I have gone to give blood 2x in the past 2 weeks. My iron was 11.7 the first time and 11.9 the last time. I started taking copper, 2 mg. , and Iron, 18 mg. per day. I also started eating more red meat and liver. How long does it take Iron to increase in the blood? Is this a sign that I MAY have a problem with Hepcidin?
why the hell is shiiit iron added to almost EVERY processed food !!! try this , take some crackers or cereal and smash it with water and stir it with a magnet and see the huge amounts of filings you get. WHAT THE HELL ??? why is that added ????
It all started after worl war 1 Somebody thought every pregnant woman is iron defecient because of the hemodillution that naturaly serves mom and baby, So they thought why not give iron to every body so that we resolve the iron defeciency epeidemic So started adding iron to food And the real problem is that iron is disregulated because of copper and magnesium defeciency Anemia and hepatic iron accumulation are well-known consequences of nutritional copper deficiency in humans and laboratory animals (1-3). The perturbations in iron metabolism arise because metabolic pathways involving the 2 metals are linked at least in part through the copper-containing proteins ceruloplasmin (Cp)6 and hephaestin (3). These proteins appear to aid in the release of iron into the plasma by working in concert with ferroportin (Fpn), the cell surface iron export protein. Fpn is expressed predominantly in reticuloendothelial macrophages of the liver and spleen and duodenal enterocytes (4,5). After Fpn exports iron, the Fe2+ must be oxidized to Fe3+ before it can bind to its transport protein transferrin. It is widely thought that circulating Cp, a ferroxidase, serves this function. A reduction in the activity of Cp, which carries 70-95% of the copper in the plasma, is one of the earliest manifestations of copper deficiency.
a lecture you must listen to.
Excellent!
THANK YOU VERY MUCH FOR A LECTURE BY THE AUTHORITY ON THE SUBJECT. DR T GANZ HAS DONE THE ORIGINAL RESEARCH ON THESE TOPICS.
Excellent lecture .
I have gone to give blood 2x in the past 2 weeks. My iron was 11.7 the first time and 11.9 the last time. I started taking copper, 2 mg. , and Iron, 18 mg. per day. I also started eating more red meat and liver. How long does it take Iron to increase in the blood? Is this a sign that I MAY have a problem with Hepcidin?
Had you used a proper microphone then this might have been of use.
why the hell is shiiit iron added to almost EVERY processed food !!! try this , take some crackers or cereal and smash it with water and stir it with a magnet and see the huge amounts of filings you get. WHAT THE HELL ??? why is that added ????
It all started after worl war 1
Somebody thought every pregnant woman is iron defecient because of the hemodillution that naturaly serves mom and baby,
So they thought why not give iron to every body so that we resolve the iron defeciency epeidemic
So started adding iron to food
And the real problem is that iron is disregulated because of copper and magnesium defeciency
Anemia and hepatic iron accumulation are well-known consequences of nutritional copper deficiency in humans and laboratory animals (1-3). The perturbations in iron metabolism arise because metabolic pathways involving the 2 metals are linked at least in part through the copper-containing proteins ceruloplasmin (Cp)6 and hephaestin (3). These proteins appear to aid in the release of iron into the plasma by working in concert with ferroportin (Fpn), the cell surface iron export protein. Fpn is expressed predominantly in reticuloendothelial macrophages of the liver and spleen and duodenal enterocytes (4,5). After Fpn exports iron, the Fe2+ must be oxidized to Fe3+ before it can bind to its transport protein transferrin. It is widely thought that circulating Cp, a ferroxidase, serves this function. A reduction in the activity of Cp, which carries 70-95% of the copper in the plasma, is one of the earliest manifestations of copper deficiency.