I think that you made a small mistake when describing afterload. Afterload is not the amount of blood left after ejection of blood, but rather the pressure that the heart has to build up, in order to eject blood against the pressure of the blood that is already in the cardiovascular system within the arteries. The amount of blood left after ejection is known as the "left ventricular end systolic volume".
Afterload is the tension the heart must produce to force open the semilunar valves and eject the blood. EDV (end diastolic volume) is the volume left in the ventricle after filling. SO ESV-EDV= stroke volume. Make sure to make a comment under that part of the video so students don't get confused why studying. Other than that, great video!
Hemoglobin/hematocrit can also be increased, due to concentration. In fact, my nursing book specifically points out that it can be raised, but not the possibility of a decrease (though, that does make logical sense to me-blood out = lower levels).
Hi, could you clarify whether the vasoconstriction happens in the venous or arterial side? To increase SVR, I believe the arterioles vasoconstrict to divert blood to essential organs.
Andy Nian The vasoconstriction concerned in maintaining perfusion is on the arterial side. The effect is to increase systolic pressure to allow perfusion of vital organs, local tone determines to some extent which organs are considered “vital”. Think BP = CO x SVR = SV x HR x SVR. Venous constriction increases cardiac return and so increases the ventricular preload and, within certain conditions, increases stroke volume.
You are claiming that hematocrit count goes down but doctors say it goes up. Please consult professionally and correct. Remuneration accepted by me gratefully.
adham hussiun Adrenaline is not ideal in hypovolaemia as it has combined alpha and beta stimulant properties and can actually reduce carotid blood flow and, therefore, brain perfusion.
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I think that you made a small mistake when describing afterload. Afterload is not the amount of blood left after ejection of blood, but rather the pressure that the heart has to build up, in order to eject blood against the pressure of the blood that is already in the cardiovascular system within the arteries. The amount of blood left after ejection is known as the "left ventricular end systolic volume".
fernandoalo Preach. Usually Khan Academy is a pretty reputable source...
you are correct, I noticed that too
fernandoalo Bruhh
Me too notic that was not sure about this point
Thanks
Yeah i noticed that too
Afterload is the tension the heart must produce to force open the semilunar valves and eject the blood. EDV (end diastolic volume) is the volume left in the ventricle after filling. SO ESV-EDV= stroke volume. Make sure to make a comment under that part of the video so students don't get confused why studying. Other than that, great video!
so true
It is EDV-ESV=StrokeVolume.
I use all of the Khan Academy videos while learning all types of topics in nursing school. Wonderful learning tools!
same for me ,, even i study in french ,, but its very useful
Cells At Work Episode 12~13
SAME XDDDDDDDD
You genius! :D
lol
These are great learning tools! I am going to show your videos to my nursing professors, thanks!!
Wow. Now I clear off all the mysteries about it. Thank you so much, sir.
Hemoglobin/hematocrit can also be increased, due to concentration. In fact, my nursing book specifically points out that it can be raised, but not the possibility of a decrease (though, that does make logical sense to me-blood out = lower levels).
Hi, could you clarify whether the vasoconstriction happens in the venous or arterial side? To increase SVR, I believe the arterioles vasoconstrict to divert blood to essential organs.
Andy Nian
The vasoconstriction concerned in maintaining perfusion is on the arterial side. The effect is to increase systolic pressure to allow perfusion of vital organs, local tone determines to some extent which organs are considered “vital”. Think BP = CO x SVR = SV x HR x SVR.
Venous constriction increases cardiac return and so increases the ventricular preload and, within certain conditions, increases stroke volume.
Que bueno eres para explicar informacion. Muchas gracias amigo ayudo mucho! :)
Can blood transfusion save Hypovalemic shock?
thanks a lot...I was nuch confused about physiology of shock...it helped m a lot from concepts to end treatment...❤
the sound is really low..
I JUST LOVE YOU GUYS !!
my mother died on what they call hypovolemic shock.but in our hospitals there is a lot of negligence
wouldnt you say the h and h could be high if it wasn't due to blood loss but because of other fluid loss?
After load is not end systolic volume … period
thank you so much!!
Thank you sir ♥️
thank you☺
You are claiming that hematocrit count goes down but doctors say it goes up. Please consult professionally and correct. Remuneration accepted by me gratefully.
brilliantly put
Love you Khan let me go update my account!
Wow y'all are AMAZING
This is very informative😍
YOU NEED TO MAKE IT AUDIBLE!!!!! I HAVE IT ON MAX AND CAN HARDLY HEAR IT
Thanks khan !! :)
NYC video sir keep it up
Sound is low 🙁
i know that we should't give the patient adrenaline but why ?
adham hussiun
Adrenaline is not ideal in hypovolaemia as it has combined alpha and beta stimulant properties and can actually reduce carotid blood flow and, therefore, brain perfusion.
map is not co x svr. bp is co x svr, smh
(Not so) fun fact: this is how Selena died
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