I haven't yet read his 2013 article on the differentiation (how to distinguish in the first year but how about 18 month +) endocrinological immune based factors from various forms of insecure attachment with respect to autism spectrum disorders. I would venture to say, however, as a developmental therapist working with families of children 0-3 years of age diagnosed/misdiagnosed secure vs, insecure based attachment practices and deepening attunement and expanded reciprocal emotional ideation (e.g., two-reciprocal emotional problem solving to simple to complex affect based reciprocal symbolic play sequences) does of course tremendously affect the LHPA (limbic, hypothalamus, pituitary, adrenal) axis and autonomic regulation (i.e., fight, flight, freeze, feint) responses. in fact the entire prefrontal cortex or executive functioning with subcortical functioning . I wonder, though, how much can be accurately teased out in the combination of the two? How much is it immunological vs insecure and of the course the combination or synthesis of the two? What came first a chicken or egg situation? What I mean is if we take an over abundance of cortisol and testosterone beginning prenatally, i.e., environmental neurotoxins, and thus dramatically altering affect regulation, synaptic formation/neuronal right to left hemisphere connectivity resulting in developmental disorders such as ASD, what happens when we add in the many layers of insecure attachment practices, neglect and abuse to less severe ambivalent attachment? . Both iendorcrinological facts and unhealthy nurturance based practices impact the LHPA axis (our autonomic regulation and immune system). My point is in some cases the two are clear cut but in many cases, if we take the full impact of epigenetic dyadic and larger social-emotional relational practices not at all!
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Excellent info. Good to know
I haven't yet read his 2013 article on the differentiation (how to distinguish in the first year but how about 18 month +) endocrinological immune based factors from various forms of insecure attachment with respect to autism spectrum disorders. I would venture to say, however, as a developmental therapist working with families of children 0-3 years of age diagnosed/misdiagnosed secure vs, insecure based attachment practices and deepening attunement and expanded reciprocal emotional ideation (e.g., two-reciprocal emotional problem solving to simple to complex affect based reciprocal symbolic play sequences) does of course tremendously affect the LHPA (limbic, hypothalamus, pituitary, adrenal) axis and autonomic regulation (i.e., fight, flight, freeze, feint) responses. in fact the entire prefrontal cortex or executive functioning with subcortical functioning .
I wonder, though, how much can be accurately teased out in the combination of the two? How much is it immunological vs insecure and of the course the combination or synthesis of the two? What came first a chicken or egg situation? What I mean is if we take an over abundance of cortisol and testosterone beginning prenatally, i.e., environmental neurotoxins, and thus dramatically altering affect regulation, synaptic formation/neuronal right to left hemisphere connectivity resulting in developmental disorders such as ASD, what happens when we add in the many layers of insecure attachment practices, neglect and abuse to less severe ambivalent attachment? . Both iendorcrinological facts and unhealthy nurturance based practices impact the LHPA axis (our autonomic regulation and immune system). My point is in some cases the two are clear cut but in many cases, if we take the full impact of epigenetic dyadic and larger social-emotional relational practices not at all!
Allan Schore contact: see www.allanschore.com