Thanks sir We are operating ventilators since many years but your videos help us to clear our concept and our ability and quality to manage patient in icu increasing day by day I am icu incharge of multiespeciality hospital Your videos help us me lot Thank you very much
Very simple and short video. These indications are absolutely correct in a healthy person, or at least in the absence of lung disease. I just have one question: What do you think happens to the right ventricular afterload after intubation in an ARDS patient with a significant proportion of atelectasis?
My 2 hrs of research on this particular topic on internet has been summed up by you so beautifully. I was so confused because i mixed up the physiological concepts. Couldnt thank you more ❤ keep clearing our concepts sir ❤ big fan❤
Thanks sir.। I remember once HOD sir asked this thing in seminar discussion.। And we all residents were confused😵, Thanks a lot for explaining such a simple way.। Plz keep on posting such basic concepts videos, Thanks to your venti. Videos also🙏
great video. Just a quick question. I understand how the increased intrathoracic pressure will aid the LV contraction allowing better ejection of blood but I dont understand how that would lower afterload. In my thinking it would make it easier for the Left ventricle to reach and overcome the afterload and eject more blood systemically. But Isn't the value of afterload dependent on the systemic vascular resistance and systemic blood pressure. Thats the main thing that has always confused me.
When you have increased intrathoracic pressure and lower extrathoracic pressure... its easier for the LV to pump from high gradient to low gradient... Can also think of it like increased thoracic pressure leads to less resistance in the proximal outlet tract and thus LV can push easily therefore decreased afterload. After load is a measurement of the pressure the LV has to generate to push out blood. hope that helps
Sir can you please help me? I'm suffering from respiratory muscle weakness due to myotonic muscular dystrophy,,shortness of breath with sleeping problem but no doctor i find who help me and prescribed me any NIV device.. Sir can you guide me how to i get any NIV device and its setting for helpful to me for my breathing.. please sir please
How sir due to positive ventilation alveoli open wide so compress the vessel so resistance increase so aftee load to right ventrivle iincrease same due to vessel compression blood squce to left chamber so pre load increase but chronic it may reduce pre load yo left chamber But how after load of left chsmber depent on perepheral resistance so we cant judge after load here am i correct
Hello dr Iam making many test of sperm doctors says you have Streptoccocus aureus and they giving me clindamycin tab and linezolid tab and still not treated me I wanna your advice please
Sir so in a pt of ckd with acute pulmonary edema and is having hypotension is it okay to use CPAP right sir ..some wrong concepts like CPAP would decrease bp exists in our college .. please clarify sir. Thank you.
don't trust me or someone, listen to them and then cross check with the books available,.. out of few clear indications on bipap /NIV, pulmonary odema is one the indiciations. hypotension you can manage with vasopressors.
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These 5 min change my life it made so much sense to me when you break it down like this
Glad it helped , thanks for your kind words .
Thanks sir
We are operating ventilators since many years but your videos help us to clear our concept and our ability and quality to manage patient in icu increasing day by day
I am icu incharge of multiespeciality hospital
Your videos help us me lot
Thank you very much
thanks a lot for your comments. glad to hear its helping.
thank you sir PERFECT explanation
Awesome, thank you sir
Great Sir. Explained very simplified way. With single class I got idea. Thank you Sir
You are most welcome. thanks
Amazing 👌
Very simple and short video.
These indications are absolutely correct in a healthy person, or at least in the absence of lung disease.
I just have one question:
What do you think happens to the right ventricular afterload after intubation in an ARDS patient with a significant proportion of atelectasis?
@@stefanofresilli2749 ards and atelactatsis … usually both same time difficult .. but anyways afterload increases on RV
Nice sir
EXCELLENT EXPLANATION SIR.THANK YOU SIR
Thank you sir for explaining. You made it easier to understand
My 2 hrs of research on this particular topic on internet has been summed up by you so beautifully.
I was so confused because i mixed up the physiological concepts.
Couldnt thank you more ❤ keep clearing our concepts sir ❤ big fan❤
I'm so glad it was helpful. thank you.
Good explanation,thankyou .
Thanks sir.। I remember once HOD sir asked this thing in seminar discussion.।
And we all residents were confused😵,
Thanks a lot for explaining such a simple way.।
Plz keep on posting such basic concepts videos,
Thanks to your venti. Videos also🙏
glad it helped.
Nice video...was able to recall the topic very easily...Thank you
Glad to hear that
The other important thing LVEDP decrease which help in heart failure
Nice 👍
Thanks ✌
great video. Just a quick question. I understand how the increased intrathoracic pressure will aid the LV contraction allowing better ejection of blood but I dont understand how that would lower afterload. In my thinking it would make it easier for the Left ventricle to reach and overcome the afterload and eject more blood systemically. But Isn't the value of afterload dependent on the systemic vascular resistance and systemic blood pressure. Thats the main thing that has always confused me.
When you have increased intrathoracic pressure and lower extrathoracic pressure... its easier for the LV to pump from high gradient to low gradient...
Can also think of it like increased thoracic pressure leads to less resistance in the proximal outlet tract and thus LV can push easily therefore decreased afterload. After load is a measurement of the pressure the LV has to generate to push out blood. hope that helps
Thank you so much sir
Most welcome
What is its clinical significance ?
Sir so during normal breathing systemic venous return increases and pt on NIV/Invasive Venous return decreases??
Yes, exactly
Ty for this vid but Sir, can you please make a video on RRT ??
Sir can you please help me?
I'm suffering from respiratory muscle weakness due to myotonic muscular dystrophy,,shortness of breath with sleeping problem but no doctor i find who help me and prescribed me any NIV device..
Sir can you guide me how to i get any NIV device and its setting for helpful to me for my breathing.. please sir please
Dear viewer, this is a medical educational channel not a medics advice … kindly contact a good physician or neurologist or super speciality hospital
@@TheICUChannel sir any doctor you know who prescribed this ??
How sir due to positive ventilation alveoli open wide so compress the vessel so resistance increase so aftee load to right ventrivle iincrease same due to vessel compression blood squce to left chamber so pre load increase but chronic it may reduce pre load yo left chamber
But how after load of left chsmber depent on perepheral resistance so we cant judge after load here am i correct
Hello dr
Iam making many test of sperm doctors says you have Streptoccocus aureus and they giving me clindamycin tab and linezolid tab
and still not treated me
I wanna your advice please
👍
Sir so in a pt of ckd with acute pulmonary edema and is having hypotension is it okay to use CPAP right sir ..some wrong concepts like CPAP would decrease bp exists in our college .. please clarify sir. Thank you.
don't trust me or someone, listen to them and then cross check with the books available,..
out of few clear indications on bipap /NIV, pulmonary odema is one the indiciations. hypotension you can manage with vasopressors.
Only afterload of RV increasing, also everything else decreasing☺