I just wanted to let u know, u have no idea how much these videos help medical students like me to help polish our basic concepts. Please do keep up the good work! I also love the exam question series as well (a kind request to do more of those)
I like the way you speak about this topic. Not only did you transfer the knowledge into my head, but also you made my day because of how excited and happy you are to teach this subject. Never lose that enthusiasm! Thank you!
When i don't understand lectures in my univ i just come and listen to your explanation ! i get big knowledge which helped me to pass my oral tests and QCM as well 🙏🏻 so whatever i say won't express how thankful i am to you 🥰
Awesome! Thanks heaps, I was struggling to understand my lecturer - searched RUclips for an explanation and found ya channel! Wow weeeeee, you make it simple to understand!!! Many thanks, from Dan in NZ 🙂
I am not a med student, I have mid thoracic dexocurvature this makes perfect sense as to why when I am lying on my back for too long my heart rate increases like crazy. I knew it was spinal related but most doctors in the U.S treat the body in parts instead as a whole. They look at me crazy as I say my scoliosis is triggering my heart increase.
Explained clearly... Thank u so much sir for making us understand very clearly ...keep doing more videos sir... We will support u definitely...No video is as understandable as ur videos
Mike, this was a perfect opportunity to address an entirely unaddressed neural regulation of the heart, via the necessary neuroanatomical distinction of the unmyelinated dorsal motor vagus and myelinated ventral vagus in the nucleus ambiguus. It is only the nucleus ambiguus which has RSA (respiratory sinus arrhythmia) not the dorsal branch of the vagus. These two branches ventral and dorsal vagus are phylogenetically distinct and hierarchically ordered. Essentially, there are two parasympathetic systems, one more ancient which we share with reptiles and one with the emergence of mammals In the nucleus ambiguus we have special visceral efferent fibers that connect as unified circuit five cranial nerves with the ventral vagus to form a unified circuit (V, VII, IX, X, XI) and two visceromotor pathways, one to the sinoatrial node of the heart and the other to the bronchi. We can refer this as our social emotional engagement system in our transition from a-social reptiles to social mammals. So our autonomic nervous and it neural regulation it is not just a two tiered forever Langley balanced motor system of antagonists, i.e., sympathetic-adrenal mobilization, fight/flight and parasympathetic rest and digest but rather a three tiered system. Supradiaphragmatic (i.e., our social-emotional ventral vagus system, trigeminal and striated facial muscles for smiling, vocalization, pharynx and larynx, vagus and head turning) which in fact regulates bi-directionally, afferent and efferent, heart rate variability measured of course through RSA (again only found here in the nucleus ambiguus). The later circuit (V, VII, IX, X and XI) are directly connected by the two visceromotor pathways, one to the sinoatrial node and other bronchi. The parasympathetic dorsal vagus (or vegetative vagus) is now more accurately distinguished from the later evolutionary maturation/neuroanatomical phylogenetic distinct ventral vagus nerve. Another way of functionally/operationally putting this is if our newer parasympathetic branch, ventral vagus, is optimally working (i.e., social-emotional engagement, looking, listening, vocalization) then our sympathetic-adrenal system is precisely not mobilized for defense but rather rather for play and engagement. However, following the Jacksonian principles of Dissolution applied here, if our later phylogenetically distinct myelinated ventral vagus (our social engagement/co-regulated system with our conspecifics) is not working then we become (or rather are) adaptively mobilized for sympathetic fight/flight defensive behaviors (and thus what is metaphorically referred to as our "ventral brake" if you will is removed). Consequently, increasing sympathetic-adrenal output regulation of the heart, etc. However, if we are in a state of withdrawal/shutdown and or dissociation, then our dorsal vagus (our vegetative vagus that we share and is dominant in reptiles but re-purposed in mammals especially humans) becomes adaptively mobilized as we then go into a more primitive reptilian state of feint or immobilization, which for humans (not reptiles as their oxygen needs are 80% less) is potentially lethal as we go into not only tachycardia but neurogenic bradycardia. Please update with Stephen Porges, Polyvagal Theory. Thanks
Fantastic explanation! I have a question, though. If for some odd reason a parasympathetic nerve fiber to the heart touched a sympathetic fiber to the heart, could the brain in principle make the heart pump more forcefully while attempting to slow it down instead?
So sympathetic nervous sytem has noradrenaline and parasympathetic has acetylcholine ? (how come it isn't adrenaline for sympathetic instead of noradrenaline)
You say at the end of the class that this is how the central nervous system tells the heart to speed up or slow down, but all parts of this video focus on elements of the peripheral nervous system. Can you explain this please?
Hey Doctor My heart is pumping like crazy at the wrong times I’m very healthy and have no anxiety so I’m wondering why it’s doing this Even the palpitations don’t cause anxiety so I’m wondering what I can do to slow my heart down. What do you reccomend ?
Not sure if you got an answer yet but it is "innervate", meaning when a nerve supplies a part of the body. So he is referring to nerves that supply the SA node in this particular video.
I don't understand what you are saying. Action potentials in specialized cells like SA node, AV node & purkinje cells happen because of Calcium not because of Sodium. Sodium only enter the cells through sodium funny channels & take the potential to threshold then calcium enter through slow L type channels & depolarized the cells. Sodium depolarization happens in atrial & ventricular cells.
😂 When your T1, T2, T3,T4,T5 Spine fiber Disc Fails=It is time to compress yr Heart Nerves= its os time yr Heart Beat is going to STOP soon.😊.Expected😅
I just wanted to let u know, u have no idea how much these videos help medical students like me to help polish our basic concepts. Please do keep up the good work! I also love the exam question series as well (a kind request to do more of those)
Affri Ashroff Thank you! That is very kind of you and we will make sure we do more exam questions! 😊
I like the way you speak about this topic. Not only did you transfer the knowledge into my head, but also you made my day because of how excited and happy you are to teach this subject. Never lose that enthusiasm! Thank you!
Always good to further explain the sympathic vs. the parasympatic effects of the heart. You explained this very clearly!
When i don't understand lectures in my univ i just come and listen to your explanation ! i get big knowledge which helped me to pass my oral tests and QCM as well 🙏🏻 so whatever i say won't express how thankful i am to you 🥰
First time watching you... Perfectly explained. I'm so lucky that I found this channel. Huge respects from Pakistan
i have my midterms coming up next week and ive been super nervous, your videos, although theyre old have been very helpful. I cannot thank you enough
Literally has everything I was looking for explained in a logical and easy to understand mannor. Excellent video, many thanks.
Thank you for explaining...
By reading book I caught not understand this topic... now it is clear... love from India 🇮🇳 ❤
Two concepts explained in one video, simply amazing!!!
Your videos literally save me, EVERY TIME!! Amazing explanations
Awesome explanation….. I was struggling with the textbook not at all getting the concept and I got this beautiful video in RUclips. Thank you so much
Awesome! Thanks heaps, I was struggling to understand my lecturer - searched RUclips for an explanation and found ya channel! Wow weeeeee, you make it simple to understand!!! Many thanks, from Dan in NZ 🙂
Conducting system description made easier ,very helpful,thnx doc
I am not a med student, I have mid thoracic dexocurvature this makes perfect sense as to why when I am lying on my back for too long my heart rate increases like crazy.
I knew it was spinal related but most doctors in the U.S treat the body in parts instead as a whole.
They look at me crazy as I say my scoliosis is triggering my heart increase.
wonderfully explained.
JUST WHAT I NEEDED.
This was the thing that I really need to know
Thank you
THANK YOU SO MUCH DR MIKE
HONESTLY
This video really helped me a lot! thank you.
Amazing ❤
Thank you sir❤
Explained clearly... Thank u so much sir for making us understand very clearly ...keep doing more videos sir... We will support u definitely...No video is as understandable as ur videos
best video on this topic, thank you!
Best video on RUclips
Mike, this was a perfect opportunity to address an entirely unaddressed neural regulation of the heart, via the necessary neuroanatomical distinction of the unmyelinated dorsal motor vagus and myelinated ventral vagus in the nucleus ambiguus. It is only the nucleus ambiguus which has RSA (respiratory sinus arrhythmia) not the dorsal branch of the vagus. These two branches ventral and dorsal vagus are phylogenetically distinct and hierarchically ordered.
Essentially, there are two parasympathetic systems, one more ancient which we share with reptiles and one with the emergence of mammals In the nucleus ambiguus we have special visceral efferent fibers that connect as unified circuit five cranial nerves with the ventral vagus to form a unified circuit (V, VII, IX, X, XI) and two visceromotor pathways, one to the sinoatrial node of the heart and the other to the bronchi. We can refer this as our social emotional engagement system in our transition from a-social reptiles to social mammals.
So our autonomic nervous and it neural regulation it is not just a two tiered forever Langley balanced motor system of antagonists, i.e., sympathetic-adrenal mobilization, fight/flight and parasympathetic rest and digest but rather a three tiered system. Supradiaphragmatic (i.e., our social-emotional ventral vagus system, trigeminal and striated facial muscles for smiling, vocalization, pharynx and larynx, vagus and head turning) which in fact regulates bi-directionally, afferent and efferent, heart rate variability measured of course through RSA (again only found here in the nucleus ambiguus). The later circuit (V, VII, IX, X and XI) are directly connected by the two visceromotor pathways, one to the sinoatrial node and other bronchi. The parasympathetic dorsal vagus (or vegetative vagus) is now more accurately distinguished from the later evolutionary maturation/neuroanatomical phylogenetic distinct ventral vagus nerve.
Another way of functionally/operationally putting this is if our newer parasympathetic branch, ventral vagus, is optimally working (i.e., social-emotional engagement, looking, listening, vocalization) then our sympathetic-adrenal system is precisely not mobilized for defense but rather rather for play and engagement. However, following the Jacksonian principles of Dissolution applied here, if our later phylogenetically distinct myelinated ventral vagus (our social engagement/co-regulated system with our conspecifics) is not working then we become (or rather are) adaptively mobilized for sympathetic fight/flight defensive behaviors (and thus what is metaphorically referred to as our "ventral brake" if you will is removed). Consequently, increasing sympathetic-adrenal output regulation of the heart, etc.
However, if we are in a state of withdrawal/shutdown and or dissociation, then our dorsal vagus (our vegetative vagus that we share and is dominant in reptiles but re-purposed in mammals especially humans) becomes adaptively mobilized as we then go into a more primitive reptilian state of feint or immobilization, which for humans (not reptiles as their oxygen needs are 80% less) is potentially lethal as we go into not only tachycardia but neurogenic bradycardia. Please update with Stephen Porges, Polyvagal Theory. Thanks
Well done u r number one
Thank you so much dr Mike you are the best ❤❤❤
Thank You Dr Mike .
Excellent explanation. Thanks a bunch
Extremely PERFECT.
This helped A lot
Thnkuuu very very much sir
This is amazing
Hoping for more and more videos🙏
thanks soo much ! Really good explanation !!
Absolutely the best.
amazing video as always!!
Marvelous 💜🤍
Sweet! Love this guy.
Fantastic explanation!
I have a question, though. If for some odd reason a parasympathetic nerve fiber to the heart touched a sympathetic fiber to the heart, could the brain in principle make the heart pump more forcefully while attempting to slow it down instead?
just outstanding.. if one video could make someone like subscribe and comment without requesting fr it, so here it is
I have vagus nerve activity and I forgot to ask the specialist that diagnosed me that what do I do to prevent issues and what am I susceptible too
Excellent dr.mike loved it
Thank you for everything ☺️
i love this, thankyouuu!
Great video!
Job done
Amazing. Thank you so so much 😭❤️
Thank you very much ✨
Thank you so much!
AMAZING 😍😍😍😍
☺️
Thanks sir
Thank you so much for these videos 🤟
Thanks Doc
Awesome explanation 👍👌
Very interesting - can you expand this into howsomeone with POTS could slow their hearts down?!
Nice explanation
Thank you
Untrue you can control it threw setting tempo, and releasing back into the automatic system. Ive been toying around with it the past few days.
❤❤❤🎉❤🎉❤❤😊 شكرا ليك كتييييييييييييييييييير
Would like to ask. Dose the right vagus Inhibit the VA node
So sympathetic nervous sytem has noradrenaline and parasympathetic has acetylcholine ? (how come it isn't adrenaline for sympathetic instead of noradrenaline)
Dr. Mike, how does this explanation tie in the the "little brain" now known to exist in the heart?
Does magnesium also plays any role in regulating heart beat
You say at the end of the class that this is how the central nervous system tells the heart to speed up or slow down, but all parts of this video focus on elements of the peripheral nervous system. Can you explain this please?
very helpful
Hey Doctor
My heart is pumping like crazy at the wrong times
I’m very healthy and have no anxiety so I’m wondering why it’s doing this
Even the palpitations don’t cause anxiety so I’m wondering what I can do to slow my heart down.
What do you reccomend ?
massage your carotid sinus
Thanks
thankyou, nice vedio
So is it true that heart can beat by itself without the brain how is that possible when the brain is regulating the speed of heart?
could i get a definition for what you mean by innovate? as in innovates the SA node?
Not sure if you got an answer yet but it is "innervate", meaning when a nerve supplies a part of the body. So he is referring to nerves that supply the SA node in this particular video.
@@yehia_s ah shit, I have in my assignment already. thanks though!
@@flaherty5090 Sorry I didn’t see your comment sooner, all the best with your assignment!
Thnxxxx❤💜❤💜❤💜❤💜
Okay I really would much rather pay you the 36k that's currently going to my uni
really though thank you so much
I don't understand what you are saying. Action potentials in specialized cells like SA node, AV node & purkinje cells happen because of Calcium not because of Sodium. Sodium only enter the cells through sodium funny channels & take the potential to threshold then calcium enter through slow L type channels & depolarized the cells. Sodium depolarization happens in atrial & ventricular cells.
People are watching these videos in uni???? i'm watching them for my A-levels xD
lmao same
AMAZINGGG
😊
I love you
👏
😂 When your T1, T2, T3,T4,T5 Spine fiber Disc Fails=It is time to compress yr Heart Nerves= its os time yr Heart Beat is going to STOP soon.😊.Expected😅
👍
I passed because you of
Online devised antropy
You be surprised how many MD's that laugh at Chiropractors and yet they have no understanding of this at all.
push the cam much further away, these are getting boring now.
Mark Adams why does the camera need to be pushed back?
Thank you