Yes because sodium lowers extracellular fluid volume and that initiates aldosterone secretion (hyperaldosteronis) which then leads to sodium reabsorption of Na and the K+ gets out so we have hypokalemia.
There is inc hco3- loss . Which causes decrease activity of basolateral na+ and cl- activity. Causing decreased sodium and hence hyperaldosteronism . So eventually there is hypokalemia my friend. Aldosterone causes Inc na reabsorption and potassium and H+ secretion . Potassium and ammonia are inversely related so high potassium due to hypoaldosteron nwill cause low ammoniogenesis.
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Shouldn't NaHCO3 loss cause hyperaldosteronism instead of hypoaldosteronism to result in hypokalemia in type II RTA?
yes i have the same question
Yes because sodium lowers extracellular fluid volume and that initiates aldosterone secretion (hyperaldosteronis) which then leads to sodium reabsorption of Na and the K+ gets out so we have hypokalemia.
Best video on RTA
Watching this just before an exam. Thanks a lot. God bless
This was so dam good! Explained first aid where it makes sense now! Please continue to make videos.
There is inc hco3- loss . Which causes decrease activity of basolateral na+ and cl- activity. Causing decreased sodium and hence hyperaldosteronism . So eventually there is hypokalemia my friend. Aldosterone causes Inc na reabsorption and potassium and H+ secretion .
Potassium and ammonia are inversely related so high potassium due to hypoaldosteron nwill cause low ammoniogenesis.
I have sjogrens and in five years my gfr dropped 20 points I wonder if I have this. Nephrologist says she's not concerned at this point
Besttttt
awesome, thank you
I think the correct words at 1st column x 3rd line of the chart are Serum Potassium (K+) instead of Serum pH again, right?
Thnx