RUclips commenting system clearly has some bugs. Here is the original comment which led to this video (I had to copy and paste it here - the original poster did it but youtube "held it for review" and even though I approved it, it seems have disappeared into cyberspace.) Here is the full text: @petertownsend252 A viewer suggested topic I hope makes it into one of your upcoming videos. Can you do a video on lipoprotein (a), aka Lp(a)? More specifically, the highly correlative (and probable causal) relationship between the level of Lp(a) and arteriosclerosis cardiovascular disease (ASCVD) risk. Enormous implications relating to use of statins and also the Lean Mass Hyper-Responder (LMHR) phenotype and associated ongoing LMHR study being sponsored by Dave Feldman, Nick Norwitz, and others through the Citizen Science Foundation that is funding the study. For reasons unexplained, the LMHR study design does not consider the role Lp(a) plays in lowering the incidence of plaque and associated ASCVD. In the first instance, the level of Lp(a) is genetically determined. As yet, there is no pill to lower the level of Lp(a). Of course, the Pharma industry is working on one. In the meantime, the CDC states, "Lp(a) levels cannot be controlled by healthy eating and exercising". The CDC and others leverage the genetic (as yet untreatable) nature of Lp(a), as a pretext to double and triple down on the use of statins to lower LDL and purportedly lower the overall risk of ASCVD until a new magic Lp(a) lowering pill arrives on the scene to save the day. The CDC and others choose to willfully ignore published research showing that the level of Lp(a) can, in fact, be affected by diet. More specifically, "A low carbohydrate/high saturated fat diet was shown to result in a 15% decrease in lipoprotein(a) levels, whereas a decrease in saturated fat was associated with a 10-15% increase in lipoprotein(a) levels." [Reference below] The obvious problem here (and source of the willful ignorance) is that the known dietary influences on Lp(a) run counter to the doctrinal dietary talking points of the CDC and USDA and counter to the business interests of Big Food, Big Pharma, and the entirety of the healthcare delivery industry. Another important Lp(a) genetic factor is the Lp(a) null allele found at marker rs41272114 on chromosome 6. Approximately 3% of the general population carries the Lp(a) null allele. The Lp(a) null allele results in a non-detectable level of Lp(a) and is clinically proven and well known to be associated with a low ASCVD risk, regardless of the level of LDL. In my case, I carry the Lp(a) null allele and have a non-detectable level (
Nice video this morning, Russ. I enjoyed it. I have had my Lp-a tested. But I did not have a before and after test. While successfully reversing my CAC by 30%. I am at 20mg/dl, and it's my understanding that the body uses Lp-a in what it considers emergency situations to stop bleeding. So some is useful. My thoughts on LMHR with clear arteries and no cardiac events even with high Lp-a is that these people do not suffer from focal vitamin C deficiency! Not having focal vitamin C deficiency means they have what I call an endothelial shield 🛡 With this in mind, it doesn't matter where their cholesterol is or even what their Lp-a is. As is in my case, my endothelial shield 🛡 is being rebuilt as indicated by my last CAC. I even have a 39 HDL. I have no concerns where my Lp-a or LDL is. I just watched a Berberine video I was amazes at how Berberine was shown to shrink plaque by more than 3% in just 4 months. It also lowered triglycerides as well as LDL. It is excellent at helping to maintain heart sinus rhythm as well. I discontinued my 25mg dose of Metropolol in favor of 750mg dose of Berberine 2x per day because the Metropolol was causing some weight gain from lower metabolism, including some sleep disturbances. Now, my deep sleep has nearly doubled. So I'm doing very well. Thanks again!
Super - those are interesting results, especially about shrinking plaque. I was actually going to get a CIMT to see how my plaque was (if any) but unfortunately, that doctor had to stop taking Medicare patients, so I'm back to square one on that. My primary doctor doesn't seem to be clear on whether what they offer is an actual CIMT or not. So I just have to push on and keep trying I guess.
@@SET12DSP I think youtube doesn't allow links in comments unless by the creator. It may be a setting somewhere. I'll look into that. Try the comment with just describing how to get the the video.
Thanks Russ, another great video; very informative. I had my LP(a) tested about 18 months ago. After receiving the results, I got kind of nervous about the level (58.6 nmol/L) until I figured out the difference in testing units. 58.6 nmol seems pretty decent while 58.6 mg would be concerning. Over the last two years I’ve been about 95% strict carnivore (I.e. lots of saturated fat, almost zero carbs.) The test is only $30, I think I’ll have it done again to see if there’s been any change. Thanks again for your time and effort on these videos, I really look forward to your releases on Friday.
Thanks for your support and interest. Yeah, while my numbers were low, like I said, I was confused. Figured maybe the tests had too much variation, especially at lower numbers, but I'm pretty sure now it was just the units. So that 14 nmol/L I got was actually probably slightly better than the 7 mg/dL.
It actually doesn't seem to be on the radar for most family doctors. I had to ask for that first test and it never really got interpreted for me. Even when I was going to a doctor (the one who ordered the 2023 test) I couldn't really get a good answer about low Lp(a) being protective. He kept on saying stuff like "you don't have to worry about it unless it gets a lot higher" but didn't seem to want to actually say "you're at lower risk because it's so low."
I'm a lmhr...Good vid...And I bet you are right as to what they have planned next for us..Also,I gather you have seen orieo cookies vs statins. Dr. Berry interviewed the researcher,downright entertaining.
In May last year I went for a health check up at my local doctor's surgery, I am a 68 yo white male, the blood test revealed a 21% chance of me having a heart attack in the next 10 years due to a raised cholesterol level. The doctor immediately advised that I start taking statins, in fact he was very keen on this, no discussion about using diet or lifestyle. Anyway I looked at the side effects of statins and decided not to take them, rather I would change my diet and get more exercise. 10 months later that percentage has dropped to 13.6%, I have lost 10 kilos in weight, I no longer get indigestion every night or occasional acid reflex, and my blood pressure has returned to normal levels, yet the doctor still wanted me to take statins! I said I would prefer to continue with my diet and exercise. What's going on? Why the pressure to take statins? By the way, in terms of diet I cut out sugar, white bread, cakes, biscuits, pizza, pie, chips, chocolate, pasties etc. It was hard for a couple of weeks but then my body adapted and now I'm fine.
My first question is: "How was your risk calculated?" There are several calculators and the AHA/ACC still uses the 2013 Pooled Cohort Equation calculator even though it has been shown repeatedly that the 2018 calculator is more accurate. Try this one to be sure you're getting the best info: globalrph.com/medcalcs/pooled-cohort-2018-revised-10-year-risk/ I found that the 2013 calculator was nearly twice as high as the 2018 calculator when estimating risk. Check out my risk calculator videos by visiting my channel page. There's a couple of them in there. The pressure to take statins seems to come from doctors applying population statistics to individuals, or blindly following standards of care (they could get in trouble if they don't I guess).
@@54000biker I don't have a lot of faith in the calculators, given that they ignore Lp(a) and Trigs (and by extension, Trigs/HDL ratio) but at least the 2018 PCE calculator can be used (if it has good results) to keep a doctor from hounding you.
I’m 72 and 3 years ago did all the things you’ve done and lost 60 pounds (27kg) then experienced the same unexpected side effects, heartburn gone, blood pressure normal yet am told to take a statin, no discussion at all, just … you must take it. I have not and am becoming more and more resistant to statin therapy.
RUclips commenting system clearly has some bugs. Here is the original comment which led to this video (I had to copy and paste it here - the original poster did it but youtube "held it for review" and even though I approved it, it seems have disappeared into cyberspace.) Here is the full text:
@petertownsend252
A viewer suggested topic I hope makes it into one of your upcoming videos. Can you do a video on lipoprotein (a), aka Lp(a)? More specifically, the highly correlative (and probable causal) relationship between the level of Lp(a) and arteriosclerosis cardiovascular disease (ASCVD) risk.
Enormous implications relating to use of statins and also the Lean Mass Hyper-Responder (LMHR) phenotype and associated ongoing LMHR study being sponsored by Dave Feldman, Nick Norwitz, and others through the Citizen Science Foundation that is funding the study. For reasons unexplained, the LMHR study design does not consider the role Lp(a) plays in lowering the incidence of plaque and associated ASCVD.
In the first instance, the level of Lp(a) is genetically determined. As yet, there is no pill to lower the level of Lp(a). Of course, the Pharma industry is working on one. In the meantime, the CDC states, "Lp(a) levels cannot be controlled by healthy eating and exercising". The CDC and others leverage the genetic (as yet untreatable) nature of Lp(a), as a pretext to double and triple down on the use of statins to lower LDL and purportedly lower the overall risk of ASCVD until a new magic Lp(a) lowering pill arrives on the scene to save the day.
The CDC and others choose to willfully ignore published research showing that the level of Lp(a) can, in fact, be affected by diet. More specifically, "A low carbohydrate/high saturated fat diet was shown to result in a 15% decrease in lipoprotein(a) levels, whereas a decrease in saturated fat was associated with a 10-15% increase in lipoprotein(a) levels." [Reference below]
The obvious problem here (and source of the willful ignorance) is that the known dietary influences on Lp(a) run counter to the doctrinal dietary talking points of the CDC and USDA and counter to the business interests of Big Food, Big Pharma, and the entirety of the healthcare delivery industry.
Another important Lp(a) genetic factor is the Lp(a) null allele found at marker rs41272114 on chromosome 6. Approximately 3% of the general population carries the Lp(a) null allele. The Lp(a) null allele results in a non-detectable level of Lp(a) and is clinically proven and well known to be associated with a low ASCVD risk, regardless of the level of LDL.
In my case, I carry the Lp(a) null allele and have a non-detectable level (
Appreciate all your information and pursuit of new diagnostic data! 👍😃
Nice video this morning, Russ. I enjoyed it.
I have had my Lp-a tested. But I did not have a before and after test. While successfully reversing my CAC by 30%.
I am at 20mg/dl, and it's my understanding that the body uses Lp-a in what it considers emergency situations to stop bleeding. So some is useful.
My thoughts on LMHR with clear arteries and no cardiac events even with high Lp-a is that these people do not suffer from focal vitamin C deficiency!
Not having focal vitamin C deficiency means they have what I call an endothelial shield 🛡
With this in mind, it doesn't matter where their cholesterol is or even what their Lp-a is.
As is in my case, my endothelial shield 🛡 is being rebuilt as indicated by my last CAC. I even have a 39 HDL. I have no concerns where my Lp-a or LDL is.
I just watched a Berberine video I was amazes at how Berberine was shown to shrink plaque by more than 3% in just 4 months. It also lowered triglycerides as well as LDL. It is excellent at helping to maintain heart sinus rhythm as well.
I discontinued my 25mg dose of Metropolol in favor of 750mg dose of Berberine 2x per day because the Metropolol was causing some weight gain from lower metabolism, including some sleep disturbances. Now, my deep sleep has nearly doubled.
So I'm doing very well.
Thanks again!
Super - those are interesting results, especially about shrinking plaque. I was actually going to get a CIMT to see how my plaque was (if any) but unfortunately, that doctor had to stop taking Medicare patients, so I'm back to square one on that. My primary doctor doesn't seem to be clear on whether what they offer is an actual CIMT or not. So I just have to push on and keep trying I guess.
@mystatinfreelife Don't understand it, Russ. Won't let me post the Berberine u-tube link, and it's their own video 25 min. by Dr. Been!
@@SET12DSP I think youtube doesn't allow links in comments unless by the creator. It may be a setting somewhere. I'll look into that. Try the comment with just describing how to get the the video.
@@mystatinfreelife Berberine Treats Atherosclerosis in humans ( small study) is the title.
Thanks!
Thanks Russ, another great video; very informative. I had my LP(a) tested about 18 months ago. After receiving the results, I got kind of nervous about the level (58.6 nmol/L) until I figured out the difference in testing units. 58.6 nmol seems pretty decent while 58.6 mg would be concerning.
Over the last two years I’ve been about 95% strict carnivore (I.e. lots of saturated fat, almost zero carbs.) The test is only $30, I think I’ll have it done again to see if there’s been any change.
Thanks again for your time and effort on these videos, I really look forward to your releases on Friday.
Thanks for your support and interest. Yeah, while my numbers were low, like I said, I was confused. Figured maybe the tests had too much variation, especially at lower numbers, but I'm pretty sure now it was just the units. So that 14 nmol/L I got was actually probably slightly better than the 7 mg/dL.
Thank you!
Great video Russ, I never had this tested.
It actually doesn't seem to be on the radar for most family doctors. I had to ask for that first test and it never really got interpreted for me. Even when I was going to a doctor (the one who ordered the 2023 test) I couldn't really get a good answer about low Lp(a) being protective. He kept on saying stuff like "you don't have to worry about it unless it gets a lot higher" but didn't seem to want to actually say "you're at lower risk because it's so low."
@mystatinfreelife Same here, no explanation. Well, you know time is money!
I was my GP's first for an Lp-a test.
@@SET12DSP Yeah, you probably didn't have the time to explain it to your doctor. 🤣
I'm a lmhr...Good vid...And I bet you are right as to what they have planned next for us..Also,I gather you have seen orieo cookies vs statins. Dr. Berry interviewed the researcher,downright entertaining.
I've seen a few videos by Nick. I'll look for the Dr. Berry interview. Thanks.
In May last year I went for a health check up at my local doctor's surgery, I am a 68 yo white male, the blood test revealed a 21% chance of me having a heart attack in the next 10 years due to a raised cholesterol level. The doctor immediately advised that I start taking statins, in fact he was very keen on this, no discussion about using diet or lifestyle. Anyway I looked at the side effects of statins and decided not to take them, rather I would change my diet and get more exercise. 10 months later that percentage has dropped to 13.6%, I have lost 10 kilos in weight, I no longer get indigestion every night or occasional acid reflex, and my blood pressure has returned to normal levels, yet the doctor still wanted me to take statins! I said I would prefer to continue with my diet and exercise.
What's going on? Why the pressure to take statins?
By the way, in terms of diet I cut out sugar, white bread, cakes, biscuits, pizza, pie, chips, chocolate, pasties etc. It was hard for a couple of weeks but then my body adapted and now I'm fine.
My first question is: "How was your risk calculated?" There are several calculators and the AHA/ACC still uses the 2013 Pooled Cohort Equation calculator even though it has been shown repeatedly that the 2018 calculator is more accurate. Try this one to be sure you're getting the best info: globalrph.com/medcalcs/pooled-cohort-2018-revised-10-year-risk/
I found that the 2013 calculator was nearly twice as high as the 2018 calculator when estimating risk. Check out my risk calculator videos by visiting my channel page. There's a couple of them in there.
The pressure to take statins seems to come from doctors applying population statistics to individuals, or blindly following standards of care (they could get in trouble if they don't I guess).
@@mystatinfreelife I have no idea how it was calculated, it is a test run by the NHS and relates to nothing I can find online.
@@54000biker I don't have a lot of faith in the calculators, given that they ignore Lp(a) and Trigs (and by extension, Trigs/HDL ratio) but at least the 2018 PCE calculator can be used (if it has good results) to keep a doctor from hounding you.
I’m 72 and 3 years ago did all the things you’ve done and lost 60 pounds (27kg) then experienced the same unexpected side effects, heartburn gone, blood pressure normal yet am told to take a statin, no discussion at all, just … you must take it. I have not and am becoming more and more resistant to statin therapy.