Classic Case: ABPA
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- Опубликовано: 9 май 2022
- This is a classic case of ABPA.
Here is a link to the article I mentioned in the video:
pubmed.ncbi.nlm.nih.gov/23593402/
Textbooks I like for chest radiology-
Med students and all residents: Felson’s Principles of Chest Roentgenology
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Radiology residents: Thoracic Imaging: Pulmonary and Cardiovascular Radiology
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Thoracic radiology fellows: Muller’s Imaging of the Chest: Expert Radiology Series
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Classic finger-in-glove! Dr. Agrawal, if this finding wasn't present, could you still make the diagnosis of ABPA in this patient?
Great question, I wish I mentioned this in the video. You don't need to have mucus plugging (either high or low attenuation) to call ABPA. Bronchiectasis in a patient with asthma is enough to suggest it.
There is an entity called ABPA - S, where patients don't have any HRCT findings,but serology for Aspergillus fumigatus is positive.The other entities are ABPA - B ( With Bronchiectasis),ABPA - HAM(With High attenuation mucus plugging, which is pathognomic of ABPA)
@@vishnuchaitanya1835 thanks that is very helpful!
excellent explanation, great teacher, thank you!
This is really helpful. Thanks a lot!
Loads of love ❤️❤️
Great case !
Thank you Sir 🙏
Nice explanation sir..
thank you so much
may you start series of lectures about nodular lung disease
Here you go: ruclips.net/video/TYiERbT9X_s/видео.html
Great vid. Thanks
Please make video on vasculitis. Thank you
nice explanation
amazing
Sir what is exact haunts field unit attenuation value of thick mucous secretion in abpa or just we will corelate with muscles hu???
I have a question sir….thick secretion are produced by fungus or cystic fibrosis….?
Sir its mean we can also find low attenuation mucous plugging in a patient of cystic fibrosis with abpa ???because in this case still no deposition of calcium and manganese salt in mucous…….am i right sir????????
Would you please explain the finding of chronic necrotizing aspergillosis, and we'll know that bronchiectasis worsening is related to that.
Many thanks
chronic necrotizing aspergillosis is an indolent infection, very often cavitary. Patients are not severely immunocompromised like those who get angioinvasive aspergillus, but they may have chronic liver failure or some other chronic condition. The lung often has pre-existing disease like fibrosis from sarcoid or some other such process that alters the lung architecture.
Sir why abpa is seen inside patient of asthma and cf??????can it occur without these?????
Kindly, the sign of finger-in-glove is perplexing from the adjacent hilum. Are there any helpful clues to resolve this dilemma?
Hi, the main thing is that it does not taper like the surrounding vessels. It is much thicker than the vessels surrounding it.
What are the treatments for ABPA? I have a 4cm mass and MD wants to give high does steroids over two months. I heard about Anti Fungal meds to lower the steroid dosage. ? any ideas????
What do lungs look like with Mycoplasma and Lyme. Thanks
Mycoplasma, what you see are thickening of the normal lines in the lungs and cloudy lesions. It usually affects both lungs. Lyme, not sure. I don't think I have seen a case before of Lyme affecting the lungs.
You mean to say if mucous secretion is of high attenuation its case of cystic fibrosis with abpa…..?if mucous is of low attenuation than other underlying cause except cf?
Yes, in a patient with CF, high attenuation mucus plugging means they also have ABPA. If it is low attenuation mucus plugging, it is nonspecific. It could be ABPA but may just be mucus plugging from CF.
@@ThoracicRadiology respected sir i m still confused?
@@naeemasr5022 hi, I edited my previous reply. Let me know if you would still like clarification. Thanks for your question!
Y u so amazing?
I have ABPA , it’s horrible just horrible, it’s debilitating
Is there not any treatment for it? You just have to live with it?