Pathophysiology of Diastolic and Systolic CHF
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- Опубликовано: 7 сен 2024
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Lecture on the pathophysiology of congestive heart failure, including a discussion of cardiac remodeling, and the differences between systolic and diastolic heart failure.
Wow!! RN student here....God will SURELY bless you for making these videos available!!! The light bulb just went on...I can't thank you enough....I am in my last semester of school, and have used many of your videos to get me through...again, thank you!
Thanks so much for sharing your feedback Sherrie!
I am a last semester RN student. Thank you for your videos! I am a visual learner and your videos have helped me tremendously. You have just covered a whole chapter for me that I now interpret and understand in 21:17 minutes/seconds. Thank you again for all of the insightful information on your videos! They have helped me immensely.
So glad to hear Health Ed Solutions was helpful!
wonder if you ever got the RN license?
4th year pharm student, holy shit this was the best damn 20 min review of patho chf, way more understandable and to the point than others
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Awesome review and discussion of CHF. I'm a nursing student, and your lecture has been extremely insightful for my research. Thx a bunch.
I don't know you, but my God I respect you. Thanks for creating this video.
You are able to take complex medical information and break it down to a clinically relevant format, that is easily assimilated and can be retained for the long term, fantastic job, thank you for all your effort.
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With much thanks and gratitude!! Your lectures are pulling me through RN school!! Thanks for all your time and all you do
This is so great. I have worked on Cardiac units for 12 years This is so well illustrated. It helps to have taken care of hundreds of these patients also but this puts the physiologic dynamics into place. Keep it up don't change a thing. Speech cadence is wonderful and easy to listen to.
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yet another nursing student who relies on Khan Academy to clear things up... sometime the instructors breeze through this stuff so fast, students can't get an understanding. These videos are truly a lifesaver (and a grade saver)... and an RN maker!!!
Awesome to hear! Glad Health Ed Solutions could help :)
Much more understandable and to the point than the 3 hour lecture which covered the same topic in class.
Thanks!
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You have the best videos and always look for you on youtube. Thank you so much for all your videos.
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The way you explain things are wonderful!
Very thorough as presented the concepts integrated the visual diagram with clear concise explanation of all processes.
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sir, you have been the best teacher so far. your online lectures on youtube are amazingly awesome. there is a request for more pathophysiology lectures on mitral valve stenosis , murmurs , pulmonary hypertension and some other common congenital conditions. your lectures are helping me a lot! thank you !
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Great video...graduate student....you have definitely increased my understanding of systolic versus diastolic. Great job and thank you so much!
u give excellent explanations every time i look up....makes all complicated topics simple.Really lov ur lectures.Keep uploading .
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
Very helpful, studying for my Nursing exam and you clarified a lot for me. Thanks for sharing!
Glad it helped Annie!
This video is great! I am a RN student looking for some clarity about the CHF and I definitely found it! Thanks so much, Andrew!
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Fantastic explanation of pathophys of CHF! Makes it so easy to understand!
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GREAT lecturer. Concepts are broken down to be understood easily plus the board and colors make it easy to follow.
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Thanks so much for these videos - it really helps as I only get a brief overview in lecture due to time constraints over summer semester. Your video really help me with my critical care respiratory lecture and I'm sure it will do the same for the up coming cardio lecture.
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This video was very helpful. Thank you for your dedication to teaching.
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You're awesome and your lectures are saving my butt in EMT school.
Great, easy to understand explanations, with an adequate amount of depth to trigger memory of the physiology. I really like the "Ink" drawings...great teaching tool (my A&P instructor used it very effectively, also)! I'm in my last semester of a BSN program, and I'm always looking for really well-done lectures for review. Thanks!
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Thank you for posting this. This video helps me to enhance my memory and understanding after reading my Med-Surg textbook. Thank you so much. :)
nice and simple easy to understand explanation of CHF thanks
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Really good video-lecture! Simple, str8 to the point, usefull for the medical student but even for somebody specializing in cardio, as a review. Thanx!
Nice video, but it needs to explain why output is low in diastolic heart failure. I think Rajiv diastolic heart failure video really gets it right
Thanks for the feedback! We will be rolling out new lessons soon. Thanks for watching.
Thank you for your video. Can you please make more videos on pathophysiology, symptoms, signs ,investigations and treatments of diseases according to the systems?
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
Very helpful. I almost watched many of your video lectures on youtube and all are awesome. Thanks Andrew.
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
Thank you for saving my life in medschool. A fan all the way from Egypt :)
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Thank you very much for posting this, as a RN student I'd always struggled with cardio-pathophysiology so this was very helpful. You have a great way of explaining things :)
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Hey there. The protein that Andrew is talking about refers to albumin and other blood proteins. By changing the amount of protein in your diet , you don't actually change your blood protein levels as they are all regulated quite strictly.
A low sodium diet would be more beneficial in a HF patient. Lower sodium can result in lower blood pressure, lower afterload and potentially fewer HF symptoms.
A million thanks from the bottom of my heart❤
Thank you Nubia! You are so welcome.
Your videos are fantastic. Thank you so much!
Appreciate that!
Hello,this video is really very well explained thankyou for posting! I did not understand edema formation in case of diastolic failure can you please explain it?
Very informational with great visual aids -- very helpful!
Thanks so much
Very helpful! Thank you Andrew
Another great video, Andrew. Thank you!
Keep watching, we appreciate the support!
Thank you for ur videos! I find it all great and very understandable. I'd like to comment about the carotid body that stimulated by decrease blood pressure. If I'm not mistaken, receptor that response to pressure changes is baroreceptor which is carotid sinus in the proximal of internal carotid artery, and to chemical changes is chemoreceptor which are carotid body and in the aortic arc. So I guess you mean carotid sinus, didnt u?
:)
I believe this depends on where you are in the body. For example, if you're running from danger the Sympathetic NS will cause vasodilation in skeletal muscles to get more blood to them, but you'll see vasoconstriction in your digestive organs, etc. since you don't need them to be working as much at the moment.
Great video! Thanks Andrew! Your videos are really a great help on my upcoming exam. :) God bless you!
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More on ace inhibitor effects please!
thanks so much mr wolf!! thats some messy congestion going on. thanks for explaining it! had to concentrate really hard to follow everything you were saying haha but very thankful nonetheless :) lol and the details for how oedema occurs now makes so much sense :) haha *thumbs up!*
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Great lecture!
I understand the difference between systolic and diastolic HF but I start to get confused when they talk about the two types of diastolic failure - decreased compliance and decreased distensibility. Any insight? They all start to sound the same to me.
Great vid. What program do you use to write like this and record it?
Great lecture.
Which Angiotensin (I or II) travels to myocardium and remodels it? 4:00 - 4:15
must be 2 if ACEi is used to tx it
This was really helpful! Thank you very much
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nice channel. thanx for the explanation u give in this video.
Many thanks Wolf, you save my ass in final exams in med school. Big fan. Your channel link is not included its mention. And pls could you do a lecture on GII
Very nicely explained!
Thank you!
Very thorough and
Hi Andrew. Thanks for your fine insight into CHF. I am a paramedic student doing a paper on the subject for a Pathophysiology course and am enjoying learning more about the subject and the cardiovascular system in general.
This point you outline about what is happening in systemic flow at the capilary level, with oncotic pressure and it's role in increased venous pressure makes me wonder if there is value for CHF patients to consider low protein diets, is this something that is recomended?
Keep watching, we appreciate the support!
that at least some reduction in diastolic pressure and peripheral edema would be acheived.
great lecture! thanks for this
i think what you said about diastolic dysfunction cause is wrong, hypertension causes systolic dysfunction because the LV has to exert much higher power to overcome the pressure in the aorta and open in it. thus, hypertension causes systolic dysfunction.
correct me if i'm wrong.
much obliged
Does the diastolic CHF also result when the mitral valve regurgitates? This also comes with overall fatigue and a cough (often to begin with in the morning)
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@@HealthEdSolutions look forward to your reply.
Great review!! Thanks so much!
Low protein levels would put less strain on kidneys and liver. Typical diets high in protein include a lot of meat and significant fat and cholesteral tends to be along for the ride which will likely challenge the heart with further arteriosclerosis, a possible underlying culprit. As for fluid, with less protein in the vasculature there'd be less oncotic pressure at the capilaries and so less fluid pressure on the venous side. If I'm on the right track then it would be logical to expect..
Thank you for your exellent video Andrew. Could you please make some videos about pharmacokinetics or pharmacodynamics if you have time ;)
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
Yamazing video and amazing explanations. Thank you a lot
Appreciate it!
Will u please make and upload some lectures over digestive system( including the liver) and some of the main GI diseases and their related treatments
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
What about jugular vein distention? Is this specific to right- or left-sided CHF?
Thank you for the lecture, can you please explain the edema that the liver has in RHF, is it caused my increase in oncotic pressure?
Hi there! Dr. Wolf and the rest of the team here at Health Ed Solutions will be releasing new content weekly starting very soon :)
No, it does the exact opposite. It activates smooth muscle activity of the arterioles resulting in vasoconstriction
thank you! this is really useful!
Okay thank you !!
Thank you for the video
You r excellent, thank you sooooo much, The heart facinates me !!
You are just amazing
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Thank you for making this video. This is a great Lecture, you have made it easier to understand! :)
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Excellent. Very clear.
Great video for nurses! But not detailed enough for Medstudents/doctors :(
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Very educative. pls upload more videos
Thanks for the feedback! We will be rolling out new lessons soon. Thanks for watching.
That face of a boy with glasses is really cute. U got skills in drawing. Lol
Thanks a ton :) God Bless You
Isn't the main symptom of LVF chest pain due to increased oxygen demand of the LV due to hypertrophy?
I think earlier in the process you'll have dyspnea from the pulmonary congestion and overall fatigue. The oxygen demand won't cause infarction/ pain that quickly.
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Is cyanosis specific to left sided heart failure?
I know it is caused by pump failure. Our debate is that I say as the pump fails, the EF decreases the blood pressure drops and the RAAS activates? Then the lack of pressure is detected by baroreceptors and epithelial cells in the kidney? Or does the RAAS become active due to increased heart rate?
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thanks, buddy
Very good job.
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So the RAAS is activated- that would lead to an increase in cardiac output! so how can we say the cardiac output will be decreased. Is there a published reference for decrease in cardiac output
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I would like you to clarify the onset of HF for me please. Is it in response to decreased pressure in the afferent tubule and baroreceptors, or is it in response to tachycardia. I have an ongoing dispute with a colleague. I say decreased pressure.
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really good video!
great video! thanks
You’re welcome. Glad you found it helpful!
can anybody tell me what software is that!!!! (drawing one)
Thanks for your inquiry. The whiteboard lessons are created using a Wacom Bamboo tablet. Thanks for watching!
Andrew d Great.. Cheers!!!
Keep watching, we appreciate the support. Cheers!!
Thank You! :)
Why does decreased urine output develop with systolic heart failure?
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Can someone have a regular blood pressure with signs and symptoms of RF and LF? The patient is also obese with a history of CAD and CABG done. This is not a real patient.
If someone had a CHF exasberation episode, what part of the pathophysiology would cause them to sweat perfusely and feel very warm all over?
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thank you so much :) keep doing this :)
increased edema right?
you rock
Thank you so much!
end up pause end up pause end up pause..doc spead up
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