Moved to a moldy home, couldn't sleep, a root canal flared up almost immediately, whole side of face & eye swelled up, continued to have health & teeth issues until getting Essentiale N, ozone, Glutithione IV's and an ozone injected below gum line. This after 6yrs sitting in the dark w/a laundry list of systoms. Doctors couldn't help, until finding a functional LLMD.
Dr Jill can you tell us the most affordable way to get tested for mold? Most of us can’t afford this unfortunately. I believe the mold has affected my heart with crazy palpations
Yes, the very best way is history and physical exam and visual contrast testing. All of which are free.💕 I also recommend the Dust test thedusttest.com/?_ef_transaction_id=&oid=2&affid=17
When someone ignores the thing that has more coherent scientific data in all of medicine, then they lose the ability to appeal to science after that. You can't appeal to conspiracy away the most validated hypothesis.
@@dr.jackwolfson6906There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect. Those with monogenic homozygous hypercholesterolemia can have a heart attack or stroke by age 5, monogenic heterogenous by 25-30, polygenic homozygous by age 35, and those with polygenic heterogenous could make it up to 50-55 years of age before their first event, depending on their sex. Inversely HetFHBL pretty much abolishes ASCVD even if they engage in unhealthy behaviors, like smoking, that increase risk. The Mendelian randomization studies show the log-linear association beautifully. Randomized trials including more than 2 million participants with over 20 million person-years of follow-up and over 150,000 further confirm this dose dependent relationship, add to that separate meta-analyses of over 200 prospective cohort studies. We also have a risk reduction proportional to the LDL-C reduction with multiple drugs even though they work through multiple different mechanisms to lower the LDL including statins, PSK9 inhibitors, Ezetimibe, Bempedoic acid.
@@dr.jackwolfson6906 There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect. Those with monogenic homozygous hypercholesterolemia can have a heart attack or stroke by age 5, monogenic heterogenous by 25-30, polygenic homozygous by age 35, and those with polygenic heterogenous could make it up to 50-55 years of age before their first event, depending on their sex. Inversely HetFHBL pretty much abolishes ASCVD even if they engage in unhealthy behaviors, like smoking, that increase risk. The Mendelian randomization studies show the log-linear association beautifully. Randomized trials including more than 2 million participants with over 20 million person-years of follow-up and over 150,000 further confirm this dose dependent relationship, add to that separate meta-analyses of over 200 prospective cohort studies. We also have a risk reduction proportional to the LDL-C reduction with multiple drugs even though they work through multiple different mechanisms to lower the LDL including statins, PSK9 inhibitors, Ezetimibe, Bempedoic acid.
@@dr.jackwolfson6906 There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect.
mold itself is not the issue, it is indoor toxic levels without adequate ventilation and the mycotoxins the mold produces. This is why outdoor molds are seldom an issue but toxic levels of hidden indoor mold can cause grave illness
@JillCarnahanMD So mold isn't the issue, but mold toxin is the issue. So hand in glove that it's a distinction without a difference. So humid FL must be having huge levels of mold illness and death, right? Got it, doc. Are you an expert witness for plaintiffs' attorneys? If so, what's your hourly rate for trial testimony? Half day minimum?
@jim2376 - why don't you conduct an experiment to see whether mold is or isn't a serious health risk? Everyday, pour some water on piece of drywall by your bed until the wall starts to discolor (and continue to do so once the discoloration appears). Then wait 3-6 months. The impact will happen slowly. At first you'll think it's all in your own head. Within a few weeks your symptoms will be significant enough that you can't ignore them. And eventually they'll be so severe that you'll wish you never tried it. If you're such a skeptic, let's see if you'll stay a skeptic.
I wish this conversation was more focused on mold, barely touched the surface.
I know we’ll have to do another episode!🥰
Moved to a moldy home, couldn't sleep, a root canal flared up almost immediately, whole side of face & eye swelled up, continued to have health & teeth issues until getting Essentiale N, ozone, Glutithione IV's and an ozone injected below gum line. This after 6yrs sitting in the dark w/a laundry list of systoms. Doctors couldn't help, until finding a functional LLMD.
I am glad you found someone to help you! Mold is no joke :(
Mold effects on heart starts at 8:45
Dr Jill can you tell us the most affordable way to get tested for mold? Most of us can’t afford this unfortunately. I believe the mold has affected my heart with crazy palpations
You can test your self or test your home. Both are expensive.
Yes, the very best way is history and physical exam and visual contrast testing. All of which are free.💕 I also recommend the Dust test thedusttest.com/?_ef_transaction_id=&oid=2&affid=17
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Palo.a en Su un m@@dr.jackwolfson6906
Realtime was covered by Medicare, say was because don't know what's been changed in the last year.
When someone ignores the thing that has more coherent scientific data in all of medicine, then they lose the ability to appeal to science after that. You can't appeal to conspiracy away the most validated hypothesis.
I wonder what you are referring to "coherent" scientific data? Please explain more.
Yes, please tell me more?
@@dr.jackwolfson6906There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect. Those with monogenic homozygous hypercholesterolemia can have a heart attack or stroke by age 5, monogenic heterogenous by 25-30, polygenic homozygous by age 35, and those with polygenic heterogenous could make it up to 50-55 years of age before their first event, depending on their sex. Inversely HetFHBL pretty much abolishes ASCVD even if they engage in unhealthy behaviors, like smoking, that increase risk. The Mendelian randomization studies show the log-linear association beautifully. Randomized trials including more than 2 million participants with over 20 million person-years of follow-up and over 150,000 further confirm this dose dependent relationship, add to that separate meta-analyses of over 200 prospective cohort studies. We also have a risk reduction proportional to the LDL-C reduction with multiple drugs even though they work through multiple different mechanisms to lower the LDL including statins, PSK9 inhibitors, Ezetimibe, Bempedoic acid.
@@dr.jackwolfson6906 There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect. Those with monogenic homozygous hypercholesterolemia can have a heart attack or stroke by age 5, monogenic heterogenous by 25-30, polygenic homozygous by age 35, and those with polygenic heterogenous could make it up to 50-55 years of age before their first event, depending on their sex. Inversely HetFHBL pretty much abolishes ASCVD even if they engage in unhealthy behaviors, like smoking, that increase risk. The Mendelian randomization studies show the log-linear association beautifully. Randomized trials including more than 2 million participants with over 20 million person-years of follow-up and over 150,000 further confirm this dose dependent relationship, add to that separate meta-analyses of over 200 prospective cohort studies. We also have a risk reduction proportional to the LDL-C reduction with multiple drugs even though they work through multiple different mechanisms to lower the LDL including statins, PSK9 inhibitors, Ezetimibe, Bempedoic acid.
@@dr.jackwolfson6906 There is coherence in regards to ApoBs casual role in ASCVD. “The relationship between low-density lipoprotein cholesterol (LDL-C) and risk for ASCVD is one of the most highly established and investigated issues in the entirety of modern medicine.” There's more scientific evidence supporting the relationship between LDL and CVD than any other topic. There is a consistent dose-dependent log-linear association between the absolute magnitude of exposure of the vasculature to LDL-C and the risk of ASCVD, with the inverse having the opposite effect.
24:34 Was that Dr. Sentra? 😢
Dr Howard Elkin
If mold caused health problems, everyone in FL would be sick or dead. This is fishing for legal and medical work.
mold itself is not the issue, it is indoor toxic levels without adequate ventilation and the mycotoxins the mold produces. This is why outdoor molds are seldom an issue but toxic levels of hidden indoor mold can cause grave illness
@JillCarnahanMD So mold isn't the issue, but mold toxin is the issue. So hand in glove that it's a distinction without a difference. So humid FL must be having huge levels of mold illness and death, right? Got it, doc. Are you an expert witness for plaintiffs' attorneys? If so, what's your hourly rate for trial testimony? Half day minimum?
@@jim2376 No, I just help patients heal from mold-related illness. I have zero interest in legal work. 🙂
@jim2376 - why don't you conduct an experiment to see whether mold is or isn't a serious health risk? Everyday, pour some water on piece of drywall by your bed until the wall starts to discolor (and continue to do so once the discoloration appears). Then wait 3-6 months. The impact will happen slowly. At first you'll think it's all in your own head. Within a few weeks your symptoms will be significant enough that you can't ignore them. And eventually they'll be so severe that you'll wish you never tried it. If you're such a skeptic, let's see if you'll stay a skeptic.