leave out data that does not support their theory !
True, however it does so only as a function of system microbial dysbiosis and fungal infection of blood vessels.
eeeeeexcellent point because fungi is HUGE - VERY big problem, and then the oxalates that come with that.....yes yes and YES.. So thankful you brought that up. People are so narrow!
Bring back Dr Cowan please
Great info, thanks Mike
Triglycerides to HDL ratio
I wonder who paid for the study to be done🤔. It seems to me there is good reason Type 2 diabetes strokes and Heart attacks have become more prevalent in these past 8 decades.
It all comes down to mitochondrial function. If its not working correctly then glucose oxidation is impaired. When cant use glucose correctly, insulin goes up. Now you have higher levels of glucose from it not being matabolized and higher levels of insulin as an attempt to force glucose oxidation. This signals cortisol, oxidative stress and inflammation which over time cause all helath related issues. I think everything comes back to mitochondrial function.
Always enjoy and find informative these types of reviews. Thank you very much. I am an ardent viewer.
It’s important to understand that avoiding carbohydrate does not improve fasting insulin. In fact up regulating glucagon and cortisol can increase insulin resistance, fasting blood sugar, and insulin
Weird, i am almost totally carb free and have been for 3 years. My fasting insulin is 3.
If your glucose intake is low, so goes your insulin levels. That's why it reverses T2D. Maybe I read your post wrong?
It improved mine. Down to 5.
@@DaveIrish66 in the short run, yes......but if you resumed any carbs, you'd go right back to it...THAT is not then a cure. Food is just one part of diabetes. It's stress, not being breastfed, infections, injections, sleep habits, ptsd, heavy metals, fungi build up, eeeetc. Soooo much more is going on with the mitochondria
it generally does; short term it might not if the change is drastic
@@Jennifer-gr7hn if carbs are a poison to you, then resuming ingesting poison will bring back the symptoms of poisoning
that said, even after resuming carbs (slowly) he would have lowered insulin response for quite some time
Great video. Keep of this graduate level education you provide!
Everyone needs to understand how to separate the money hungry cardiology machine causes and treatments from the real science.
Your videos help us all understand how to do that for our own health wellbeing.
Sort of like being able to separate raisins from rabbit poop.
They can look a lot a like until you pop them in your mouth.
Mike ,thanks for a very good vid! i appreciate your info!
No correlation Or mention...with LDL means that it has no Statistical. basis in the study.
The measurement units are confusing. My insulin levels are reported in uIU/mL. How would I convert the units used in the Dutch study to these units?
10:46 It is confusing: Glucose below and higher than 7 mmol/L are associated with increased risk? Does it have to mean prediabetes (5,6-6,9 mmol/L) and diabetes (7 mmol/L), but not less than 5,6 mmol/L which is normal, right?
What is your take on the CIMT test for identifying vulnerable plaque?
Dr Ford Brewer says this is the test to have.
I thoroughly enjoyed rewatching this 💕👜🌸🥰
Therefore, it may be the secondary effects of insulin levels that may lead to plaque development rather than the primary effects of insulin itself. What the insulin molecule affects other physiological processes which are secondary effects that may be the culprit!
Thank YOU 😊
Glucose is acidic & excess glucose in the blood stream causes the blood pH to become acidic. This acidic environment damages the inner lining of the blood vessels causing atherosclerosis.
Gluconeogenesis provides the exact dose & correct type of glucose as required. All glucose consumed is excess & is removed from the blood stream by being processed into fat. Causing obesity.
Fructose cannot be metabolized by our cells nor processed into fat via insulin. It can only be processed by the liver, causing non-alcoholic fatty liver disease & causes the blood sugar levels to remain elevated for extended periods.
Producing insulin & adrenaline to process excess glucose depletes one of essential vitamins & minerals. Causing nutrient deficient disorders such as type 2 diabetes.
Atherosclerosis, type 2 diabetes & obesity are symptoms of the same metabolic disorders caused by sugar & all food digested as sugar. This includes all grains, fruit & high glycemic vegetable matter. The cure is to simply remove the cause.
Seed & vegetable oils are the worse substances one can consume to destroy their liver.
Sorry but you’re wrong on all points besides the seed oils thing at the end. Check the Jay Feldman wellness channel for a better picture of how fructose is metabolized and why we don’t want to rely on gluconeogenesis nor be a “fat burner”
@@aspiresk8boarding
Why not? It keeps insuline low
@@aspiresk8boarding He's another fool preaching the nonsensical Lipid Hypothesis & a high carb/sugar diet. IF you don't wake up & keep believing such nonsense, you will learn the truth in the hardest way.
@@_Hal9000 unsustainable and managing symptoms. It won't fix the fungi and metal overload, it won't cure the ptsd an childhood trauma, it won't deal with the blue light the Nitric oxide deficiencies, eeeetc. Oversimpificaiton and not a fix in the long run
@@Jennifer-gr7hn
Actually it kinda is easy.
How did the human body just survive all the time....
Low carb, keeps the mood stable and the brain more focused.
Brain wants ketons more than carbs. This helps too with immune issues. Stable mood helps with the coping from trauma or living in general, less stress better sleep.
But indeed, the way to get there can be hard, since sugar is very addictive.
If no gene defect is present the NO is produced in the cell from Arginin which is in a lot of foods.
Blue light... broo. Just put the phone down... good mood = less need for distraction.
The nordic phenotype human used carbs in summer and autumn to fatten itself for winter.
We can do something similar to hibernation.
Make America Healthy Again
Make the world healthy again
@@M1979WBthey are already healthier they ban a lot of the chemicals we have in our foods already
I did calcium ct for 3 yrs. Had high blood sugars. One artery did reduce when I got my blood sugar down. The other one increased.
This is why I think I have increased calcium showing up in one coronary artery.
Would love to know if this is healing or getting worse.
Thanks
You should debate Simon Hill and show em how its done 🙏🏼
But is serum insulin a true cause or just a marker for other things e.g. diabetes? IE people with high serum insulin are probably diabetic or at least moving in that direction
that's the problem with epidemiology, we can draw hypotheses but not confirm anything
Ischemia is pronounced "iskeemia"
and Kuopio is pronounced ku- (as in couscous) o- (as in Ontario) pi- (as in pit) o (as in Ontario; Finnish is phonetically pretty consistent)
will you do a video on saw palmetto extract and other prostate supplements and dosages?
Looking at the table going from group with low insulin values to the highest insulin values interplak Hemorrhage went from 32% to 35% with no error bars..... so basically the same. I see NO RESULT here. AND calcification score actually went down in the higher insulin level group. ?!?
Please provide link to the article
Thanks
I sleep badly and mostly angry at stuff. Stents etc blah blah . Trying my best to eat wel etc!!! Must be cursed I guess
While the study is on the cutting edge, one study isn't sufficient to base anything on. Let's maintain perspective. What are you suggesting is actionable resulting from this study?
Mike, did you delete your video about Trglycerides/HDL Ratio video? I can't find the long form video, but there is still a short from that video. If you discovered something new, you'd need to delete that short too
My dad is getting a cardiac bypass next month. It’s got me wondering about my cardiovascular status. One day when I get health coverage, I’m gonna get that stuff checked out.
Wanna know your cardiovascular status? Get a CAC score. It will tell you where you stand with calcium buildup in the coronary arteries. The test is about $100 and at many imaging centers you don't need a doctor's order to get it. Easy-peasy. I just paid $99 for mine.
@ … Solid advice. I’ll look into that, good sir
@@swamphawk6227 very limited 'advice,' and also, don't wait for coverage. Get to a functional medicine practitioner, spend the money (if you have an iPhone, eat out, drive a car...it's worth the sacrifice), and get checked out. I will pray for your dad. Mine is also having that (maybe) next month at all but thanks to his nurse daughter thinker guardian angel, I had to save his life from it twice now as the doctors weren't looking at HIM but at labs and I said "he doesn't look good, and he's still flippin bleeding from the cardiac Cath site, and his kidneys are now compromised from the lack of IV fluids you gave him! His liver and kidneys are messed up and it may not be worth it but we'll see after some detoxing I have him on, glutathione, and baths, saunas...to see if he's better able to withstand the bypass. VERY risky surgery. Tell me about it, medical ptsd for me as a nurse and patient and I'm scared for him but stay strong for him. I cry when I get home. My gut doesn't feel good about it....
Just assume that you do have cardiovascular problems and change your lifestyle accordingly. Better diet, more exercise, more sleep, less stress. No need to wait for health coverage.
@@christopherstewart9874 … True. I have a decent lifestyle, but I can do better.
Is this "vulnerable plaque" even plaque at all, or actually blood clots, which some argue are actually the real cause of plaque in the first place. Bearing in mind that real plaque is behind the artery wall and cant be dislodged to cause problems downstream.
until it ruptures and breaks off but even if it just builds up with stable plaque it can still cause angina and ischemia
What would be considered high insulin levels? Both fasted and pospandrial?
The best and only scientific method is to do a Glucose tolerance test with insulin measurements.
This is confirmed in the book by Professor Joseph Kraft, and known as the Kraft Test.
Anything other than that is just not good enough….
Patients are therefore being ‘short changed’ by the medical profession, when they have an Hba1c, or a fasting glucose level to assess their health…
Inexplicabe that they did not report LDL.
Carnivore Diet, very low carbs works for me.
I wonder if LDL wasn’t included because they would need to adjust for who takes a statin and who doesn’t.
Yes very important the LDL, check that out please
Define medium and high insulin please
Is there a benefit for taking a statin if one has had multiple TIA strokes.
Your title is wrong. I think you meant to start with "Insulin and Arterial Plaque".
As my old pharmaceutical representative manager used to say to us: “So Doc, what this means for your patient is…“
As a nurse with a philosophical, theological ethics and a patient, I'd never ever be able to do that line of work - pharma rep.
@ I worked in Pharma for 12 years. It was a straight business at that time - 1990 - 2002. The company I worked for was ethical. No buying of prescriptions and they followed AMA guidelines. Things have changed and the pendulum must come back for the sake of everyone’s health and well-being.
@@Jennifer-gr7hn you can also add a$$h0le to your ‘extraordinary’ academic CV. You made a lot of assumptions about me that are not anywhere near the truth. Get a life.
Yeah im 28 and have had high cortisol and insulin resistance and now I have coronary artery plague 😒
I’m sure they left the LDL out on purpose. I guarantee you if the LDL corresponded with more plaque it would’ve been on there.
Great! Now how do I measure my insulin? 😊
you go to a functional medicine doctor (save yourself time, money, aggravation with the mainstream incase they won't do it) and get it ordered :)
@Jennifer-gr7hn thanks
That makes zero sense they had total cholesterol and HDL but not LDL in an ASCVD study
Name of blood test?
So if we have a higher fasted insulin number if we can get it down it will reduce the damage/plaque in theory potentially? What are those median numbers to aim for? Mine was in low teens so can't be the units of measure showing on this study. I know I need to get it down. I've heard others say 5 is what to aim for?
On glucose meter like diabetics use, glucose should run below 100 about an hour and a half after eating. A1C should be no higher than about 5.5. Dr. Paul Mason states that A1C is not accurate in lots of cases where the person does not eat much carbs because the blood cells don’t turn over as fast as normal. Mike should comment on this.
But it says higher fasting insulin levels, so what does that mean?
And no triglycerides either?
Surprised?
The statin use % goes up because more people on statins are dying
💪🏿💪🏿
💪💪
It seems like you're saying insulin isn't all that bad. Very confusing!
Finally
Still worshipping Ansel Keys?
❤❤❤❤❤❤❤❤❤❤
I wish, pray, hope and long to find a functional ENDOCRINologist......the sickest of patients, are under the 'care' of endocrinologists
Blah blah blah my God, get to the point
You speak way too much and too fast. You have nothing to say, you just read. Not helpful, confusing, zero benefit.
And guess what raises insulin? High fat diets.
LOL. Yeah right.
Sugar raises insulin not fat.
@@Travel_Fanatic24 Exactly right. I'm sure that KenWang was joking.
It's mixing of saturated fat and high glycemic index substances.
@@Mark4Jesus nothing to do with saturated fat
Thanks for the video. Very interesting. Just fyi, ischemic is pronounced /ɪˈskiː.mɪk/ not /I' she mik/ The sch makes a hard k sound like in ski. Thanks again!