CARDIOVASCULAR DRUGS; DIGITALIS & ANTI ARRYTHMIC DRUGS by Professor Fink
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- Опубликовано: 10 июн 2013
- In this video lecture, Professor Fink reviews the Cardiac Glycosides (digoxin; digitalis) and then the Cardiac Antiarrythmics (quinidine & lidocaine). He then reviews the actions of Nitroglycerin and the drug-drug interaction with sildenafil (Viagra).
Check-out professor fink's web-site for additional resources in Biology, Anatomy, Physiology & Pharmacology: www.professorfink.com
Down-loadable e-Books of the Lecture Outlines by Professor Fink can be purchased from the WLAC Bookstore at: wlac.redshelf.com/
“Hard Copy” Lecture Outlines can be purchased from the WLAC Bookstore at: onlinestore.wlac.edu/fink.asp
You're students are blessed to have you!
FINK!! I took your biology and anatomy classes years ago and am now going back to your videos to brush up on Pharm for my NCLEX!
Keep up the excellent work!
Thank you for explaining this better than any of my professors have.
Thank you so much.
2020 and he’s still the best !
Dr. Fink is through , technical and analytical in the conceptions of pharmacological sciences . These lectures provide excellent applications toward my P.hd .
He just saved my semester
Wow, you make things SO easy to digest! You have a gift. Thank you for sharing it!
This video is amazing!! I am studying for my Canadian Registered Nursing Exam in June and this has been a great review. Thanks a ton :)
Wow this guy's awesome, he actually seems to give a shit. Nothing at all like my teacher.
you are the man, Professor.
Great explaining
Thank you very much Sir .
you are the best one !!!!!!!!
Excellent lecture and teacher as the usual 👍👌
Quick question: Since decreasing myocardial contractility leads to a decreased CO, wouldn’t using antiarrythmics in general, and Lidocaine especially, lower the CO ? Since they slow down heart rate, and especially with Lidocaine, there is a decrease in contractiliy due to blocked action potentials in all excitable cells, including the heart muscle …
This is a bit confusing, when we know one of the causes of decreased CO and Heart Failure are the cardiac arrhythmias…
The lidocaine is administered to reduce & prevent abnormal electrical activity of the heart (such as ectopic foci; PVCs). The dose should not cause slowing of normal RESTING Heart Rate. Similarly, a "Beta Blocker" is used to reduce & prevent increased heart rate related to sympathetic stress response, but should not cause slowing of normal RESTING Heart Rate.
Hello everyone! Guys, if there is anyone who is studying in US med uni, could you please comment down this comment. I would really be grateful if someone would share with me "lecture notes" or similar notes that uni professors share with you. (Not the common books like Robbins...). I would love to see comments here. Thank you so much sir Fink as well.💯
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Professor Fink, Professor Fink, he'll make you laugh, he'll make you think.