Thank you for wonderful series , as always. What was the final diagnosis in the case shown in this lecture..was it toxoplasmosis or ? Multiple pyogenic abscesses. Thanks
Sorry, that wasn't clear. The case shown throughout the lecture is multiple abscesses. The clue is the diffusion imaging (1:55). Toxoplasmosis usually doesn't have that internal restricted diffusion like that.
Calcium has complex behaviors on MRI. Hydrated calcium, or calcium in the presence of water, tends to cause the region to be T1 hyperintense. Dense calcium, like bone or dystrophic calcification, will be dark on all sequences because it lacks free water to create signal. Check out this reference: pubs.rsna.org/doi/10.1148/radiology.179.1.1848714
Caution regarding need to use generic not trade names. Thank you for an outstanding series!
Yeah, that's a good point, although unfortunately here many patients don't know the generic names. Probably best to be familiar with both.
Thanks alot for these great lectures.... they are very interesting....
Glad you like them!
Wonderful work
Go on
Thanks
Thank you! Cheers!
Revisit and thumb up.
Thank you for wonderful series , as always.
What was the final diagnosis in the case shown in this lecture..was it toxoplasmosis or ? Multiple pyogenic abscesses.
Thanks
Sorry, that wasn't clear. The case shown throughout the lecture is multiple abscesses. The clue is the diffusion imaging (1:55). Toxoplasmosis usually doesn't have that internal restricted diffusion like that.
@@LearnNeuroradiology ok. Thanks a lot
Calcium hyperintense on nonenhanced T1WI? Isn't it hypointense?thank you
Calcium has complex behaviors on MRI. Hydrated calcium, or calcium in the presence of water, tends to cause the region to be T1 hyperintense. Dense calcium, like bone or dystrophic calcification, will be dark on all sequences because it lacks free water to create signal.
Check out this reference:
pubs.rsna.org/doi/10.1148/radiology.179.1.1848714
@@LearnNeuroradiology thanks! I've looked it up. It makes sense now! Outstanding work!