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Thank you so much Dale! This is exactly the way I learn best. This video has saved me
Thank you Dale, it is easy to understand, best teaching
Simplest explanation
this was a great explanation thankyouuuuuuu
Isn’t it that lidocaine blocks the sodium channels in Inactivated state , which means depolarisation has already occurred , so there might be just a small deflection rather than such an appreciable change in slope ??
So good!! thank you
Is there anything dietary that can alter sodium channels, intracellular calcium, potassium channels or acetylcholinesterase ?
I thought lidocaine shortened the refractory period? Class Ia lengthen (quinidine), 1b shorten (lidocaine), 1c neutral (flecanide)
I think you are right. What he is describing is more akin to amio (though acting on potassium and not sodium channels)
Thank you so much Dale! This is exactly the way I learn best. This video has saved me
Thank you Dale, it is easy to understand, best teaching
Simplest explanation
this was a great explanation thankyouuuuuuu
Isn’t it that lidocaine blocks the sodium channels in Inactivated state , which means depolarisation has already occurred , so there might be just a small deflection rather than such an appreciable change in slope ??
So good!! thank you
Is there anything dietary that can alter sodium channels, intracellular calcium, potassium channels or acetylcholinesterase ?
I thought lidocaine shortened the refractory period? Class Ia lengthen (quinidine), 1b shorten (lidocaine), 1c neutral (flecanide)
I think you are right. What he is describing is more akin to amio (though acting on potassium and not sodium channels)