Is there indication of actual mitochondrial function actively releasing wavelengths of measurable light, that then is used in other functional ways for mitochondrial bioenergetics?
How might the lab environment, say in artificial light exposure, affect the bioenergetics of the mitochondria in different brain regions, differently than in daylight light energy inputs (or in mouse habitual darkness behavioral environment)? Sure, that is adding a huge number of variables, but I'm concerned about how wavelengths of different light, entering different depths of the brain tissue alter electron transport chain functionally. Does, blue -light dominant lab light already adversely affect mitochondrial energy functionality, at quantum biology levels that ultimately will need careful study?
Mitochondria evolve faster, much faster than their hosts due to a much faster life cycle. They evolve within the constraints of their host cells. There are slow and fast evolutionary forces acting on mitochondrial survival, within the host and survival to propagate to a new developing host. I think you need to switch perspectives faster, host centric? Mitochondria centric? The Krebbs cycle just confused me at college but its only part of the function. There is so much future study in this and I am waiting for it to mainstream.
I wish I could have had this done in different years: after being molested, left Air Force, all the bad things that happened to me. No wonder I left Florida. One day this will be available for living brain tissue to prove damage when stressors are unbearable. Now how do you compensate?
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Such fascinating emerging knowledge of vastly complex system! Great explorations!
Preach Picard!! ❤❤❤
Is there indication of actual mitochondrial function actively releasing wavelengths of measurable light, that then is used in other functional ways for mitochondrial bioenergetics?
How might the lab environment, say in artificial light exposure, affect the bioenergetics of the mitochondria in different brain regions, differently than in daylight light energy inputs (or in mouse habitual darkness behavioral environment)? Sure, that is adding a huge number of variables, but I'm concerned about how wavelengths of different light, entering different depths of the brain tissue alter electron transport chain functionally. Does, blue -light dominant lab light already adversely affect mitochondrial energy functionality, at quantum biology levels that ultimately will need careful study?
Each cell types correlates different mitochondria type. or .. each cell type molds mitochondria to fit in its hosting cell ??
Mitochondria evolve faster, much faster than their hosts due to a much faster life cycle. They evolve within the constraints of their host cells. There are slow and fast evolutionary forces acting on mitochondrial survival, within the host and survival to propagate to a new developing host. I think you need to switch perspectives faster, host centric? Mitochondria centric? The Krebbs cycle just confused me at college but its only part of the function. There is so much future study in this and I am waiting for it to mainstream.
suggest Nick lane lectures on the Kreb cycle
I wish I could have had this done in different years: after being molested, left Air Force, all the bad things that happened to me.
No wonder I left Florida.
One day this will be available for living brain tissue to prove damage when stressors are unbearable.
Now how do you compensate?
You want a stressor: take a Respiratory therapist, chronically stressed by: nursing, codes, short staffed and demanding number of patients.
The First 5 minutes are a waste of time
It’s me ____, now here’s ____ who has accomplished ____ research at ____ university: Hi it’s me ___, now here’s the speaker you came for