Renin-Angiotensin-Aldosterone System
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- Опубликовано: 8 сен 2024
- www.handwritten... - This tutorial explores the Renin-Angiotensin-Aldosterone System, its role in Blood Pressure, the enzymes, involved, and how drugs act upon the system. For more entirely FREE medical tutorials visit www.handwritten...
Aldosterone actually will cause sodium to be reabsorbed in the collecting duct by the creation and insertion of more sodium channels, and this will cause water to follow and be reabsorbed as well. ADH will cause aquaporin channels to be inserted into the collecting tubule and cause water reabsorption to occur
It also forms ENaC channel in the distal convoculated tubule..also aids in Na reabsorption
@@53deshantshah19 isliye neet pg tough in logo ki physiology jaha khatam hote h waha apni shuru Hoti h
Ur right respect
Fun fact, ACE doesn't actually "live in the lung" but in all blood vessels, most of the converting happens in the lung though because it is so highly vascularised
But the kidney is vascularised better than the lungs
My anatomy professor showed one of your videos in class and now I use them for studying and understanding the material for the exams! Thank you so much!
I love your videos they are short simple and precise. you help me a great deal when I am reading for my exams you save me from turning lots of pages, especially in exam fever mode. may God bless your work.
this is lovely but it is adh that has an effect on the permeability of the collecting ducts causing more water to be reabsorbed into the blood. Aldosterone creates more sodium channels in the apical membrane of the distal convoluted tubule and then increases the activity of the sodium potassium pump in the basolateral membrane of the blood vessel. This causes k to be secreted from the blood and Na+ to be reabsorbed into the blood.
Isis Pinner ..and wherever Na+ goes, H2O follows by osmosis
how about u make a vid
Hi, this is correct but very confusing for many people. To clarify Sartans are Angiotensin II receptor blockers that act on the AT1 receptor. Sometimes this receptor is called Angiotensin II-AT1 Receptor, to avoid this confusion.
Well explained in easy understandable way. Thank you.
Ahhh...wow! You explain so simply...it's such a blessing to all nursing/med students! You're such a God-sent!
I FUCKING LOVE YOU! I'd rather watch a 4 minute video about this and learn something. Than whole lecture of a professor knowing nonsense
Wow this is great !
Just wonderful, I have been researching "dietary management of kidney cancer" for a while now, and I think this has helped. You ever tried - Aenarter Uncomplicated Assistance - (do a search on google ) ? It is an awesome one of a kind guide for discovering how to help your kidney function without the hard work. Ive heard some decent things about it and my partner got great success with it.
Videos like these are why the internet is so freaking awesome
You r amazing thank you SOOOO much for all you doing for us this is really helpful and it does help me i have exam tomorrow you made my day ..keep going.
my nursing professor did a shitty job on teaching us this, and you made it so easy to understand, thank you so much!
Best explanation I have ever gotten.
this just helped me with my Anatomy test soooo much!
Thanks for this video, it's great audio quality and a very clear visual aid.
I’m studying autacoids and idk what I would’ve done without you
Amazing explanation thank you! I needed the clarification and now I'm able to follow my chart more carefully. God Bless You Happy Easter
Very simple to understand thank you
Love the neatness and handwriting
and yes the release of Aldosterone does increase the re-absorption of Na+ so by default H2O follows. thus the body excretes K+ and absorbs Na+ and H2O in the presence of Aldosterone.
Everything is so clear. Thank you!!!!
Amazingly clear. Thank you.
Great explanation using it to study for my pcat !!
I never understood that when I was in college! I owe you!
Aldosteron does not act on the collecting duct! It's on the DISTAL CONVOLUTED TUBULE!!
like this comment ppl its da truth
Florentina Krikela it acts on both the DCT and collecting ducts..
Idil Truth! It acts on both
Florentina Krikela You should really provide a source...
It acts in both 🙄
Yay!Mahalo for the well-drawn and well-detailed explanation.
what's the little diagram next to BP up? I can't figure it out?
Otherwise, this is the most clear and concise explanation that exists in the world today of this pathway.
Your videos are life saving, thanks!
Yes - both vasopressin (same as antidiuretic hormone (ADH)) and aldosterone work to increase blood pressure and blood volume. But by different mechanisms.
So, starting with aldosterone - this is released by the outer layer (cortex) of the adrenal gland which sits on top of of each kidney. Aldosterone causes increased sodium reabsorption into the blood and water follows it into the blood, thereby increasing blood volume and blood pressure.
thank you so much, I get intertwined in RAAS mechanism
Thank you! Concise and very helpful
This was on point. Thanks for your time.
Cheers mate, bless you and youtube, much nicer than reading textbooks =)
Great explanation, however it is ADH that aids in the retention of only water. Aldosterone aids in the retention of both "Na+" and "water"
Great explanation!
thank you so much! I finally understand the RAAS system!
lovely clearly explained video, thanks!
This would be a much greater video if it cut out the ACE drug info (maybe a brief mention of it) and focused on how this system is activated since it can become active in multiple ways. so if anyone actually reads these please make a new video that is only about the RAAS system. Its one of the most important systems in the body and deserves not to be overlooked.
Thanks!
Wow...very nice explanation 👍
Your videos are awesome. I think Sharpie should send you funds for advertising their markers! If you had time, I think another video explaining which electrolytes are affected in the tubules, loops, collecting duct etc as well as how diuretics, ACE-I, ARBs etc also affect these areas as well. Regardless, you've helped a poor PA graduate with hopefully passing her boards very soon!
Video is good..it helps me a lot in my exams...voice thdi clear hti bs...
Aldosterone causes sodium retention which causes osmosis and ADH increases the water permeability of collecting ducts, right?
Genius !
everything in one simple handwriting !
The RAAS system does NOT start with the liver’s production of angiotensinogen. It starts with the kidneys dectecting a decreased amount of perfusion which starts the secretion of Renin by the JXT cells, which then mix in with angiotensinogen and so forth. The name of this mechanism is also the order it follows as well, (1)RENIN-(2)ANGIOTENSIN-(3)ALDOSTERONE system.
I don't know who you are but I love you man thank youu
you should explain mechanism of aldosterone and which channel it upregulates (ENaC) so you can differentiate it from the effects of arginine vassopressin (AVP)
Great, explained very nicely
Don't forget Aliskiren, a direct renin inhibitor. Great video though!
I like your drawing of the liver - it's cute
wtf man hahahah..ur comment LOL
U need to take a break bruh
@@prateekbhunker2388 the comment is 6 year old you too need a break bro xd
WHAT 'a clear explanation thank uuuuu
Nice and clear and explained well conceptually......Thanks mate.
Superb overview
Gracías
Very clear and easy to understand. Thank you.
This is so professional ,thank you ,and please keep going the good work and don,t stop
very good sir thank you so much.
Great video! Quick question - you said that aldosterone acts on the collecting ducts of the nephron and this causes the nephron to retain water and this increases blood pressure. If the nephron retains more water then less is reabsorbed into the blood - so therefore how does blood pressure rise? You stated in your other video that aldosterone acts on the distal tubule (you didn't mention the collecting ducts) and you said it's action here was to increase the rate of water excretion? How can there be an increased rate of water excretion and an increase in blood pressure simultaneously?
Thats very concise and informative.
plz keep it up for use of Doctors serving the ill Humanity world wide.
Thanks A Lot.
Beautifully explained!!
thank you good sir.
would like to ask a stupid question:
wouldn't aldosterone also increase the reabsorption of Na+ and H2O in the distal tubule?
It does. This isn't a stupid question.
This is very helpful thanks but i have a suggestion if u use more colour its more useful and become cool
Nice video, sums it up really well.
Thank you so much, this helped me a lot
Thank you!
ANG II acts in the hypothalamus, where it stimulates ADH release and causes thirst!!
Hey ! Great video, it helped me a lot ! Please upload the PDF drawing on ur website !
Great video! However, there is one mistake towards the end of the video where you state that Sartans can inhibit the cardiac and vascular remodeling. This is actually the only thing that Sartans CAN NOT do when compared to ACE Inhibitors, thereby making ACE inhibitors more effective at targeting the effects of Angiotensin 2.
correct
That was awesome thanks
Very easy and helpful!
Thanks a lot good sir. Really helped clear things up
this is an awesome video - thanks so much :)
Really clear and good thank you!
Thankssss ,, it really helped alot .
this guy is brilliant!
very clear thanks !
Thanks for this tutorial!!!
thanks!!
Love the work you are doing, you have my support
Thanx man!
very very nice
very useful! Thanks much
thanks for posting!! i'd been looking for a good animation about this to better understand it, and this definitely helped! :)
Wow, thank you for taking the time for these videos! They're amazing!
was helpful thank you
I don't really understand how the relationship between Vassopressin/Adiuretin and Aldosteron is. They both seem to effect blood-pressure, thirst and h2o-resorption in a similar way. I woul be very thankful for some clarity:-)
My God THANK YOU! It was explained so weirdly in my uni presentations that I felt like a moron for not understanding it!
Thanks this video is great
how drugs that inhibit the renin-angiotensin-aldosterone system (RAAS) may reduce blood pressure.
can you explain me about this topic plzzzz
Perfect
regarding aldosterone, i know you're probably simplifying, but new students should not be confused. aldosterone makes collecting ducts retain sodium, while AT2 stimulates ADH release that inserts aquaporins in collecting ducts and makes water flow by osmosis back into the interstitium.
Thanks
This is FANTASTIC! Awesome job! Love that it is Australian as well, and not American with words pronounced oddly!
Would be AMAZING if you did a tute for this series on the "Triple Whammy" (effect of NSAIDs, diuretics and ACE-I) - I have searched the internet relentlessly and never found one! Please please please! Thank you x10000000 for your fabulous videos!
Nice video, i like it !
Great video!
OUTSTANDING...the only thing I had to learn in addition to your description is that it is the JGA in the kidneys that sense the drop in pressure and causes the release of Renin. Otherwise absolutely OUTSTANDING!!!
Aldosterone doesn't retain water.. it causes reabsorption of sodium.
Awesome! Thank you!