That's millions of times better than the board; the information is more organized here, and you're more at ease. Still, we have no complaints, as it's free.
Excellent video, thank you. One small correction-diabetic neuropathy is understood to be the result of direct axonal degeneration, with damage to Schwann cells being implicated to a lesser extent. See Feldman et al. (2019) and Statpearls
ZACH, please make more of the "study case" playlist! IT'S UNDERSTANDABLE AND MAKE SENSE A LOT! i'm looking forward to see more of the playlist! Keep it up, God bless you!
This is an awesome video. Very helpful and understandable. I just want to say that I prefer the board videos where you explain more detailly. But you are the best ❤
Hey! i would love to see a DKA vs HHS video please! I am studying for my PCCN. i love watching your videos because i can actually understand the "why" behind things. thanks for all your time put into these videos!
For Australian viewers who are new to studying, safe BGL levels here are 4-7.8 (DiabetesAustralia) mmol/L. American values are 72-108 mg/dL. We measure mmol per litre & they measure mg per deci liter.
Im from kurdistan and i want to say thank you sooo much for your detail information ,Im in dental collage 🦷 and needed your information to understand .✨✨🙌🏼
This method is better than the whiteboard. It's more organized and smoothly connected as well, hence you're able to teach faster than you did with the whiteboard. I'm glad for this teaching. Succinct and superb. As always, thank you so much 😁
You should add other types of diabetes in the future, particularly MODY and 3c. I suspect MODY is grossly underdiagnosed and thrown into the T2D or T1D types way too much!
7:57 - Why can glucose not be utilised in diabetes? Surely the high glucose content in the blood doesnt require insulin to enter cells via facilitated diffusion using GLUT1 receptors? What is meant here by not being able to utilise carbohydrates?
I think that GLUT1 alone cannot provide enough glucose for some tissues to use, for example muscular tissue. That's why insulin reduction determines metabolic switch to lipolysis.
In T2DM, how does glucose get into the cell to cause increased sorbitol when there is insulin resistance preventing glucose from being taken up by cells?
White Board was Gold It was Hell of a Big Boring Lecture . If diagrams were there This could've been ur block buster of the year 🤟 But any ways you still Tried Your Heart out to teach us Thank yu Ninja sir ❤
I dont get it If in the type one these complete deficiency of insulin secretion And in type 2 theres still insulin production but not as adequete Then how is it possible that in type 2 theres more hyperglycemia compared to type 1(where there is NO insulin compared to insufficient amount in type 2)
Please let’s reverse back to the use of our white board learning module this would give us the real classroom room learning experience, something that really marked different than any other learning platforms( if so less) 😢 .
My PNS and ANS is fried ... So if I have a BM my sugar levels drop how I see it is I damage my thoracic spine in 01 I got EBV that Triggered G.B.S with autonomic dysfunction in 09 ... I was drinking a lot of Coca-Cola unknown to me it was keeping autonomic Dysreflexia under control?? And this? In away! ... As im bowel triggered the caffeine would stimulate the the GI track and the supar would keep my level up...
What about later stages of T2 diabetes where insulin production by the body is greatly decreased (or completely gone) and insensitivity is increased. For instance, what if someone came to the ER due to dizzy spells and an inability to walk short distances without fainting but previous to that hadent seen a doctor for over a decade. Would their T2 diabetes have progressed far enough to warrant lipolysis and ketone bodies in the blood since glucose wouldent be a viable source of energy anymore. Would we then see symptoms of DKA in T2 diabetics similarly to T1 diabetics? My thought process is that there would be protein and glucose loss through glomerular damage which would pull water from the system. Patient becomes dehydrated and begins exhibiting orthostatic tachycardia and only visits the ER when the issues become prevalent enough.
That's millions of times better than the board; the information is more organized here, and you're more at ease. Still, we have no complaints, as it's free.
I like this new method of teaching, not a time consumer and flow smoother
Thank you for taking your time and creating these medical lectures in the most comprehensive, yet easy-to-digest ways.
Please revert back to the original whiteboard Easy to get bored with thus this format.
Yes please
I agree
PLEASE 🙏
Pls sir
No! This format is clearer, only that it should be larger, as we don't see anything.
Pleaae bring back the white board!
Excellent video, thank you. One small correction-diabetic neuropathy is understood to be the result of direct axonal degeneration, with damage to Schwann cells being implicated to a lesser extent. See Feldman et al. (2019) and Statpearls
i honestly am fine with both the board and this . Im just here to learn and grateful for the help !
Zach if u don’t mind …
I want to enter your brain to see how he made any topic easy & digestible to be understood
Thanks 👏🏻
You lectures are directly going into the brain when you are explaining on whiteboard with your brilliant hands. So please keep up on whiteboard
ZACH, please make more of the "study case" playlist!
IT'S UNDERSTANDABLE AND MAKE SENSE A LOT!
i'm looking forward to see more of the playlist!
Keep it up, God bless you!
I'm pratheesh from India (Tamil Nadu).big fan of you sir.say one hii sir..
Why are you every where bro ?😂
@ok61😂😂😂😂😂😂😂😂😂09
This is an awesome video. Very helpful and understandable. I just want to say that I prefer the board videos where you explain more detailly. But you are the best ❤
Hey! i would love to see a DKA vs HHS video please! I am studying for my PCCN. i love watching your videos because i can actually understand the "why" behind things. thanks for all your time put into these videos!
He has one video published long time ago when i was still in med school.
I absolutely love your videos. You aid me in actually understanding the foundation to things.
Where is the dka vs hhs video? Once watched but not seeing it anymore
Thanks a lot for breaking it into digestible chunks!!
Thanks for uploading 3 dyas before my exam
Clinical medicine lectures are life savers 😊
For Australian viewers who are new to studying, safe BGL levels here are 4-7.8 (DiabetesAustralia) mmol/L. American values are 72-108 mg/dL.
We measure mmol per litre & they measure mg per deci liter.
This is a great video! Please consider doing a DKA and HHNS video! This would be extremely helpful as a new endocrine APN
Can anyone please give me pdf of this class!?
Im from kurdistan and i want to say thank you sooo much for your detail information ,Im in dental collage 🦷 and needed your information to understand .✨✨🙌🏼
This really came at the right time ! Thank youuuu
Thankyou for these! You surely are a life saver. I love how easily these summary charts in your videos help me correlate it to a real life patient
Oh just realized Zach renewed the diabetes video. That's cool!
This is better than the whiteboard for meeeeeee, THANK U SO MUCH
This method is better than the whiteboard. It's more organized and smoothly connected as well, hence you're able to teach faster than you did with the whiteboard.
I'm glad for this teaching. Succinct and superb. As always, thank you so much 😁
Please pleeeaassseeee revert ti the whiteboard that is what make ur identity 🥺🥺🥺🥺🥺💔💔
True😢
I recommend to say what does those abbreviations means
Thank you Sir.. Understanding wasn't hard with this. Thank you!
Will there be notes posted soon for this?
thank you so much for your help
Honestly AMAZING!! LOVING this new series - thank you so so much :))
Please, is there any video on diabetic emergency?
Great work ❤
Hell yeah love this!
Thanks ! Very complete revision
Honestly thank you ninja nerd
Does this video say that CKD results in a bump up in the GFR??
Bring back the whiteboard pleaseeeee😭🙏🏻
I think when managing type 2 Dm you should start with low dose metformin then taper up after 3 months based on A1C before adding a second OHGA?
The best always! Thank you!
Thank you very much!
Zach! i love you.
God bless you
Wow on the 30th minute, you can easily lower A1c with carb restriction without meds
Please do the white board.
Great lecture.
You should add other types of diabetes in the future, particularly MODY and 3c. I suspect MODY is grossly underdiagnosed and thrown into the T2D or T1D types way too much!
Thanks
Thank you sir
7:57 - Why can glucose not be utilised in diabetes? Surely the high glucose content in the blood doesnt require insulin to enter cells via facilitated diffusion using GLUT1 receptors? What is meant here by not being able to utilise carbohydrates?
I think that GLUT1 alone cannot provide enough glucose for some tissues to use, for example muscular tissue. That's why insulin reduction determines metabolic switch to lipolysis.
Because the insuline resistance , the receptor cannot be acctivated properly and GLUT molecules cannot go into the membrane
Thanks so much 🎉🎉🎉🎉❤❤❤
White board still my favourite teaching way.. Regards
great lecture
Love you from Ghana
In DKA even if the AG is closed don't you want to continue the ins gtt until the BG's are stable for 24hrs or ar least 3 BG's
In T2DM, how does glucose get into the cell to cause increased sorbitol when there is insulin resistance preventing glucose from being taken up by cells?
Insuline resistance appears only in fat tissue and muscle, others sites with others glut receptors dont get the resistance.
@@joao09101 thanks
I am actually crying because I finally understand. Thank you.
thank you
Thanks and love you too
How many different types 1and 2
Asante sanaa🎉
thanks ninja 😊
Sir,You didn't include 1.5KPD and 1.5 LADA
White Board was Gold It was Hell of a Big Boring Lecture . If diagrams were there This could've been ur block buster of the year 🤟 But any ways you still Tried Your Heart out to teach us Thank yu Ninja sir ❤
I dont get it
If in the type one these complete deficiency of insulin secretion
And in type 2 theres still insulin production but not as adequete
Then how is it possible that in type 2 theres more hyperglycemia compared to type 1(where there is NO insulin compared to insufficient amount in type 2)
Please let’s reverse back to the use of our white board learning module this would give us the real classroom room learning experience, something that really marked different than any other learning platforms( if so less) 😢 .
life saver
My PNS and ANS is fried ... So if I have a BM my sugar levels drop how I see it is I damage my thoracic spine in 01 I got EBV that Triggered G.B.S with autonomic dysfunction in 09 ... I was drinking a lot of Coca-Cola unknown to me it was keeping autonomic Dysreflexia under control?? And this? In away! ... As im bowel triggered the caffeine would stimulate the the GI track and the supar would keep my level up...
Why do u delete the old video🙄🙄🙄
It is in retired playlist not deleted
Sir are you tired of drawing in whiteboard.......latest updated contents may attract some viewers but traditional way of teaching has its own aroma
How many views does it have now?
1.2k
12k
Where is Old DKA video 👀💔
Sir this method isn’t working like your previous method on white board sir please revert back to whiteboard plzzzzzzzzzz
Love
19:04 I thought the problem is exactly the opposite 🙃
now surgeon could finally mx DKA😎
top
❤❤
What about later stages of T2 diabetes where insulin production by the body is greatly decreased (or completely gone) and insensitivity is increased. For instance, what if someone came to the ER due to dizzy spells and an inability to walk short distances without fainting but previous to that hadent seen a doctor for over a decade. Would their T2 diabetes have progressed far enough to warrant lipolysis and ketone bodies in the blood since glucose wouldent be a viable source of energy anymore. Would we then see symptoms of DKA in T2 diabetics similarly to T1 diabetics? My thought process is that there would be protein and glucose loss through glomerular damage which would pull water from the system. Patient becomes dehydrated and begins exhibiting orthostatic tachycardia and only visits the ER when the issues become prevalent enough.
thats cool but ... we want the board back :(( #oldschool
White board😢😢😢😢
8:51
❤
Sorry you are wrong I got TD1 @57 years old
❤❤❤❤
Wilson Laura Anderson Amy Williams Eric
👏🏽
it can take wayyyy to long to find out you have type 1 diabetes
White board was better brother!
May Allah Guide you to islam
Please turn to whiteboard, this format is boring
Chalk and talk is better not this
Best ninja 🥷
what a lecture. You have just nailed it. thank you so much. 🤍
marry me🥹🤎
Look what I found! A fresh video! Thank you for your efforts!
thanks
Thank you sir
this is soo good, described everything from pathology to how it impacts everyday clinical scenarios. thank you a bunch