Thank you! you took very complex concepts and explained them in a way that makes them easy to grasp. I have a question related to a comment you made in 38:27 " During equalization your just correcting for error or zeroing and levelling of the transducer, and you are ensuring that both pressures are equally levelled, just inside the guide" so where is the right place for the wire sensor to be: Just outside, just inside of the guide, maybe in the middle?
Thank you for your lecture sir. I wonder where can i find the value of MI risk from all nonsignificant lesion '~2%' at 8 month at 15:46 time in your lecture?
This is from the initial FAME-2 trial publication, the non-significant arm follow-up results: De Bruyne B, Pijls N, Kalesan B, et al. Fractional flow reserve-guided PCI versus medical therapy in stable coronary disease. N Engl J Med 2012; 367: 991-1001. All the numbers from that slide are either FAME 1 or FAME 2 trials publications, as I indicate.
Why is it so that if FFR is 0.8, the pressure waveform of the distal coronary vessel loses the dicrotic notch and gets ventricularised ? I didn't get it...
I think he told as the distal pressure wire is facing the LV, 2-3 cm distal to the lesion, it should be ventricualrised and not related to the FFR value.
Ventricularization occurs whenever there is loss of communication between the aorta and the coronary wire. when hyperemia occurs there is flow turbulance past the stenosis due to which the "linear and stream line communication" between the pre-and post stenosis column of blood is lost. This causes pressure ventricularization
Thank you. If your question is regarding the effect of Impella on FFR, you may review my Impella talk ruclips.net/video/FF9C5VxIJZM/видео.html, minute 22:37. I cite 2 references, especially an important one that showed that Impella increases distal coronary pressure even past a significant stenosis (unlike IABP). Alqarqaz M, Basir M, Alaswad K, O’Neill W. Effects of Impella on Coronary Perfusion in Patients With Critical Coronary Artery Stenosis. Circ Cardiovasc Interv. 2018;11(4):e005870.
I have no words to express my gratitude to you. Please upload more videos. You are such an amazing teacher
Sincere thanks to Dr Hanna . Explained very well. Being an engineer i am able to understand these topics very clearly.
Thank you very much Dr Hanna
Excellent teaching as usual
Please continue to teach us more tricks in interventional cardiology
I thought ffr and ifr were that simple. After watching this video, i realize i know nothing. Thank u sir 🙏🙏
we all thank god that sent you sir to help us to master intervention cardiology graduated from your school .
Thank you immensely Dr Hanna .. the session is most valuable and brilliantly explained.
Simply the best lecture
Thanks for such a wonderful thorough and well explained lecture
Perfect lecture!
very useful and instructive , your hard work is not in vain sir
The Video we were all waiting for Thank u so much
Very helpful ,appreciate the effort&the sweat and blood went into it.quite surprising to find such quality material for no charges.
Big salute 🫡🫡🫡
Thank you! you took very complex concepts and explained them in a way that makes them easy to grasp. I have a question related to a comment you made in 38:27 " During equalization your just correcting for error or zeroing and levelling of the transducer, and you are ensuring that both pressures are equally levelled, just inside the guide" so where is the right place for the wire sensor to be: Just outside, just inside of the guide, maybe in the middle?
I place it just at the inside of guide, barely
Beautifully explained 👍
Amazing lecture sir as usual , am so thankfull to u sir
Thank you for the amazing lecture
Wonderful presentation, sir
Great great talk.
Thank you for your lecture sir.
I wonder where can i find the value of MI risk from all nonsignificant lesion '~2%' at 8 month at 15:46 time in your lecture?
This is from the initial FAME-2 trial publication, the non-significant arm follow-up results: De Bruyne B, Pijls N, Kalesan B, et al. Fractional flow reserve-guided
PCI versus medical therapy in stable coronary disease. N Engl J Med 2012; 367:
991-1001.
All the numbers from that slide are either FAME 1 or FAME 2 trials publications, as I indicate.
thank you sir ❤
Why is it so that if FFR is 0.8, the pressure waveform of the distal coronary vessel loses the dicrotic notch and gets ventricularised ? I didn't get it...
I think he told as the distal pressure wire is facing the LV, 2-3 cm distal to the lesion, it should be ventricualrised and not related to the FFR value.
Ventricularization occurs whenever there is loss of communication between the aorta and the coronary wire. when hyperemia occurs there is flow turbulance past the stenosis due to which the "linear and stream line communication" between the pre-and post stenosis column of blood is lost. This causes pressure ventricularization
I explain it verbally under 7:16, this is my preferred explanation:
"When FFR is
Perfect knowledge sharing. Thanks for uploading here. Do you have more details on Impella working or can share a link?
Thank you. If your question is regarding the effect of Impella on FFR, you may review my Impella talk ruclips.net/video/FF9C5VxIJZM/видео.html, minute 22:37. I cite 2 references, especially an important one that showed that Impella increases distal coronary pressure even past a significant stenosis (unlike IABP).
Alqarqaz M, Basir M, Alaswad K, O’Neill W. Effects of Impella on Coronary Perfusion in Patients With Critical Coronary Artery Stenosis. Circ Cardiovasc Interv. 2018;11(4):e005870.
Very conceftual
O'Connell Passage