Carnivore here. My V.A. doctors mention statins and I say No thanks. They even sent me some w/o asking me. My HDL:Triglyceride ratio is 1.3. I said If you are concerned, give me a CAC test. I used to eat low-fat and plant-based like bran flakes and almond milk, thinking they are healthy. But plants were killing me. Destroyed my gut. Low carb has turned around many issues and I lost weight
Congratulations! I went through that as well. Low-fat for years, oatmeal every day, beans, legumes, etc. When my total cholesterol went down to 124 the doc was so proud I listened to him. Then I came to my senses. Now I don't trust Big Pharma, the mass health care system OR my doc. I have to keep saying NO to statins. I have an appointment with an endocrinologist who is a doctor or osteopathy (DO, not MD), and they are said to have a more holistic approach. Let's see ...
My Mum had very high cholesterol,when they first started testing her in her 50’s,her doctor was always alarmed telling her to go low fat definitely don’t eat eggs,luckily she didn’t listen and lived to 92.
Had to send the lecture to my friends. There are many and very good presentations on Statins and LDL, but Dr. David Diamond or Triple D like his friends are calling him, is killing it. :D
Thank you. Very clear and easy to understand. I am a LMHR and have noticed since becoming a carnivore that my 14 grandchildren have been unable to share their viruses with me. I will keep exercising and eating in this manner for healthy longevity. My stomach feels so much better. (I'm 60 and work full-time as a minister of a church).
😂 I love how you phrased that! “Unable to share their viruses with me”. I have been blessed with similar results this fall as well. I’m not full carnivore, but I try my best and working towards it.
The reason they love statins is because it's a drug that "works as intended", which isn't that common with drugs for chronic illness. The problem is the intended use is wrong, and the side-effects are horrifying (my grandmother died slowly from these, muscle atrophy and effectively disabled, when she was fine prior).
@@williambelmont9601 The same happened to our father: Before he was put on statins, he was active and strong. The last few years of his life, his muscles were wasting away, and he was in constant pain. He also was increasingly struggling with concentration; for example falling asleep while in company, etc. Finally his lungs were too weak, and failed him, after undergoing anaesthesia. All cells in our body need cholesterol to heal&repair, and the older we get, even moreso... How can this not be obvious?
Please focus your next talk in making fun of Peter Attia and his exact arguments against high LDL. He is very influential and his book is being read by millions. So it is very important for all of us to know your thoughts in: -What he has read an referenced, -What he is interpreting, -What he got right, -What he got wrong, -Science that he is ignoring -How should we interpret his references -The correct argument about LDL But focus on him and his exact arguments. Indeed title the talk with his name: "What Peter Attia is getting wrong" Thanks. I hope to see this soon. Otherwise we all may think that Attia is somehow right relying on his interpretation of Mendelian randomisation studies.
After following most of the Keto diet lifestyle, I have transformed my metabolic status to very healthy, but since I had stents I had to visit my cardiologist last week for a stress test. He wasn't interested in my weight loss, my reversal of diabetes, my Keto transformation, my previous 7 minute stress test against this 14 minute stress test, all he wanted to do was get me back on Statins because of high LDL (of course my Triglycerides were very low). I asked him about any test in Australia for LDL particle size testing (because of my Stents I cannot have a CAC) and he didn't know it, when I questioned the benefits of Statins he told me about the Jupiter study. I then decided to end the conversation and not see him anymore. Unfortunate because he seems a very intelligent professional who has probably spent years of his life training to be a cardiologist.
That' so sad. I am a medic, a consultant psychiatrist, and I have to admit it took me to develop diabetes before the scales fell off my eyes. Prior to that, I believed I knew everything about diabetes. While it's not my place to offer my patients nutritional advice, they stare at me in disbelief anytime I quickly broach the idea of improving metabolic health by going on a low carb/keto/carnivore diet. The population, especially here in the UK, have been hoodwinked and the absence of critical thinking amongst people, including the so called experts, is frightening bizarre.
Over the last 12 months I have been told by FIVE different cardiologists that I am not a candidate for a Coronary CT Angiogram (CCTA) because I have a high BMI, a high CAC score and a LAD stent in place. Through my own research, I have discovered that in the year 2023 there is a "state of the art" CCTA that CAN BE DONE to monitor my known CAD. That CCTA is available through the CLEERLY company. Please look up and review multiple RUclips videos presented by the CLEERLY radiology group. We patients with CAD must stick together and look out for one another because most cardiologists are clueless when it comes to the changing principles/theories of atherosclerosis and lipidology.
@@naijaman6639I sincerely hope that you, as a psychiatrist, have checked out dr. Georgia Ede and dr. Chris Palmer, and the Metabolic Mind channel on youtube.
Thank you! I took crestor and stopped due to severe side effects. The cardiologist did not record the side effects in my chart, so you know they were not reported to any overlords. I was told all the side effects were 'nocebos.'
Irritating, isn't it?? Mine experience wasn't with a statin, but a change from my name-brand BP medication to the generic version; after using the generic for less than two months, I was waking up in the middle of the night, feeling like my heart was going to explode if I didn't sit up. Slept sitting up for several days while I tried to figure out why this was happening, and realized the only thing that changed had been that med. Still had about 8 days of the name-brand left, so took it for two days---slept soundly. Went back to the generic, and again, the sudden waking with my heart pounding and me gasping for breath. My cardiologist put a Holter monitor on me for 48 hours, so I thought I'd take the brand-name one day, the generic the next day, to see if there was any difference. My cardiologist was shocked when the results came back---he showed me the difference and said "No wonder you were waking up." Put me back on the name-brand. But I'm sure he didn't report the side effect to the drug company! Makes you wonder how the statistics would be on ALL drugs if side effects were actually and accurately reported. Wishing you good health, Robyn3349!
It's not just the side effects if statins, it's that they have no significant benefits. Many are given a false sense of security and fail to make the changes in diet and lifestyle that do make a difference.
@@jerseyjim9092 i’ve been on a keto diet for at least three years. Nothing changes I don’t lose weight I don’t lose belly fat and no I’m not doing it wrong. My LDL has been high for almost two years. Previous 315 now Currently 288, from red yeast rice. When I started getting so extremely fatigued I began to wonder if the doctors are right about my high LDL. All my other numbers look good. As pain in my chest mostly where the top of my lungs were started to bother me. When I couldn’t mow the lawn more than 10 minutes without having to sit down. When vacuuming one room in the house exhausted me, When I could only clean the bathroom mirror, countertop and sink without being utterly exhausted I gave in to the Crestor. It’s been two weeks and yesterday not only was I able to scrub down my entire bathroom, (rested) then I vacuumed and double shampooed the spare bedroom. Every night before bed I make sure I get all the dishes in the dishwasher and wipe down the counters. It’s been a force to reckon with let me tell you but last night when I did it I was a little tired but I didn’t have to just come utterly using every ounce of energy to get it done like before. If I have any side effects I’ll immediately stop and use the red yeast rice. I am also taking 100mg Co Q10 twice a day because I heard a lot of the side effects, like sore muscles, are because the statins deplete the CoQ10. Fingers Crossed.
10:27 Mic Drop!! For the past 4 years I've been tracking my cholesterol more closely, my TG consistently quite low, HDL very good and within the 'range' HDL/TG ratio perfect, LDL 230-240. Not worried in the slightest, in fact looking forward to my 70's lol.
@@lvenable07 a widow maker builds over time and it may be helpful to consider his dietary patterns of the past and present - hypertension causes damage to the artery walls, and then cholesterol comes as part of the repair squad. ♥️♥️ wishing hubby a quick recovery ❤️🩹
@@lvenable07 Sorry to hear your hubby experiencing this event - best wishes your way for healing ♥️ A widowmaker becomes same over extended period of arterial assault - likely from years of hypertension, metabolic dysfunction, which causes injury to the inside of the arterial walls and which then prompts the body to try and heal using cholesterol as a patch. In discussions with my GP, and making comment on the 'commentary' provided by the lab agencies as part of blood work results, I asked 'well, if THIS (high cholesterol) supposedly DRIVES / CREATES arterial damage, is there a way to check for arterial damage?' - and with the advent of the CAC, there is now! So I paid for a CAC ($240/Australia) which came back as '0' evidence of arterial plaque formation already in place. This result, coupled with the work of folks questionning the heart hypothesis has given me a level of confidence in my decision to resist any form of medication to 'treat' cholesterol. Could I be wrong? Of course, however I feel so healthy eating a low carb/carnivore diet that that is good enough for me.
Anyone else experienced a rise in total cholesterol when fasting longer then 12 hours. My total cholesterol is 300 if fasted 12 hours and 390 if fasted 18 hours.
The dutch professor Frans Russel has discovered why Statins cause so many side effects. It concerns the substance Lactone that is involved in Statins. Research shows that the lactone form inhibits an essential part of energy production in those mitochondria. Professor Russel: “It concerns the respiratory chain in the mitochondria, the chain in which the cell generates energy by consuming oxygen. It is severely hindered. We also show that the statins accumulate in the muscle during treatment. The stacking of Statins further contributes to the inhibition of the optimal functioning of the mitochondria.” Although in a few cases it can lead to serious muscle breakdown, by far the largest group (twenty percent of all users) suffers from muscle pain without muscle damage. Yet this muscle pain hinders daily functioning.
4:35 Dr. Dave Adlersberg FH Familial Hypercholesterolemia (FH) 15:42, 16:02 clotting factors: fibrinogen and factor 8, 16:51 FH people have viscous blood, thicker blood.
Superb presentation! Very clear explanation of the absolute vs. relative risk reduction - the statistical chicanery used in assessing pharmaceutical effectiveness. So much valuable information on what to look for in the blood work that truly denotes risk and disease. Even how stress increases fibrinogen and how that increases your risk for clotting/stokes. Loved every minute of this talk. Thank you Low Carb Conference for posting!
I wonder what the best approach is to get my doctors to watch this. As a Type 1 Diabetic who is on the Keto diet they see the higher cholesterol readings and they ALWAYS tell me I should be on a statin. It's infuriating and I wonder why I even bother going to see them.
The medical profession appears to have been ignoring a lot of elephants in the room while the fixate on orthodox opinion. Who in the medical profession could ever imagine that healthier people would be more healthy.?
I would be curious to know of the people who are dying with fh are actually heavy alcohol users? So many in my family the death certificate says atherosclerosis but really it was alcohol.
What counts as "high HDL" , or high-enough HDL to not need the statin? What was the cut-off in the studies he mentioned near the end ... what was the cut-off separating the "high HDL" group from the "low HDL" group. And if high TG and low HDL signifies metabolic dysfunction, what does low TG with low HDL signify?
My doctor said since I refuse to take a statin there's no reason for him to even order a lipid panel blood test. I said "OK". But later I realized, wow! That was a tacit admission that he ONLY looks for opportunities to sell drugs, not for problems in my health.
@@lentilpaff7864 True. But he doesn't want to see me and I don't want to see him, or any doctor, unless I have a good reason. If I can rid myself of a need to take levothyroxine, I wouldn't even have to go back for an annual TSH test. I'm now taking iodine and the companion supplements, so we'll see.
@@lentilpaff7864 True. But we don't want to see each other, or any doctor, unless I have a good reason. If I can rid myself of a need to take T4, I wouldn't even have to go back for an annual TSH test. I'm now taking [element I] and the companion supplements, so we'll see. (Restoring wrongfully deleted post.)
So sad D.Diamond did not mention that familial hypercholesterolemia group is way below 1% of population, so most talk is about FH, which is mostly irrelevant to 99% of ppl.
Around 22/23rd min...he said small dense ldl is caused by insulin resistance. Is it the only, main cause? I know I'm *not insulin resistant... But I still worry about the whole apoB/small dense ldl thingy abit. If any, what other causes are there for small dense ldl /apoB? I'm good with other biometrics... ApoB came up back mid 120's...which isn't too bad imo. But according to Attia, it should be like 75😅
@@dx398 yea, I double down on not worrying about ldl/apoB. That said, it has gone down a bit around 90...ldl 120's probably. Usually, my apoB is 70% proxy of my ldl-C fwiw. I hedge my ldl a bit in the 120/130's ranges, imo it's optimal there about...
At 8 minutes, he states that these results were “independent of any cholesterol treatment”. Does this mean that the individuals in the study had never taken statins or pre-statin medication in their lives?
This presentation made it all more clear why one study I read showed a very significant absolute difference in one study (17 %) but David Diamond was able to parse out the results identifying how knowing whether someone would have high triglycerides and low HDL WOULD def impacted while those with low triglycerides and high HDL wouldn’t have a significantly better outcome on statins. Thanks, I was trying to figure out how interpretations of studies could be so very different.
Not all cardiologists would be alarmed. Mine told me, quote, "triglycerides are for diabetes, nothing to do with your heart". End quote. I don't have a lot of faith in cardiologists, seems their ticket can come out of a cereal packet with their breakfast 😞
So what about Chris McCaskill's takedown on Plant Chompers? "You can tell how little they've read about the [Seven Countries] study" - why misrepresent the data so badly Dr. Diamond?
So he mainly talks about LDL but then talks about the JUPITER trial wherein participants had LDL below 130 mg/dl. And when he talks about absolute risk reductions percentages he doesn't talk about the timespan. Seems rather sloppy. I get the impression he doesn't really master the material as much as he likes to think or likes us to think.
![I.N "HALLUCINATION" | [Stray Kids : SKZ-PLAYER]](http://i.ytimg.com/vi/n5B5q1Hwt_U/mqdefault.jpg)
Carnivore here. My V.A. doctors mention statins and I say No thanks. They even sent me some w/o asking me. My HDL:Triglyceride ratio is 1.3. I said If you are concerned, give me a CAC test. I used to eat low-fat and plant-based like bran flakes and almond milk, thinking they are healthy. But plants were killing me. Destroyed my gut. Low carb has turned around many issues and I lost weight
Congratulations!
Congratulations! I went through that as well. Low-fat for years, oatmeal every day, beans, legumes, etc. When my total cholesterol went down to 124 the doc was so proud I listened to him. Then I came to my senses. Now I don't trust Big Pharma, the mass health care system OR my doc. I have to keep saying NO to statins. I have an appointment with an endocrinologist who is a doctor or osteopathy (DO, not MD), and they are said to have a more holistic approach. Let's see ...
My Mum had very high cholesterol,when they first started testing her in her 50’s,her doctor was always alarmed telling her to go low fat definitely don’t eat eggs,luckily she didn’t listen and lived to 92.
❤
What do you consider very high? Because some doctors will consider 140 very high, what about 350?
Had to send the lecture to my friends. There are many and very good presentations on Statins and LDL, but Dr. David Diamond or Triple D like his friends are calling him, is killing it. :D
Excellent presentation... Having high LDL is healthy!
Thank you. Very clear and easy to understand. I am a LMHR and have noticed since becoming a carnivore that my 14 grandchildren have been unable to share their viruses with me. I will keep exercising and eating in this manner for healthy longevity. My stomach feels so much better. (I'm 60 and work full-time as a minister of a church).
He gets a complete de dunk here. He is clearly unable to interpret data!
ruclips.net/video/nnyzuY-Xwe4/видео.htmlsi=aHwdu-JeCAk6uED2
Then you’ll be interested to know that Dr Diamond lies! ruclips.net/video/nnyzuY-Xwe4/видео.htmlsi=0BXfHPeQsBjuUI-H
😂 I love how you phrased that! “Unable to share their viruses with me”. I have been blessed with similar results this fall as well. I’m not full carnivore, but I try my best and working towards it.
@@saveonbuildingall the best in your health journey.
This is absolutely insane the whole world got the wool pulled over their eyes on LDL and statins...
The reason they love statins is because it's a drug that "works as intended", which isn't that common with drugs for chronic illness. The problem is the intended use is wrong, and the side-effects are horrifying (my grandmother died slowly from these, muscle atrophy and effectively disabled, when she was fine prior).
@@williambelmont9601
The same happened to our father: Before he was put on statins, he was active and strong. The last few years of his life, his muscles were wasting away, and he was in constant pain. He also was increasingly struggling with concentration; for example falling asleep while in company, etc. Finally his lungs were too weak, and failed him, after undergoing anaesthesia. All cells in our body need cholesterol to heal&repair, and the older we get, even moreso... How can this not be obvious?
Cholesterol meds cause dementia. Its a slow dying disease
The whole world also got the wool pulled over on Covid and vaccines. Big Pharma & governments are not our friends.
Please focus your next talk in making fun of Peter Attia and his exact arguments against high LDL.
He is very influential and his book is being read by millions. So it is very important for all of us to know your thoughts in:
-What he has read an referenced,
-What he is interpreting,
-What he got right,
-What he got wrong,
-Science that he is ignoring
-How should we interpret his references
-The correct argument about LDL
But focus on him and his exact arguments. Indeed title the talk with his name: "What Peter Attia is getting wrong"
Thanks. I hope to see this soon. Otherwise we all may think that Attia is somehow right relying on his interpretation of Mendelian randomisation studies.
Agree. Peter and Tom Dayspring are talking about the crinkle repeats and it’s confusing. Maybe statins benefit a high ApoB male.
I love David Diamond!! Such a great speaker!
After following most of the Keto diet lifestyle, I have transformed my metabolic status to very healthy, but since I had stents I had to visit my cardiologist last week for a stress test. He wasn't interested in my weight loss, my reversal of diabetes, my Keto transformation, my previous 7 minute stress test against this 14 minute stress test, all he wanted to do was get me back on Statins because of high LDL (of course my Triglycerides were very low). I asked him about any test in Australia for LDL particle size testing (because of my Stents I cannot have a CAC) and he didn't know it, when I questioned the benefits of Statins he told me about the Jupiter study. I then decided to end the conversation and not see him anymore. Unfortunate because he seems a very intelligent professional who has probably spent years of his life training to be a cardiologist.
That' so sad. I am a medic, a consultant psychiatrist, and I have to admit it took me to develop diabetes before the scales fell off my eyes. Prior to that, I believed I knew everything about diabetes. While it's not my place to offer my patients nutritional advice, they stare at me in disbelief anytime I quickly broach the idea of improving metabolic health by going on a low carb/keto/carnivore diet. The population, especially here in the UK, have been hoodwinked and the absence of critical thinking amongst people, including the so called experts, is frightening bizarre.
Over the last 12 months I have been told by FIVE different cardiologists that I am not a candidate for a Coronary CT Angiogram (CCTA) because I have a high BMI, a high CAC score and a LAD stent in place. Through my own research, I have discovered that in the year 2023 there is a "state of the art" CCTA that CAN BE DONE to monitor my known CAD. That CCTA is available through the CLEERLY company. Please look up and review multiple RUclips videos presented by the CLEERLY radiology group. We patients with CAD must stick together and look out for one another because most cardiologists are clueless when it comes to the changing principles/theories of atherosclerosis and lipidology.
@@naijaman6639I sincerely hope that you, as a psychiatrist, have checked out dr. Georgia Ede and dr. Chris Palmer, and the Metabolic Mind channel on youtube.
I find it fascinating to know that this reactions are so widespread in UK. US. CANADA. OZ. Why.
Have you considered ordering a partical size test and paying out of pocket for it. I did and found it very reassuring.
I watch low carb videos every day. This is a great one.
Thank you! I took crestor and stopped due to severe side effects. The cardiologist did not record the side effects in my chart, so you know they were not reported to any overlords. I was told all the side effects were 'nocebos.'
Irritating, isn't it?? Mine experience wasn't with a statin, but a change from my name-brand BP medication to the generic version; after using the generic for less than two months, I was waking up in the middle of the night, feeling like my heart was going to explode if I didn't sit up. Slept sitting up for several days while I tried to figure out why this was happening, and realized the only thing that changed had been that med. Still had about 8 days of the name-brand left, so took it for two days---slept soundly. Went back to the generic, and again, the sudden waking with my heart pounding and me gasping for breath. My cardiologist put a Holter monitor on me for 48 hours, so I thought I'd take the brand-name one day, the generic the next day, to see if there was any difference. My cardiologist was shocked when the results came back---he showed me the difference and said "No wonder you were waking up." Put me back on the name-brand. But I'm sure he didn't report the side effect to the drug company! Makes you wonder how the statistics would be on ALL drugs if side effects were actually and accurately reported. Wishing you good health, Robyn3349!
It's not just the side effects if statins, it's that they have no significant benefits. Many are given a false sense of security and fail to make the changes in diet and lifestyle that do make a difference.
www.fda.gov/safety/medwatch-fda-safety-information-and-adverse-event-reporting-program/reporting-serious-problems-fda#:~:text=If%20you%20need%20information%20or,into%20MedWatch%20or%20for%20instructions.
@@jerseyjim9092 i’ve been on a keto diet for at least three years. Nothing changes I don’t lose weight I don’t lose belly fat and no I’m not doing it wrong. My LDL has been high for almost two years. Previous 315 now Currently 288, from red yeast rice.
When I started getting so extremely fatigued I began to wonder if the doctors are right about my high LDL. All my other numbers look good. As pain in my chest mostly where the top of my lungs were started to bother me. When I couldn’t mow the lawn more than 10 minutes without having to sit down. When vacuuming one room in the house exhausted me, When I could only clean the bathroom mirror, countertop and sink without being utterly exhausted I gave in to the Crestor.
It’s been two weeks and yesterday not only was I able to scrub down my entire bathroom, (rested) then I vacuumed and double shampooed the spare bedroom.
Every night before bed I make sure I get all the dishes in the dishwasher and wipe down the counters. It’s been a force to reckon with let me tell you but last night when I did it I was a little tired but I didn’t have to just come utterly using every ounce of energy to get it done like before.
If I have any side effects I’ll immediately stop and use the red yeast rice.
I am also taking 100mg Co Q10 twice a day because I heard a lot of the side effects, like sore muscles, are because the statins deplete the CoQ10.
Fingers Crossed.
10:27 Mic Drop!! For the past 4 years I've been tracking my cholesterol more closely, my TG consistently quite low, HDL very good and within the 'range' HDL/TG ratio perfect, LDL 230-240. Not worried in the slightest, in fact looking forward to my 70's lol.
My husband had those same numbers. Ratio exactly 1. Had a widowmaker last week. Just don't know what to do anymore.
@@lvenable07 a widow maker builds over time and it may be helpful to consider his dietary patterns of the past and present - hypertension causes damage to the artery walls, and then cholesterol comes as part of the repair squad. ♥️♥️ wishing hubby a quick recovery ❤️🩹
@@lvenable07 Sorry to hear your hubby experiencing this event - best wishes your way for healing ♥️ A widowmaker becomes same over extended period of arterial assault - likely from years of hypertension, metabolic dysfunction, which causes injury to the inside of the arterial walls and which then prompts the body to try and heal using cholesterol as a patch. In discussions with my GP, and making comment on the 'commentary' provided by the lab agencies as part of blood work results, I asked 'well, if THIS (high cholesterol) supposedly DRIVES / CREATES arterial damage, is there a way to check for arterial damage?' - and with the advent of the CAC, there is now! So I paid for a CAC ($240/Australia) which came back as '0' evidence of arterial plaque formation already in place. This result, coupled with the work of folks questionning the heart hypothesis has given me a level of confidence in my decision to resist any form of medication to 'treat' cholesterol. Could I be wrong? Of course, however I feel so healthy eating a low carb/carnivore diet that that is good enough for me.
You can't even begin to explain this to family and friends. It's frightening.
Fabulous. This is the paper we have been waiting for and to present to your doctor when he/she recommends statins. My LDL is 8.5 or 328.
Anyone else experienced a rise in total cholesterol when fasting longer then 12 hours. My total cholesterol is 300 if fasted 12 hours and 390 if fasted 18 hours.
Yeah Feldman explains this. I ran two tests on myself as well to confirm it 2-3 years ago.
Yes. It goes up because you are measuring LDL and HDL. LDL is lipoprotien which carries triglycerides which needs to be mobilized when youre fasting.
Fantastic presentation. You are spot on. I’ve come to my same conclusion on my own journey to metabolic health and in treating thousands of patients.
He is misinforming ruclips.net/video/nnyzuY-Xwe4/видео.htmlsi=aHwdu-JeCAk6uED2
The dutch professor Frans Russel has discovered why Statins cause so many side effects. It concerns the substance Lactone that is involved in Statins. Research shows that the lactone form inhibits an essential part of energy production in those mitochondria. Professor Russel: “It concerns the respiratory chain in the mitochondria, the chain in which the cell generates energy by consuming oxygen. It is severely hindered. We also show that the statins accumulate in the muscle during treatment. The stacking of Statins further contributes to the inhibition of the optimal functioning of the mitochondria.” Although in a few cases it can lead to serious muscle breakdown, by far the largest group (twenty percent of all users) suffers from muscle pain without muscle damage. Yet this muscle pain hinders daily functioning.
4:35 Dr. Dave Adlersberg FH
Familial Hypercholesterolemia (FH)
15:42, 16:02 clotting factors: fibrinogen and factor 8, 16:51 FH people have viscous blood, thicker blood.
Keto three years and off statin 1 year. 67 y/o and healthier than when I retired from medicine in 2019!
Keto three years, 65 yo, now on statin because of chest pain and extreme fatigue!
@@KAT-dg6el I am not sure a statin will be of benefit. The statins have numerous side effects and will not help chest pain and fatigue.
Very well done! Sharing the heck out of this one. 😁👍
Thank you for posting this!
Thank you for this fab talk. I can now clearly show my doc why I will not take a statin.
Superb presentation! Very clear explanation of the absolute vs. relative risk reduction - the statistical chicanery used in assessing pharmaceutical effectiveness. So much valuable information on what to look for in the blood work that truly denotes risk and disease. Even how stress increases fibrinogen and how that increases your risk for clotting/stokes. Loved every minute of this talk. Thank you Low Carb Conference for posting!
I wonder what the best approach is to get my doctors to watch this. As a Type 1 Diabetic who is on the Keto diet they see the higher cholesterol readings and they ALWAYS tell me I should be on a statin. It's infuriating and I wonder why I even bother going to see them.
Hopefully your Dr will already know that Dr Diamond lies - proof ruclips.net/video/nnyzuY-Xwe4/видео.htmlsi=0BXfHPeQsBjuUI-H
Thank you. I'm off statin for the first time in 25 years. My doctor is testing me in 30 days.
And how is your LDL relevant to anything? Higher LDL lives longer. Low LDL still has heart attacks.
I wouldn’t even bother with the test..
The only thing not addressed is the statin argument that statins will prevents a thrombus/clot that breaks away from a lipid plaque.
Really interesting. Thank you for posting this.
The medical profession appears to have been ignoring a lot of elephants in the room while the fixate on orthodox opinion. Who in the medical profession could ever imagine that healthier people would be more healthy.?
I would be curious to know of the people who are dying with fh are actually heavy alcohol users? So many in my family the death certificate says atherosclerosis but really it was alcohol.
Thank you.
Thank you so much.
Wonderful ❤
What counts as "high HDL" , or high-enough HDL to not need the statin? What was the cut-off in the studies he mentioned near the end ... what was the cut-off separating the "high HDL" group from the "low HDL" group. And if high TG and low HDL signifies metabolic dysfunction, what does low TG with low HDL signify?
What is a decent low cost genetic test kit for fibrinogen... or clotting factor risk?
From Malaysia ❤.. thanks Dr..
37:00 This is huge peeps!
This is all misinformation as proved here ruclips.net/video/nnyzuY-Xwe4/видео.htmlsi=0BXfHPeQsBjuUI-H
My doctor said since I refuse to take a statin there's no reason for him to even order a lipid panel blood test. I said "OK". But later I realized, wow! That was a tacit admission that he ONLY looks for opportunities to sell drugs, not for problems in my health.
You should fire your doctor. That's not ok.
@@lentilpaff7864 True. But he doesn't want to see me and I don't want to see him, or any doctor, unless I have a good reason. If I can rid myself of a need to take levothyroxine, I wouldn't even have to go back for an annual TSH test. I'm now taking iodine and the companion supplements, so we'll see.
@@lentilpaff7864 True. But we don't want to see each other, or any doctor, unless I have a good reason. If I can rid myself of a need to take T4, I wouldn't even have to go back for an annual TSH test. I'm now taking [element I] and the companion supplements, so we'll see.
(Restoring wrongfully deleted post.)
We also need cholesterol for our hearts to function. I know of a woman whose cholesterol was very low and it affected her thyroid too. She almost died
When someone with FH takes a statin to lower LDL, do they lose the protective benefit against cancer and other causes?
When I finally just realized that I have been lied to all my life, I just remember the being a doctor was actually built-up on quackery... lol
If they can prove that high cholesteral is bad then sure, add guide lines. We have been waiting for decades.
They already made the guidelines we followed based on bad data and misinterpretation!
So sad D.Diamond did not mention that familial hypercholesterolemia group is way below 1% of population, so most talk is about FH, which is mostly irrelevant to 99% of ppl.
Well, his discussion regarding FH (the group that has the most scary studies regarding it) should reassure you even more when it comes to the 99%
@@bonnieo8 in a way, you can say that.. yet, in general ppl w genetic defects/deviations are not good to compare to normal ppl.
Attia defines FH as anyone with an LDL over 190. That's much more than 1%.
Relative risk. Always part of the percentages.
Around 22/23rd min...he said small dense ldl is caused by insulin resistance. Is it the only, main cause? I know I'm *not insulin resistant... But I still worry about the whole apoB/small dense ldl thingy abit. If any, what other causes are there for small dense ldl /apoB? I'm good with other biometrics... ApoB came up back mid 120's...which isn't too bad imo. But according to Attia, it should be like 75😅
Same here, AboB at 130 and no insulin resistance or inflammation.
@@dx398 yea, I double down on not worrying about ldl/apoB. That said, it has gone down a bit around 90...ldl 120's probably. Usually, my apoB is 70% proxy of my ldl-C fwiw. I hedge my ldl a bit in the 120/130's ranges, imo it's optimal there about...
At 8 minutes, he states that these results were “independent of any cholesterol treatment”.
Does this mean that the individuals in the study had never taken statins or pre-statin medication in their lives?
It seems so
Yes. The studies he references took place before statins were invented.
Thanks👍@@saveonbuilding
The road to to bad science is paved with good intentions.
Give blood! RBCs
Over $100 billion sales of just Lipitor, based on a lie that is sadly totally legal. I guess this is a good example of gaslighting.
@@stevet5549 No I have not figured it out. Can you give me some hints?
This presentation made it all more clear why one study I read showed a very significant absolute difference in one study (17 %) but David Diamond was able to parse out the results identifying how knowing whether someone would have high triglycerides and low HDL WOULD def impacted while those with low triglycerides and high HDL wouldn’t have a significantly better outcome on statins. Thanks, I was trying to figure out how interpretations of studies could be so very different.
Sorry for the missing word. A bit confusing, but I can understand better how these two interpretations about the impact of statins can both be true.
It's a crime!
What is FH?
Familiar Hypercholesterolemia. Its genetic.
Not all cardiologists would be alarmed. Mine told me, quote, "triglycerides are for diabetes, nothing to do with your heart". End quote. I don't have a lot of faith in cardiologists, seems their ticket can come out of a cereal packet with their breakfast 😞
FH what is it?
Familial hypercholesterolemia. It's a genetic disorder that results in very high LDL.
I would argue that they're getting less and dying less of other diseases because they're not living long enough for those diseases to manifest
So what about Chris McCaskill's takedown on Plant Chompers? "You can tell how little they've read about the [Seven Countries] study" - why misrepresent the data so badly Dr. Diamond?
So he mainly talks about LDL but then talks about the JUPITER trial wherein participants had LDL below 130 mg/dl. And when he talks about absolute risk reductions percentages he doesn't talk about the timespan. Seems rather sloppy.
I get the impression he doesn't really master the material as much as he likes to think or likes us to think.