Hi - watch all of the videos and found them extremely helpful while relearning to be able to work in CCU. I will at all times recommend these to new coworkers as they are very well explained and thorough and also easy to understand. Thank you so much! Susanne
I'm so glad you like them Susanne and so great to hear they are helpful for you. I really appreciate you spreading the word and sharing them. Thank you so much!
This series was amazing! So illuminating to hear from a great teacher like you, looking forward to continuing to work through all the videos on the channel
Even though I currently work in post-partum as a RN, I watched every video in this series! It's definitely not the ICU where I work, but I have actually seen early sepsis, some hypovolemic shock, anaphylaxis, and even a combination of neurogenic shock and hypovolemic shock! It was well worth my time. I plan on watching more videos. Thank you!
Yes! We actually had an OB nurse come to our ICU years ago and she was a rockstar. You guys really do see so much. Glad to see you are growing and learning and happy to know you enjoyed the video!
Starting residency in Anesthesiology and Intensive Care next month. Shock playlist done, onto every single video you made, as they are too great to just pass on. Hope you are well during this troublesome times in healthcare and thank you!
Hello, thank you so much for this explanation. I have a question, in cardiogenic shock, it makes sense that cpv and pcwp are increased in patient with decreased contractility such as mi. this is due to blood accumulating in the heart. But what about cardiogenic shock due to severe tachycardia where there is a decrease in diastolic filling time and preload. shouldnt the cpv and pcwp be decreased in such cases??
Excellent series! And this chart has been so helpful in my revision for a postgrad certificate in Intensive Care Advancing Practice. I'll be wearing my My ICU advantage tshirt for graduation! Thank you!
Thank you so much for these videos... I watched them all and every one of them were extremely well explained and helpful. Where does Obstructive shock (PE) fall into this? I can try and deduce it myself, but it would great if you can send the answer. Thanks again!
I'm assuming you are talking about the table used to help identify what type of shock we are dealing with? If so, clinically Obstructive shock is going to present itself usually just like Cardiogenic shock. In fact, many people just consider the obstructive shocks as types of cardiogenic shock. Hope that answers your question and thank you for the nice words and taking the time to leave a comment!
The body attempts to redistribute blood from lesser important organs. The periphery is one area that it will clamp down small vessels and shunt blood away from skin to preserve it for the core and larger vessels that is needed more.
Great question... so there is so many competing thoughts on this subject, even to the point of saying our SvO2 is no measure of tissue perfusion. I tend to side with the thinking that if we are truly in a shock state, then we have inadequate perfusion and thus less oxygen being delivered than we are consuming. To make up for this, the tissues extract MORE oxygen from the blood as it comes by, leaving us with a lower SvO2. There are talks about septic shock being "hyper dynamic" which is true as early sepsis we have increased contraction and HR, but if we are saying we are in a shock state, we still are not meeting demands with perfusion. Now as septic shock progress, for various reasons, we lose the ability to extract O2 at the tissue level. This causes cells to go in to anaerobic respiration and we begin to see our lactate climb. But, since the tissue is not extracting that O2 now, more of it is returning and this the higher SvO2. Hope that makes sense and glad to hear my videos have been helping you.
Hey sorry I missed this comment immediately. If you go the the "Community" tab from the main ICU Advantage RUclips channel, look for the most recent post with the link to the Dropbox and the most current password.
I really like this shock series. But I don't understand that why in Septic shock SVO2 decreases in the beginning and increases in later stages? Can someone answer it for me? THANKS!
Glad you liked it. It doesn't ALWAYS happen this way, but basically, early on in sepsis, we have a hyper metabolic state leading to increased O2 usage and thus decreased SVO2. Then as shock develops and fluid builds, less diffusion and O2 extraction can take place and thus the SVO2 runs high as the body cannot extract and use the O2. Make sense?
I learned more from this video series than I learned in a whole day of lecture in class. Thank you!
Wow, happy to have been able to help!
Hi - watch all of the videos and found them extremely helpful while relearning to be able to work in CCU. I will at all times recommend these to new coworkers as they are very well explained and thorough and also easy to understand.
Thank you so much!
Susanne
I'm so glad you like them Susanne and so great to hear they are helpful for you. I really appreciate you spreading the word and sharing them. Thank you so much!
This series was amazing! So illuminating to hear from a great teacher like you, looking forward to continuing to work through all the videos on the channel
Thank you! You've got a LOT to get through these days lol
Even though I currently work in post-partum as a RN, I watched every video in this series! It's definitely not the ICU where I work, but I have actually seen early sepsis, some hypovolemic shock, anaphylaxis, and even a combination of neurogenic shock and hypovolemic shock! It was well worth my time. I plan on watching more videos. Thank you!
Yes! We actually had an OB nurse come to our ICU years ago and she was a rockstar. You guys really do see so much. Glad to see you are growing and learning and happy to know you enjoyed the video!
Absolutely love every video from this playlist! So easy to understand and to remember. Thanks a lot! Now i wanna watch every video on your channel!
Thank you so much Liza! Really glad to hear you liked the series. Hope you enjoy the other videos. Quite a few for you to watch. 😊
Starting residency in Anesthesiology and Intensive Care next month. Shock playlist done, onto every single video you made, as they are too great to just pass on. Hope you are well during this troublesome times in healthcare and thank you!
Awesome! Congrats on the residency and glad you found the channel and are finding the videos helpful
You are absolutely an amazing teacher thank you so much
Wow, thank you! I really appreciate that and glad you liked it!
Great series, thank you for putting in the work!
ABSOLUTELY AMAZING SERIES THANK YOU!!!!!
YAY! This is so awesome to hear Kristen! Glad you liked it and thanks so much for taking the time to leave a comment!
Love your videos! Just found your channel today and it's gonna be a huge help for me med surg exam tomorrow
Thank you very much, very detailed, very helpful
Hello, thank you so much for this explanation. I have a question, in cardiogenic shock, it makes sense that cpv and pcwp are increased in patient with decreased contractility such as mi. this is due to blood accumulating in the heart. But what about cardiogenic shock due to severe tachycardia where there is a decrease in diastolic filling time and preload. shouldnt the cpv and pcwp be decreased in such cases??
Excellent series! And this chart has been so helpful in my revision for a postgrad certificate in Intensive Care Advancing Practice. I'll be wearing my My ICU advantage tshirt for graduation! Thank you!
Thank you so much! And how cool!!! Congrats on graduation and thank you so much for the support with the shirt purchase!
Great ending to an amazing series 🎉
Thank you very much! Seemed like a good way to bring it all together. :)
This was just amazing, so helpful.
Thank you for this and glad you enjoyed the video!
Thank you for this series! I have a test on shock next week. 😌
I'm late getting to comments! I bet you already took it. You'll have to let me know how you did!
Thank you for this! - Manila, Philippines
You’re welcome! 😊
very impressive i ever lisen this type of lacture in my life thanks for giveing us very informative lacture.
Thank you Hassnain! Glad you liked it.
Thank you so much for these videos... I watched them all and every one of them were extremely well explained and helpful. Where does Obstructive shock (PE) fall into this? I can try and deduce it myself, but it would great if you can send the answer. Thanks again!
I'm assuming you are talking about the table used to help identify what type of shock we are dealing with? If so, clinically Obstructive shock is going to present itself usually just like Cardiogenic shock. In fact, many people just consider the obstructive shocks as types of cardiogenic shock. Hope that answers your question and thank you for the nice words and taking the time to leave a comment!
Really useful summary.
Awesome to hear!
this guys gotta be really good at taking notes
😂
Simple very informative
Glad you think so!
I like your "All right!" pronunciation:))
Thank you so much! I think AACN needs to switch their videos to yours :))
I agree! ;)
Thank you.
You're welcome!
U said in septic shock there will be decrease in SVR, then how come skin becomes cold later on in septic shock??
The body attempts to redistribute blood from lesser important organs. The periphery is one area that it will clamp down small vessels and shunt blood away from skin to preserve it for the core and larger vessels that is needed more.
cuz da levo
You are super man
In early septic shock, isn't SVO2 initially elevated?
Thank you for the videos! Helping me out a lot in my critical care class!
Great question... so there is so many competing thoughts on this subject, even to the point of saying our SvO2 is no measure of tissue perfusion.
I tend to side with the thinking that if we are truly in a shock state, then we have inadequate perfusion and thus less oxygen being delivered than we are consuming. To make up for this, the tissues extract MORE oxygen from the blood as it comes by, leaving us with a lower SvO2. There are talks about septic shock being "hyper dynamic" which is true as early sepsis we have increased contraction and HR, but if we are saying we are in a shock state, we still are not meeting demands with perfusion.
Now as septic shock progress, for various reasons, we lose the ability to extract O2 at the tissue level. This causes cells to go in to anaerobic respiration and we begin to see our lactate climb. But, since the tissue is not extracting that O2 now, more of it is returning and this the higher SvO2.
Hope that makes sense and glad to hear my videos have been helping you.
@@ICUAdvantageو
Awesome video!
Glad you enjoyed it
How can I access lesson notes.?
Hey sorry I missed this comment immediately. If you go the the "Community" tab from the main ICU Advantage RUclips channel, look for the most recent post with the link to the Dropbox and the most current password.
I really like this shock series. But I don't understand that why in Septic shock SVO2 decreases in the beginning and increases in later stages? Can someone answer it for me? THANKS!
Glad you liked it. It doesn't ALWAYS happen this way, but basically, early on in sepsis, we have a hyper metabolic state leading to increased O2 usage and thus decreased SVO2. Then as shock develops and fluid builds, less diffusion and O2 extraction can take place and thus the SVO2 runs high as the body cannot extract and use the O2. Make sense?
Thank you so much!!
You are very welcome Sean!
Thank you so much 😊
You're welcome Tasha! 😊
amazing videos
thank u alot 💙💙💙
Thank you so much! Thats so awesome to hear. Glad you like them Yusur!
very helpful
Awesome! 😊
How long can a person live with septic shock? Life expectancy
Thaaaanx
Add a public comment... sorry.
very useful
Very helpful
Glad to hear that!
Very helpful
Glad it helped