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Great explanation

Thank you 🙏🏻

So good for helping me understand the RAS/RAF pathway. Thank you!

I love this it clear I appreciate you for making it!!!

Please pay attention

Please pay attention to this. Thank u.

Immunology

H5 avian viruses bird flue

do you have a video on the JAK/STAT pathway?

you have no idea how much your videos are saving me in my molec & cell class... tysm for these

thanks

You really explained things in a very clear way. I really loved your videos. Thanks for sharing your expertise. Could you please make videos in Wnt signaling?

Your MHC series are very informative! Thank you so much for these.

More videos Tp53

I found this video and a lot of your other ones very helpful in PA school! The flow of your vidoes are very digestable and broken down into specific parts, which I have appreciated! Thank you for the help and keep up the good work!

thanks!

thanks alot :D

So happy to see you're back to uploading! I've been going through your videos religiously as someone who just started their masters in cell biology. Your teaching style is so effective, thank you for the work you're doing on your channel.

Please do a lecture series on porphyrias

Thank you very very much professor for these videos, your approach is a way that the first time I watched I couldn't make connections between concepts and It was unclear, after I studied more, and then watched your video, they perfectly wrapped everything I needed to know for these subjects.

Thank you for your amazing explanation! I have no background in the subject but you made it so clear ☺

Very clear explanation.

Very helpful, thank you!

Excellent!!! Superb explanation.

Sir but cip family inhibit E CDK n A CDK not D CDK

Wow, thanks. Your videos are excellent!!!

Thanks .Consice and useful

Thanks you

It’s really good

Joe you are the best, often I come back here to review it and understand it better. What became more clear this time is that once the Rb is off the transcription factor, two things can happen with a certain chance: 1.The transcription factor will encounter a new active non phosphorylated Rb. 2.The transcription factor will encounter rna polymerase and start transcription. Step 1 will become less likely once the concentration of active Rb goes down. Also what is funny is that rna polymerase itself is restricted by active Rb, so once we get a less active Rb there will come more rna polymerase available and step 2 will become more likely.

Great video! What’s the mechanism that the RNA polymerase can ignore the stop codon in the Cu to also transcribe the Cdelta segment?

I think there might be a little mistake in the somatic recombination of light chains: I think there is no deletion or inversion of DNA between the J and C segment(s), the J-C intron is transcribed and then spliced out of the mRNA transcript

Really helpful thank you !

Thanks

wow amazing explanation just subscribed! thank you for making learning free and accessible to all

Your explanations are great! You have no idea how useful these videos are, seriously thank you!!!

Sweet! This is exactly what I was looking for! Well done. Thank you!

Amazing

Great explanation. Thanks a lot for such an amazing video.😄

😊😊😊😊😊

very easy to understand, thank you!

Thank you so much. My wife has breast cancer and I am learning a lot from watching your videos. We are in the 21st century and there is science behind something other than chop and burn. Please keep making more.

Am happy

Thank you!

And are all D-type cyclin genes upregulated in cancer.

Could signalpathways PI3K/AKT/PTEN, Ras/Raf and Wnt make CCND2 upregulation ?

Thanks so much for the video! so helpful what textbook should we read to fully understand these topics?

thanks so much

thanks so much You are awesome man

9:55 What are the other signals that could make cell divide other than growth factors?