thank you a lot.I was struggling to understand the reentry mechanism but now thanks to you it's clear.The books i read didn't emphasize enough the refractary period and that's why i had such a rough time.THANK YOU
One of the pathway needs to have long refractory and one pathway needs to have short refractory period for re entry to work. If fast conducting pathway had short refractory period than impulse will reach the pathway with long refractory period via distal connection so fast that the initially blocked pathway will still be blocked and re entry won’t be able to take place. That’s why he says in the video that the conduction through the pathway with short refractory pathway should be slow enough such that by the time it reaches the distal connection the initially blocked pathway has come out of its refractory period and re entry can take place.
Eric's Medical Lectures Hi Dr Eric, could you please explain is the reentry phenomenon transient? Putting into consideration that after the first reentry has taken place, by the time the second reentry is about to take place, the fast pathway would have entered refractory period again (owing to a new electrical signal coming from the SA node/atrial pacemaker cells) and thus lead to a normal anterograde conduction again. Thank you very much!
Great video! I am though wondering wether the information at 10:53 is accurate. The slow pathway, with slow conduction has a prolonged action potential and thus should have a long refractory time? And the fast pathway, with fast conduction has a short action potential and thus a short refractory time.
Thanks for the question. I am 100% sure that the information is accurate. The conduction velocity and the refractory time are separate, independent physiologic parameters. The conduction velocity is determined predominantly by the function of sodium channels, while the refractory period is determined largely by potassium channels. When we talk about the slow pathway having a "short refractory time", that's not the refractory time of the entire pathway taken as a whole, but rather the refractory time of a single point along that pathway, as demonstrated in the animation.
Or....... if you're not a prehospital whacker with a sub $10/hr interest in the physiology of the quintessential mechanical organ of the human body, maybe watch it more times than you spent hours in EMT-P class.
wow! Simply excellent video to better understand tachyarrhythmias ahead of cards fellowship! very grateful for your efforts, Dr. Strong!
That reentry mechanism explanation is gold, you must be a wizard
thank you a lot.I was struggling to understand the reentry mechanism but now thanks to you it's clear.The books i read didn't emphasize enough the refractary period and that's why i had such a rough time.THANK YOU
Thank you. I watch this second time. It is very helpful to me. I can’t thank you enough.
thankyou very much ，love from 3rd year medical student from China
Thank you Dr Strong. This is my third time watching this video.
Excellent, fantastic videos! Very well done and extremely informative. Thanks for your hard work putting these together, they are a huge help!
Absolutely awesome explanation of concepts I struggle to grasp.
Greetings from sunny South-Africa!
Thank you!! I'm an 2nd year medical student and your video explained what my professor couldn't explain in 3 hours
Reentry got me in chokehold for hours....thanks a lot
Thank you! It really helps when I struggle watching so many videos but still confusing. Now I’m able to understand the mechanism behind. Thanks a lot!
Many thanks for this, a very good and clear explanation of some very tricky concepts. Will be recommending this to my students.
This is GOLD , strong medicine, GOLD !!!
Who else here for Reentry
Si😂
Excellent work, thank you very much.
thanks a ton....for such a beautiful and comprehensive video...
Sir u r just too good to be explained by me,loved them,want more videos and ur no
Animation on reentry made my day
Brilliant as per usual
Nice work......تم
Brilliant! Thank you!
Excellent and meticulous work
excellent video!!
You are the best.
Thank You so much for the animations..they helped a lot
Thank you very much!
I FINALLY UNDERSTAND REENTRY HOOOOLY FFF-
I NEVER KNEW THAT IT'S JUST THAT SIMPLE WHY DO THEY HAVE TO OVERCOMPLICATE IT IN MED SCHOOL
your lesson is wonderfull
Loved the video!! It's really beneficial, thank you!
Thank you very much ^^
thank you so much
I am Thankful
Thank you Sir
thanks a lot!
So good
Thnks a lot❤️
Great teaching ...can you please explain and add about entrainment...thanks
I briefly talk about entrainment in this video on advanced EP topics (just after the 12 min mark): ruclips.net/video/Q87VV_luZO4/видео.html
Can anyone please explain why the pathway with fast conduction has long refractory time and vice versa? Thank you very much :)
Bùi Xuân Bảo Danh maybe its just aleatory and when those conditions meet the setting is ripe for arythmia
One of the pathway needs to have long refractory and one pathway needs to have short refractory period for re entry to work. If fast conducting pathway had short refractory period than impulse will reach the pathway with long refractory period via distal connection so fast that the initially blocked pathway will still be blocked and re entry won’t be able to take place. That’s why he says in the video that the conduction through the pathway with short refractory pathway should be slow enough such that by the time it reaches the distal connection the initially blocked pathway has come out of its refractory period and re entry can take place.
Eric's Medical Lectures Hi Dr Eric, could you please explain is the reentry phenomenon transient? Putting into consideration that after the first reentry has taken place, by the time the second reentry is about to take place, the fast pathway would have entered refractory period again (owing to a new electrical signal coming from the SA node/atrial pacemaker cells) and thus lead to a normal anterograde conduction again.
Thank you very much!
What is meant by premature stimulus here?
Reentry 8:13
nice
Great video! I am though wondering wether the information at 10:53 is accurate.
The slow pathway, with slow conduction has a prolonged action potential and thus should have a long refractory time?
And the fast pathway, with fast conduction has a short action potential and thus a short refractory time.
Thanks for the question. I am 100% sure that the information is accurate. The conduction velocity and the refractory time are separate, independent physiologic parameters. The conduction velocity is determined predominantly by the function of sodium channels, while the refractory period is determined largely by potassium channels. When we talk about the slow pathway having a "short refractory time", that's not the refractory time of the entire pathway taken as a whole, but rather the refractory time of a single point along that pathway, as demonstrated in the animation.
I now see the difference. Thank you very much for this explanation.
7:30 I get that ATP depletion and digoxin toxicity decrease Na-K ATPase activity, but how does hypokalemia influence it?
How will Na-K ATPase work with low potassium on blood serum?
@@Dequiter Oh boy, thx! How embarrassing :D
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Hello..new friend here..lets connect for succes
Or....... if you're not a prehospital whacker with a sub $10/hr interest in the physiology of the quintessential mechanical organ of the human body, maybe watch it more times than you spent hours in EMT-P class.
the lectures is very slow it needs more hurry.