Clear to you perhaps, but only because you’ve studied the subject before. For a beginner who has only seen the prior videos in the playlist, this humoral immunity video after 2:22 is incredibly difficult to follow.
Such a useful and amazing channel!! Everytime I’m overwhelmed regarding my studies, this channel lifts me up and makes me more interested to learn than ❤❤
Very clear explanation, easy to understand. I'd like to suggest ( ask) that you make sometime a video to explain what is known abour how the excess of iron in erythrocytes due to hemochromatosis, damages macrophages that are recycling erythrocytes and may cause arthritis. Your work is great, thanks!
Brilliant! Thank you for these videos. I imagine many people like myself have become interested in the immune system because of the present Coronavirus pandemic. These presentations are about as clear as you can get!
How much manner a B cell or T cell can be activated ? and what is the purpose of each different immunoglobulin ? and how in reality the immune response are regulated by Treg cell ? and for finish ... Why we need CD8 cell that recognize only a specific MHC-1 corrupted by a specific virus if we can directly recognize that the MHC-1 is corrupted ? and after that we kill the cell
Umoral imunity (Anticorps) can eliminate bacteria whitout involving celular imunity? If no, what happend when you have antiicorps and celular imunity system is low? Thanks.
Didn’t understand how class switching and infinity maturation took place until after seeing this super simplified video. It doesn’t get to the nitty gritty stuff but it still explains the pathophysiological process better than any 2 hour video that breaks down the cytokines and cellular interactions like Najeeb, and FA General Principles. One thing I’m still confused about is I realized a lot of materials get carried away with trying to explain that naive B cell activation could be as a result of direct interaction with naive T-helper or from IL-4 and IL-5 from another APC activated T-helper2 and that either B7-CD28 or CD40-CD40L interaction can be the co-stimulatory signal for T-helper2 activation as a result correct? Example so any APC perhaps dendritic cells can use B7-CD28 interaction as a costimulatory signal for T cell activation from which it releases IL-4 and IL-5 for B cell activation OR B cells can use CD40-CD40L interaction as a costimulatory signal for when it acts as a APC and binds directly to the naive T-helper2 causing the activation of the cell and the subsequent release of IL-4 and IL-5 for the activation of the APC B cell. And macrophages also contain both B7 and CD40 however the CD40-CD40L interaction only acts to stimulate any type of naive T cells? Yea the confusion is deep with this one or I’m just uncertain as to what I’ve read and I have yet to do any q-banks…
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If i understand anything related to humoral immunity, it's because of this video
Spectacular way of explaining, highly recommend ❤❤
What an excellent diction, not too fast, so clear, just perfect! Graphics are great, too!
Clear to you perhaps, but only because you’ve studied the subject before. For a beginner who has only seen the prior videos in the playlist, this humoral immunity video after 2:22 is incredibly difficult to follow.
👌👌🙌👏👏 I would suggest students to watch this video after 1 reading of humoral immunity.
Hey
Such a useful and amazing channel!! Everytime I’m overwhelmed regarding my studies, this channel lifts me up and makes me more interested to learn than ❤❤
مستغرب انو مشاهدات القناة هذي مرا منخفضة رغم الشرح الرهيب الي يقدموه
Ummmm what?
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Excellent video
it clear to understand and perfectly explained..thanks and keep it up.
Excellent video with good explanation.
very helpful plz keep up. easiest way to refresh
I love this Video ❤❤
Good stuff 👍🏽
Your Videos are extremely help ful ..
Your explanation is very nice 😜😜😜
Very clear explanation, easy to understand. I'd like to suggest ( ask) that you make sometime a video to explain what is known abour how the excess of iron in erythrocytes due to hemochromatosis, damages macrophages that are recycling erythrocytes and may cause arthritis. Your work is great, thanks!
Excellent 😭
Thanks 😅
Well demo thank you
Brilliant! Thank you for these videos. I imagine many people like myself have become interested in the immune system because of the present Coronavirus pandemic. These presentations are about as clear as you can get!
So interesting 😅😊
My body does this! Hell yeah
Video on the immune system producing antibodies that attack other cells such as platelets (ITP) would be interesting.
Thank you
Ngl I'm very confused but the video is great
How much manner a B cell or T cell can be activated ?
and what is the purpose of each different immunoglobulin ? and how in reality the immune response are regulated by Treg cell ?
and for finish ...
Why we need CD8 cell that recognize only a specific MHC-1 corrupted by a specific virus if we can directly recognize that the MHC-1 is corrupted ? and after that we kill the cell
perfect
Thank you so much
Umoral imunity (Anticorps) can eliminate bacteria whitout involving celular imunity? If no, what happend when you have antiicorps and celular imunity system is low? Thanks.
This means that the pathogen can produce antibodies.
Plz provide in hindi too.
😀😆
Didn’t understand how class switching and infinity maturation took place until after seeing this super simplified video. It doesn’t get to the nitty gritty stuff but it still explains the pathophysiological process better than any 2 hour video that breaks down the cytokines and cellular interactions like Najeeb, and FA General Principles. One thing I’m still confused about is I realized a lot of materials get carried away with trying to explain that naive B cell activation could be as a result of direct interaction with naive T-helper or from IL-4 and IL-5 from another APC activated T-helper2 and that either B7-CD28 or CD40-CD40L interaction can be the co-stimulatory signal for T-helper2 activation as a result correct?
Example so any APC perhaps dendritic cells can use B7-CD28 interaction as a costimulatory signal for T cell activation from which it releases IL-4 and IL-5 for B cell activation OR B cells can use CD40-CD40L interaction as a costimulatory signal for when it acts as a APC and binds directly to the naive T-helper2 causing the activation of the cell and the subsequent release of IL-4 and IL-5 for the activation of the APC B cell. And macrophages also contain both B7 and CD40 however the CD40-CD40L interaction only acts to stimulate any type of naive T cells?
Yea the confusion is deep with this one or I’m just uncertain as to what I’ve read and I have yet to do any q-banks…