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Why Sparsantan improve proteinurea but not eGFR in FSGS??
If maladaptive way is important why not develop in Donors for kidney to develop FSGS post donation
Rutuximab in treatment
MMF roll in FSGS
At which point you can replace IVIG with low IgG with Rituximab treatment or based of infection risk not only IgG levels
Limits of IFTA and eGFR to use or not CNIs
APOL1 in FSGS
Why Sparsantan improve proteinurea but not eGFR in FSGS??
If maladaptive way is important why not develop in Donors for kidney to develop FSGS post donation
Rutuximab in treatment
MMF roll in FSGS
At which point you can replace IVIG with low IgG with Rituximab treatment or based of infection risk not only IgG levels
Limits of IFTA and eGFR to use or not CNIs
APOL1 in FSGS