Syndrome of Inappropriate Antidiuertic hormone (SIADH) and Diabetes Insipidus (DI). *Video content correction added in comments* For more fun information, visit / tootrn or www.tootRN.com
Another great video breaking complex nursing concepts into simpler smaller easy to understand pieces!!! Thanks so much TootRN! Another awesome video and so simple!!! Thanks Jannah!!!
You are amazeballs!... I WISH you were my instructor during nursing school. THANK YOU for taking the time out of your life to create these videos. So grateful & truly appreciative. :)
** Mistake noted! I need to make a correction in my statement of edema and hypertension: Cerebral edema can be seen, generally not peripheral… this would be a very late and severe sign. In GENERAL, increased ADH causes water retention without extracellular fluid volume expansion (so, generally no peripheral edema and increase in pressure- however, there are some severe cases of rapid onset that this may be present). The water retention causes hyponatremia -> key feature in SIADH. A problem of water metabolism vs. no abnormalities in total body sodium metabolism. Though there is an increase in total body fluid, the easiest way to think of it is: it’s evenly dispersed throughout the compartments -> euvolemic hyponatremia. Sorry for the confusing mistake I noticed today! **
+tootRN, LLC. Great video! I just want to say that the reason that someone is EUVOLEMIC is because RAAS system is still working, meaning aldosterone is still working on the kidney. as they retain lots of fluid, they trigger the RAAS and therefore aldosterone causes the kidneys to respond by getting rid of sodium. this exacerbates the hyponatremia and also keeps them from getting too volume up. Thanks for making these videos, I am a resident MD and the videos are helping me review the material so I can teach medical students! Keep up the good work!
Thank you soooooooo much I spent all this time at school program being confused and unclear about this and you just explained I in 13 minutes wow you truly are amazing ! Thank you again
thank you! clear, less confusing and to the point. Only question is wouldn't you also treat the blood pressure with vasopressin or desmopressin with DI and would you give anti hypertensives with SIADH?
Hi tootRNA very informative video which provided me with better understanding of SIADH and DI.A question I would like to ask you about the syndrome related to DI. I drink less fluid but my fluid output is high. My BP is fine but I don't feel thirsty/ dehydrated. After drinking a hot drink I have to run to the toilet to empty it.
I read that... Lowering sodium diet no more than 3g of sodium per day which helps decrease urine output for DI patients. I don't understand why treat DI with sodium if their sodium is already high. Do you mind explaining this? - Thank you!
Another great video breaking complex nursing concepts into simpler smaller easy to understand pieces!!! Thanks so much TootRN! Another awesome video and so simple!!! Thanks Jannah!!!
Great "toot"ering, but I think your volume is way too low. Other than that, you are awesome. Thanks! Be blessed!
Thank you! Yes- looks like I didn't have the best mic settings when I made these older vids :) Lesson learned :) I appreciate your feedback!
So happy I saw this video before my exam tomorrow! You really explain things so well!! Thanks!!!
You are amazeballs!... I WISH you were my instructor during nursing school. THANK YOU for taking the time out of your life to create these videos. So grateful & truly appreciative. :)
Thanks!! I'm so happy to help!
** Mistake noted! I need to make a correction in my statement of edema and hypertension: Cerebral edema can be seen, generally not peripheral… this would be a very late and severe sign. In GENERAL, increased ADH causes water retention without extracellular fluid volume expansion (so, generally no peripheral edema and increase in pressure- however, there are some severe cases of rapid onset that this may be present). The water retention causes hyponatremia -> key feature in SIADH. A problem of water metabolism vs. no abnormalities in total body sodium metabolism. Though there is an increase in total body fluid, the easiest way to think of it is: it’s evenly dispersed throughout the compartments -> euvolemic hyponatremia. Sorry for the confusing mistake I noticed today! **
+tootRN, LLC.
Great video! I just want to say that the reason that someone is EUVOLEMIC is because RAAS system is still working, meaning aldosterone is still working on the kidney. as they retain lots of fluid, they trigger the RAAS and therefore aldosterone causes the kidneys to respond by getting rid of sodium. this exacerbates the hyponatremia and also keeps them from getting too volume up.
Thanks for making these videos, I am a resident MD and the videos are helping me review the material so I can teach medical students! Keep up the good work!
actually I mis spoke, aldosterone is decreased when there is too much fluid on board, and the absence of aldosterone causes sodium excretion…
+Jamie Santistevan Thanks, Jamie!!
Thanks a lot
This is right the first statement is wrong i suppose )
this video is really hard to hear
Thank you soooooooo much I spent all this time at school program being confused and unclear about this and you just explained I in 13 minutes wow you truly are amazing ! Thank you again
Congrat dear!!! Very amazing presentation. Honestly u are a good lecturer.
Thank you TootRN!
You are awesome =) I just graduated and this was a great review.
Thank you! That really helped me understand it better!
Awesome video !:) thank you
thanks, this video explains so much, I can do well on exam now. Thanks!
Yay! You're very welcome!
thanks for making this easy to understand! good job! looking forward to more videos :)
Thanks! I'm glad you found it helpful!!
thank you! clear, less confusing and to the point. Only question is wouldn't you also treat the blood pressure with vasopressin or desmopressin with DI and would you give anti hypertensives with SIADH?
This was extremely helpful! thank you!!
This was very helpful thanks for sharing.....
Ty ! Awesome explanation
awesome video. Also I love your instagram! What program did you use for the drawing? I'd love to try it out, thanks!
+Joshua Michael Rivera Sketchbook Pro :-)
I love this video, but is there a way you can tweak up the volume? It's a little hard to hear!
For sure!
Your amazing thank you!!
this is awesome, you should submit your videos to the khan academy!
Thanks!!
hi, can't thank you enough for this video on SIADH and Di. Can you please do a review on addisons and cushings. Thank!
thank you so much for the sharing :)
LOVE it....thank you so much.
I loooove your videos!
Thank you, that helped me a bunch :))
Hi tootRNA very informative video which provided me with better understanding of SIADH and DI.A question I would like to ask you about the syndrome related to DI. I drink less fluid but my fluid output is high. My BP is fine but I don't feel thirsty/ dehydrated. After drinking a hot drink I have to run to the toilet to empty it.
Allot of your videos are hard to hear, You explain the content great, it is just hard to hear even with the volume turned all the way up
Why is it that DI can cause increased intercranial pressure?
Why can’t people with diabetes insipidus concentrate urine? Is it a problem in the nephron?
hi, can you talk about "cerebral salt wasting syndrome (CSWS)"?
Adriana Martins Sure, I'll add that to my videos!
GREAT.THANKS
Perfect!
I read that... Lowering sodium diet no more than 3g of sodium per day which helps decrease urine output for DI patients.
I don't understand why treat DI with sodium if their sodium is already high. Do you mind explaining this? - Thank you!
very helpful
I have a question, what if the patient's underlying cause of SIADH is the usage of psychotropic meds? Do you need to quit the meds?
tnx miss toot do you have addison and cushing pleass i retain knwledge frm u :)
Not yet ;-)