CORRECTION: 2:17 Written on screen is "IX" where it should be "XII", hypoglossal nerve (audio correct). Also on protrusion of the tongue, the deviation is towards the side of the lesion (audio incorrect). Thank you to the people who commented!
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amazing video but contralateral nerve palsies divides into central and peripheral and central includes only Facial nerve VII and Hypoglossal XII not IX, thanks
You have made everything so much easier to understand; thank you. However, you said somethings respecting the CN that are very confusing for me. When you referred to the CN Hypoglossal, the tongue will not deviate away from the weak side on the contrary. When the PT protrudes the tongue (CN XII), it will deviate towards the weak side. On the other hand, when you test the CN X Vagus," Say Ahhhh," Uvula will deviate away from the weak side. if I am wrong please let me know. Thanks again.
Thank you for the comment! CN XII (hypoglossal), upper motor neuron lesions lead to the tongue deviating away from the side of the lesion due to a decussation before the CN XII nuclei causing a weaker genioglossus muscle on the contralateral side (i.e a left sided UMN injury will cause weakness of the right genioglossus, therefore the tongue deviates to the right- away from the lesion). A lower motor neuron injury would instead lead to deviation towards the side of the injury (left sided LMN injury would lead to a weaker left genioglossus, causing the tongue to deviate to the left- towards the lesion). Hope that makes sense! Thanks again 😁
You're not wrong though that was a different concept conveyed. To put it simply, the injury of 12 cn, which is a lmn injury will deviate the tongue towards the weak side as the genoglossus muscle deviates the tongue to opposite direction. So when injured its "deviation" will be lifted and the tongue will be pointing to weak side. An umn injury will have opposite affect. It is different from the 10 cn in which uvula is deviated on the opposite side as the muscle supplying it pulls it ipsilateral, unlike genoglossus. Hope this makes sense.
I think thats because the lower motor neurones are still working and undergo an adaptation which causes these lower motor neurones to become more excitable.
bcoz when there is no control over the LMN bcoz UMN has lesioned the LMN becomes uninhibited and release more neurotransmitter (im not sure wch exactly) wch act on muscle and causes sapstic paralysis whereas on LMN lesion the neurotran cant reach muscle fuber and results in flaccid paralysis
90to95 decussate at the level of pyramid called lateral corticospinal tract and rest of few not decussate called anterior corticospinal tract Not like sir you told 80 and 20 percent
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CORRECTION:
2:17 Written on screen is "IX" where it should be "XII", hypoglossal nerve (audio correct). Also on protrusion of the tongue, the deviation is towards the side of the lesion (audio incorrect). Thank you to the people who commented!
I literally broke my head on this…then checked the comments section 😂
OMG I was the same! hahahah than you so much for this @@controversybuttrue1331
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Video Timestamps:
0:00 Pyramidal and Extrapyramidal Tracts
0:26 What do the Pyramidal Tracts do?
0:35 Pyramidal Tract - Corticospinal Tract
1:28 Pyramidal Tract - Corticobulbar Tract
2:40 What do the Extrapyramidal Tracts do?
3:04 Extrapyramidal Tract - Rubrospinal Tract
3:24 Extrapyramidal Tract - Reticulospinal Tract
4:00 Extrapyramidal Tract - Vestibulospinal Tract
4:22 Extrapyramidal Tract - Tectospinal Tract / Colliculospinal Tract
5:07 Pyramidal Tract Lesions - Signs of an Upper Motor Neuron Lesion
7:30 Extrapyramidal Tract Lesions
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Venepuncture Kit:
UK: amzn.to/3r7txrW
Pocket Cards: Lab Values / References / ECG / History Taking (Cheatsheets for rotations!)
US: amzn.to/3c3UybK
UK: amzn.to/3rd37W8
Suture Practice Kit (Complete kit with pad)
US: amzn.to/3c5ZJrN
UK: amzn.to/3vO76fh
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UK: amzn.to/3eZYoo5
(Affiliate links - We get a small percentage of sales, so if you buy anything, thank you!)
Thank you so much for this great illustration, simple and easy to understand
You're welcome, glad it was useful. Thanks for watching! 😁
Thank you for the amazing simplification, much appreciated
Thank you!
Thank you so much sir
Can you make detailed video on spinal injuries like brown sequard sydrome etc plz
amazing video but contralateral nerve palsies divides into central and peripheral and central includes only Facial nerve VII and Hypoglossal XII not IX, thanks
Correction has been added, thank you for the feedback and highlighting this! 🙂
And the motor aspect of the trigeminal
What an amazing resource! Thank you for sharing
Thank you! Always appreciate the feedback 😊
Watching this video today and the best video on internet explaining the brain tracts . Thank you so much❤
Now I understand the topic is not difficult but the problem is sources and now neuroanatomy makes sense for me ❤
Thank you so much for making this! It is extremely helpful for my upcoming exam!
Kabse hein? 🤔
Good sir, you can't imagine how much it helped. Thank you very, very much!
02:45 At lower left corner it is written IX which is wrong, it is XII hypoglossal*
Nice video, very well explained though
Well spotted, you have hawk eyes! Absolutely, audio is correct but picture should show XII. Thank you for commenting!
Amazing video thank you for helping me!
You're welcome!
Nice fast and imp explanation 👍🏻👍🏻
Thank you so much mate, big 👍🏾 from Papua New Guinea🇵🇬.
Amazing explanation! Makes so much sense, thank you! :)
just made things easier for me thank you for this video
You have made everything so much easier to understand; thank you. However, you said somethings respecting the CN that are very confusing for me. When you referred to the CN Hypoglossal, the tongue will not deviate away from the weak side on the contrary. When the PT protrudes the tongue (CN XII), it will deviate towards the weak side. On the other hand, when you test the CN X Vagus," Say Ahhhh," Uvula will deviate away from the weak side. if I am wrong please let me know. Thanks again.
Thank you for the comment! CN XII (hypoglossal), upper motor neuron lesions lead to the tongue deviating away from the side of the lesion due to a decussation before the CN XII nuclei causing a weaker genioglossus muscle on the contralateral side (i.e a left sided UMN injury will cause weakness of the right genioglossus, therefore the tongue deviates to the right- away from the lesion). A lower motor neuron injury would instead lead to deviation towards the side of the injury (left sided LMN injury would lead to a weaker left genioglossus, causing the tongue to deviate to the left- towards the lesion).
Hope that makes sense! Thanks again 😁
You're not wrong though that was a different concept conveyed. To put it simply, the injury of 12 cn, which is a lmn injury will deviate the tongue towards the weak side as the genoglossus muscle deviates the tongue to opposite direction. So when injured its "deviation" will be lifted and the tongue will be pointing to weak side. An umn injury will have opposite affect. It is different from the 10 cn in which uvula is deviated on the opposite side as the muscle supplying it pulls it ipsilateral, unlike genoglossus. Hope this makes sense.
Very nice good information
Thank you for ur energy, time u use to put up this video
Good one thanks 🙏🏻
thanks so much doctor
Thank you for watching 🙂
Dope👌👌👌😎😎😎
Thank you so much for the vivid illustration
Thank you so much !! You save my life. Your videos are easy to understand and make me see them in the big picture.
thankyou so much sir for this brief explanation
i m very happy that I found your video. Would be very happy if you explained upper limb and lower limb clincal anatomy
Very excellent ❤
Thanks a lot 😊
You should mention that the extrapyramidal tract is heavy modulated by the motor nuclei, including the basal ganglia.
very good video
Thanks
Wow wow wow 👏👏 thank you so much for this video Wonderful video, you have really helped me a lot🙏😭
simple and easy to understand, thank you so much!!!
Subscribed, this was a wonderful breakdown.
Thank you very much Alex
Hello just wanna to ask Rubrospinal tract control voluntary flexor movement isn't?
thank you so much, this was really helpful, hope you make more neurology lectuers
AMAZIIIIIIIING
very comprehensive! thank you!
Precise and informative 👍👏
المختصر💙
Can someone explain to me why people say extrapyramidal tracts control automatic movements if some of them control conscious movement
Why damage on pyramidal UMNs increase muscle tone?
I think thats because the lower motor neurones are still working and undergo an adaptation which causes these lower motor neurones to become more excitable.
bcoz when there is no control over the LMN bcoz UMN has lesioned the LMN becomes uninhibited and release more neurotransmitter (im not sure wch exactly) wch act on muscle and causes sapstic paralysis whereas on LMN lesion the neurotran cant reach muscle fuber and results in flaccid paralysis
Thanks for make it simple ❤️
But why the corticobulbar tracts are pyramidal tracts and they don't pass through the pyramids??
You sir, are amazing!
Thank you!
You are amazing, thanks a lot
Thank you!! 😀
Great video! But CN XII causes tongue deviation toward the side of the lesion, not to the opposite side
Thank you for your comment - you are correct, I will pin a correction :)
god bless you
References Please? which book or article state the pathway???
This video is just for explanations. U cannot be serious
I also want to know which book is referenced :]] from Vietnam
Thank you!
Nice video I suffer much difficulty to study,🥺🥺🥺🥺🥺please do the video about sensory and motor system
Brilliant video! 🙌
Thank you! Appreciate it!
Amazing, thank you!!
Does the medial vestibulospinal tract cross in the upper medulla and descend bilaterally?
Thank you
Hey buddy nice video !
Can you please make a complete video series on ascending & descending tracts of spinal cord
Hi Adam! Glad you enjoyed it, I have 2 other videos up on the ascending and descending tracts - I know they can be a pain to learn! All the best!
Perfect 🥰💕
Which reference was used in this video?
Please Can You tell Me The Reference You Used?
Beautiful
Thanks!
🤩🤩👌👌thanks
thank u soo much
Nn esiste la traduzione in italiano?
Thankss
No problem!
Very greatful 🤍🤍🤍
Thank you! 😁
nice
great
Please change ur pencil colour, light colour is difficult to see ,,
U can use another dark colour. Witch is good for visibility..
🎉 🙏
Tq
in my language
ap ne dariya ko kozay mein band krdea
🌜🫣 2:23 🌛
💚👍🏽
90to95 decussate at the level of pyramid called lateral corticospinal tract and rest of few not decussate called anterior corticospinal tract
Not like sir you told 80 and 20 percent
GReat
Not everything hundred percent true. Hypoglossal is CN XII not IX. And 90%of CST goes contralateral side (lateral) and 10% ipsilaterally (anterior)
This explanation is wrong, please be weary. Make sure to google properly from a proper source.
Doesn't make any sense ..fail to explain so many things
Thanks for make it simple ❤️
But why the corticobulbar tracts are pyramidal tracts and they don't pass through the pyramids??
Because it’s arise from pyramidal cells of the cortex