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Dale Button
Канада
Добавлен 29 фев 2016
Hyperkalemia Explained: Identifying Shockable Rhythms in Dialysis Patients
In this video, we analyze a complex case of hyperkalemia in a patient with chronic renal failure who has missed multiple dialysis appointments. We explore the patient’s rhythm changes, treatments administered, and the progression from a sine wave pattern to shockable rhythms. We also discuss the roles of medications such as epinephrine and calcium gluconate and their effects on the patient’s cardiac stability, as well as when and why these wide complex tachycardias should be treated with defibrillation.
Timestamps:
0:01 - Introduction to Case Two
Overview of patient background and typical hyperkalemic symptoms in dialysis patients.
0:23 - Rhythm Interpretation: Initial Findings
Analysis of ini...
Timestamps:
0:01 - Introduction to Case Two
Overview of patient background and typical hyperkalemic symptoms in dialysis patients.
0:23 - Rhythm Interpretation: Initial Findings
Analysis of ini...
Просмотров: 1 060
Видео
Hyperkalemia Explained: When to Shock and When to Wait
Просмотров 1,1 тыс.Месяц назад
In this video, we analyze a case of hyperkalemia in a patient found in cardiac arrest, potentially due to crush injury and subsequent rhabdomyolysis. The patient's prolonged immobility likely led to cell damage, causing potassium release into the bloodstream and resulting in hyperkalemia. We examine ECG findings, starting with slow, wide-complex rhythms indicative of hyperkalemia, and discuss t...
Hyperkalemia Explained: Pathophysiology of Sine Waves
Просмотров 100Месяц назад
This video explores the progression of hyperkalemia from T-wave abnormalities to the sine wave pattern seen in severe cases. It explains how elevated extracellular potassium alters the resting membrane potential, leading to disruptions in sodium channel function and conduction velocity. The video reviews the effects on myocardial action potential, including Phase 0 prolongation, QRS widening, a...
Hyperkalemia Explained: Pathophysiology of Peaked T-Waves
Просмотров 2502 месяца назад
Discover how hyperkalemia affects the heart and causes distinctive ECG changes, including peaked T-waves. In this video, we break down the cardiac action potential, focusing on phase three, potassium dynamics, and how elevated serum potassium alters resting membrane potentials. Perfect for paramedics, medical students, and clinicians looking to understand the pathophysiology of hyperkalemia. Le...
REVERT Procedure for SVT: The Science Behind the Modified Valsalva Maneuver
Просмотров 1292 месяца назад
This video dives into the REVERT procedure, or modified Valsalva maneuver, and its effectiveness in treating supraventricular tachycardia (SVT). It explains how to identify SVT on an ECG, the pathophysiology of re-entry circuits, and the physiological mechanisms leveraged by the REVERT procedure to terminate SVT. The discussion covers key steps of the maneuver, why it works better than traditio...
Why Hypoxia Causes Bradycardia in Pediatric Patients: The Cardiac & Autonomic Impact Explained
Просмотров 1522 месяца назад
In this video, we delve into the reasons why hypoxia can lead to bradycardia in pediatric patients, focusing on the unique aspects of pediatric cardiac physiology and autonomic responses. Unlike adults, children rely heavily on heart rate to maintain cardiac output due to their limited stroke volume, which is less adaptable under stress. Hypoxia disrupts essential cellular functions by reducing...
How Hypoxic Vasoconstriction Leads to Pulmonary Hypertension: A Deep Dive into Causes and Effects
Просмотров 1142 месяца назад
In this video, we explore the physiological process of hypoxic vasoconstriction and how it can contribute to pulmonary hypertension. Learn about the short-term adaptive mechanisms and the long-term consequences of this process, including remodeling of vascular smooth muscle and increased pulmonary vascular resistance. Timestamps: 0:01 Introduction to Hypoxic Vasoconstriction 0:15 Purpose of Hyp...
How Calcium Gluconate Stabilizes Heart Cells in Hyperkalemia
Просмотров 4813 месяца назад
In this video, we delve into how calcium gluconate helps manage hyperkalemia by increasing the membrane threshold potential, stabilizing cardiac cells, and reducing the risk of arrhythmias. We’ll discuss the normal action potential in myocardial cells, the changes that occur with hyperkalemia, and why calcium gluconate is effective in managing these changes. Here’s a breakdown of the topics cov...
Accelerated Idioventricular Rhythms and Reperfusion
Просмотров 1323 месяца назад
In this video, we explore reperfusion rhythms in the context of return of spontaneous circulation (ROSC) post-cardiac arrest. We focus on the differentiation between Accelerated Idioventricular Rhythm (AIVR) and Ventricular Tachycardia (VT), examining key features like wide QRS complexes, rate, and P-wave dissociation. By understanding the pathophysiology behind reperfusion rhythms and their be...
Acute Pain Management Strategies and Considerations
Просмотров 15810 месяцев назад
Acute Pain Management Strategies and Considerations
Acetaminophen and NSAID Mechanism of Action for Analgesia
Просмотров 22210 месяцев назад
Acetaminophen and NSAID Mechanism of Action for Analgesia
Stroke Assessment and Los Angeles Motor Score LAMS Pathophysiology
Просмотров 39110 месяцев назад
This video will explain the anatomical function of the MCA as it relates to stroke and the Los Angeles Motor Score.
Pathophysiology and Treatment of COPD
Просмотров 64911 месяцев назад
This video will outline the mechanisms associated with emphysema and chronic bronchitis and front line treatment of COPD exacerbation. 00:00 - Emphysema pathophysiology 07:21 - Chronic bronchitis pathophysiology 11:00 - Oxygen and Ventolin treatment for COPD 13:30 - CPAP treatment for COPD 15:00 - Dexamethasone treatment for COPD
Left Bundle Branch Block Electrophysiology
Просмотров 1,4 тыс.11 месяцев назад
This video outlines the electrophysiological changes seen on a 12-lead ECG for left bundle branch block, and the rationale for these changes.
High Yield Assessment for Cardiac Ischemia
Просмотров 392Год назад
High Yield Assessment for Cardiac Ischemia
Oxytocin Pharmacology and Role in Postpartum Hemorrhage
Просмотров 3,1 тыс.Год назад
Oxytocin Pharmacology and Role in Postpartum Hemorrhage
Pathophysiology of Postpartum Hemorrhage
Просмотров 7 тыс.Год назад
Pathophysiology of Postpartum Hemorrhage
Pathophysiological Risks of Sodium Bicarbonate Administration in Diabetic Ketoacidosis
Просмотров 1,3 тыс.Год назад
Pathophysiological Risks of Sodium Bicarbonate Administration in Diabetic Ketoacidosis
Pathophysiology of Diabetic Ketoacidosis Flowchart
Просмотров 3,1 тыс.Год назад
Pathophysiology of Diabetic Ketoacidosis Flowchart
Pathophysiology of Diabetic Ketoacidosis
Просмотров 2,2 тыс.Год назад
Pathophysiology of Diabetic Ketoacidosis
Pharmacological Treatment of Hyperkalemia
Просмотров 6 тыс.Год назад
Pharmacological Treatment of Hyperkalemia
Eosinophil Infiltration and Dynamic Hyperinflation in Asthma
Просмотров 428Год назад
Eosinophil Infiltration and Dynamic Hyperinflation in Asthma
Symptomatic Bradycardia - Prehospital Management
Просмотров 5852 года назад
Symptomatic Bradycardia - Prehospital Management
Left Main Coronary Artery Occlusion - Pathophysiology
Просмотров 1,1 тыс.2 года назад
Left Main Coronary Artery Occlusion - Pathophysiology
Excellent explanation! Thanks a lot this really helps!
Only video on youtube that clearly explains this concept and not just "calcium changes the threshold potential" Thank you!
Brilliant!!
Good job sir, thanks
here is a summary that i found for those who didnt understand how ca+ rises the treashold : Threshold potential: largely determined by the voltage gated Na channel. In short, when membrane potential reaches the threshould, many voltage gated Na+ channels open and lead to Na+ influx and subsequent action potentials. Ca2+ ion alters the property of these voltage gated Na+ channels and make their "gating voltage" higher, aka less negative. By doing so, these Na+ channels open only at a higher voltage. Thus the threshold is raised and the difference between the resting potential and the threshold is increased. In other words, cell become less excitable.
thanks the only video on yt that explains this concept
But sir you have made mentioned of increased in fluid reabsorption from the kidney in CVS changes but at The renal changes you said it increases GFR which lead to increased in urination.
"The impact of hyperkalemia on the rectifier potassium channels " cleared my doubt ❤
Great class! I was looking for a class like this since a while, thanks a lot
thanks for making this video, this is an excellent presentation.
Thank you for the video, very helpful! Can you give some examples of what could cause a unidirectional block in the fast pathway?
this is so amazing ! thank you professor
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Thanks, this coupled with this video ruclips.net/video/vSrW4jg8Zq8/видео.html Helped me understand both ST depression and T wave inversion completely, for these videos complement each other. Thank you for a clear explanation
I'm bradycardia without symptoms my resting heart rate is the low fifty's to the high forty's my doctor says I'm doing fine
Great explanation, thank you so much ❤
Thank you! Very clear concept
You are just amazing ❤ thank you
Thank you. Your videos are very helpful
Sir, your videos are brilliant!!! The way you explain concepts makes it crystal clear.. Please please upload more videos on STEP 1 USMLE content. Thank you
Is this c section or nsd?
Thank you very much sir🫡🫡
Could you cover more ecg’s and videos regarding that? Thank you!
Great video! Love reviewing these topics you make.
What books do u use?
hey Dale thanks for the video It seems you are starting to make video again, I encourage you to keep going. your videos are actually really helpful. more power to you man 💪🥳
could you explain how calcium gluconate increases the threshold potential....I found this part of the video very confusing because you said calcium gluconate increases the membrane potential above the RMP? Then you said calcium gluconate increases the RMP?? Then back to calcium gluconate increases the threshold potential. All around the 7 to 7:30 mark.
You just did it less than30 minutes and it's clear thank you so much🎉🎉❤
Thank you for explaining this. Learning Reperfusion beats in Nursing School and I appreciate how you described it.
Thank you so much Dale! This is exactly the way I learn best. This video has saved me
This is such a well-detailed explanation. Thank you so much!
This video is incredible. You summed up multiple medical school topics in this description. Thank you!
Your explanation of the respiratory system is truly awesome. None of the other videos on youtube come close to how well you teach. Hats off!😊
Hello sir Isn't PR-interval implies current in AV node?
the best explanation; thank you sir !!
Super detailed!
Hello Dale, thank you for your very informative and well illustrative video. I learned a lot. Was wondering if you might look at an EKG of mine. I have a 1 mm negative post positive T wave, it is very brief following a quick return to baseline. It is on lead 1 from a at home ekg/ecg monitor. So only 1 lead (a wearable). Wondering if you might be willing to take a look at it and tell me if you think it is normal, artifact, or early signs of ischemia? Thanks T
Amazing! Thanks so much for this
By far the best video on AVNRT. Thank you
Very helpful video. Thankyou
Thank you ❤
Hyperthermia and hyperpyrexia ? Are these both same ? If no whats difference ? Why are these mechanisms happening ? Thankyou. ( thankyou for great service )
Pardon me if any rudeness in my comment ( am poor in grammer )
Thanks a lot
I have been struggling with this for a while and this is the best explanation I've come across, thank you so much!
Isn’t it that lidocaine blocks the sodium channels in Inactivated state , which means depolarisation has already occurred , so there might be just a small deflection rather than such an appreciable change in slope ??
Best video EVER!!!
Great video I just have one question. Could you explain why the speed of repolarisation i.e epicardial tissue repolarising before the subepicardial tissue dictates the net flow? Thanks
I recently had an experience with heat exhaustion and somehow stumbled upon your video - I've had a lot of teachers/mentors in my life and you are an amazing teacher - wow!
Will atropine work for afib with slow ventricular response?