You guys are amazing. In my first year of med school I was dreading chemistry, but you helped me pull through. Now I am in my final year, watching this in preparation for my final surgical exams - you might not know it, but we've come a long way!💪
Great video. Can someone explain why the body would respond to atropine but not the sympathetic nervous system? I am a little confused, if the nervous system is shut down as you said, then why would it respond to atropine? Is it just because atropine blocks the PSN?
In lecture today, my professor cited PALS and said that in all the shocks [obstructive, cariogenic, hypovolemic, & distributive (anaphylactic, septic, & neurogenic)] the BP is not like adults meaning that the pediatric patient can compensate (due to a healthier heart)and maintain a normal BP while in the compensatory phase of shock, then switch into a low BP once not being able to compensate anymore. All that makes sense, except in neurogenic shock, because the SNS compensation ability is enabled, so how can there ever be a normal BP in neurogenic shock for a pediatric patient? I asked what makes a pediatric patient with a SCI experiencing neurogenic shock different than an adult patient in regards to BP, and I was told that the child can compensate better than an adult. After asking again how that can be without a functioning SNS I was then told it depends on they type of SCI and if the child was decapitated. So, I am at a loss, please help!
s/s for neurogenic shock must be observe all the time, check vital signs, prn medications must be available at bedside, physicians tel. number/mobile number must be visible (on the wall ) for emergency tel. orders, 02 at bedside, maintain a quite environment, npo, emitional support and other related nursing intervention
Both my legs are numb. My leg started to spasm and then tightened down on my viens. I don't feel like I'm getting any blood flow. What type of doctor woukd I need?
Yes that's true but in the video he does state treating with pressors to treat hypotension from the shock, thus making it safe to administer IV fluids without risking edema.
+tshepo dibotelo yeah it is used.neurogenic shock leads to depression of sympathetic system which leads to loss of SVR. Sympathomimetic drugs includes epinephrine , norepinephrine etc which increases vascular tone and promote better perfusion .
The Heart rate is controlled by the nervous system which controls the firing rate of the SA node. From my previous readings I knew that the firing rate of the SA node is 120 bpm but what makes the heart rate less than this number is the fact that the SA node is under the control of the vagus nerve so if we lose nervous control of the heart the heart rate should increase not decrease. Am I wrong? Please explain this point and thank you very much for your great videos
Ghada Karim spinal shock is mainly due loss of sympathetic tone that is thoracolumbar outflow but meantime when sympathetic is blocked parasympathetic becomes unopposed so Brady will b there
I always thought the SA node worked independent of the nervous system, and that's why it continues to pump outside the body. A little confused by how susceptible the heart rate is to neurogenic shock
Thank you for videos! I've read in one book that in the neurogenic shock there are at first bradycardia and big CO, but after some time there is a tachycardia and small CO... I'm confused - can you explain me these things? Are they true?
sympathetic outflow occurs from T1 to L2/3, this means that if there is a transection around this point the sympathetic innovation will be lost and wide spread vasodilation occurs
This is great. Really helping me study for my RN Exam tomorrow. Khan Academy always to the rescue 🙏🏼🙏🏼
You're videos are absolutely phenomenal! I cannot stress this enough. Thank you so much for the work you are doing.
You guys are amazing. In my first year of med school I was dreading chemistry, but you helped me pull through. Now I am in my final year, watching this in preparation for my final surgical exams - you might not know it, but we've come a long way!💪
Thank you so much for this video, really fills in the gaps on my terrible textbook explanation
khan i love you im gonna pray for you everyday
Excellent 👌!
This helped me to understand and separate neurogenic shock from the other shocks I'm reviewing
You're Awesome! Thank you!
Beautifully explained
Great!👌
That was so helpful. Thank you so much!
Thank you so much! the patho really helped break it down for me!
great video, made it very easy to understand neurogenic shock
Thank you in 2020, too 😊
Great video. Can someone explain why the body would respond to atropine but not the sympathetic nervous system? I am a little confused, if the nervous system is shut down as you said, then why would it respond to atropine? Is it just because atropine blocks the PSN?
You guys are life savers!!!!!!!
Totally awesome explanation ! ! Thank you! :)
What are the baroreceptors and chemoreceptors name responsible in this machenism ?
In lecture today, my professor cited PALS and said that in all the shocks [obstructive, cariogenic, hypovolemic, & distributive (anaphylactic, septic, & neurogenic)] the BP is not like adults meaning that the pediatric patient can compensate (due to a healthier heart)and maintain a normal BP while in the compensatory phase of shock, then switch into a low BP once not being able to compensate anymore. All that makes sense, except in neurogenic shock, because the SNS compensation ability is enabled, so how can there ever be a normal BP in neurogenic shock for a pediatric patient? I asked what makes a pediatric patient with a SCI experiencing neurogenic shock different than an adult patient in regards to BP, and I was told that the child can compensate better than an adult. After asking again how that can be without a functioning SNS I was then told it depends on they type of SCI and if the child was decapitated.
So, I am at a loss, please help!
Great video, thanks!
You guys are awesome! Thank you!
spinal injury above T6 (sympathetic chain T1-T4?)
s/s for neurogenic shock must be observe all the time, check vital signs, prn medications must be available at bedside, physicians tel. number/mobile number must be visible (on the wall ) for emergency tel. orders, 02 at bedside, maintain a quite environment, npo, emitional support and other related nursing intervention
Both my legs are numb. My leg started to spasm and then tightened down on my viens. I don't feel like I'm getting any blood flow. What type of doctor woukd I need?
Great explanation!
Many are left with no info on this and the sequence before and after. Excellence info and much thanks.
Thank you! that was great!
very clear
The weak pulse that u notice in shock is due to low stroke volume and low blood volume?
no sympathetic input from the SNS
Fluid therapy is used cautiously because risk of pulmonary edema
Good to know
Plz provide me your spinal shock vedio link sir
Please, somebody, what do you do after a neurogenic shock?
U r giving IV fluids but heart is not working efficiently, won't it lead to edema?
Yes that's true but in the video he does state treating with pressors to treat hypotension from the shock, thus making it safe to administer IV fluids without risking edema.
I wonder if adrenalin (epinephrin) could be used as a treatment regime for neurogenic shock
+tshepo dibotelo yeah it is used.neurogenic shock leads to depression of sympathetic system which leads to loss of SVR. Sympathomimetic drugs includes epinephrine , norepinephrine etc which increases vascular tone and promote better perfusion .
thanks alot you made my day and I do love this black background
Thank you :)
The Heart rate is controlled by the nervous system which controls the firing rate of the SA node. From my previous readings I knew that the firing rate of the SA node is 120 bpm but what makes the heart rate less than this number is the fact that the SA node is under the control of the vagus nerve so if we lose nervous control of the heart the heart rate should increase not decrease. Am I wrong? Please explain this point and thank you very much for your great videos
Ghada Karim spinal shock is mainly due loss of sympathetic tone that is thoracolumbar outflow but meantime when sympathetic is blocked parasympathetic becomes unopposed so Brady will b there
I always thought the SA node worked independent of the nervous system, and that's why it continues to pump outside the body. A little confused by how susceptible the heart rate is to neurogenic shock
good work
I like your voice :) lol good explanation thanks~
why am i watching this im in the 8th grade lol
Thank you!
Thank you for videos!
I've read in one book that in the neurogenic shock there are at first bradycardia and big CO, but after some time there is a tachycardia and small CO... I'm confused - can you explain me these things? Are they true?
Bradycardia is a sign not a symptom
why it happen just T6 level or above ???
sympathetic outflow occurs from T1 to L2/3, this means that if there is a transection around this point the sympathetic innovation will be lost and wide spread vasodilation occurs
Good videos but the sound isn’t good on this one only on right side of it could just be me???
Great explanation!
Great explanation !
Amazing explanation!