🎓‼ Earn CE Credits for this lesson at ICU Advantage Academy: 👉🏼 adv.icu/academy 🤩🆕 NEW Beautifully Designed Hemodynamics Notes 👉🏼 adv.icu/notes-hemodynamics 📝 Older Style NOTES available to members! ► RUclips: adv.icu/ym | ► Patreon: adv.icu/pm 💲 10% off Critical Care Academy (CCRN Review): 👉🏼adv.icu/cca (USE CODE "icuadv10")
U r simply terrific. No student will listen to u and still feels empty as most of us feel after listening to our instructors. God bless u pls and keep up the good work 🙏👍👌👏👏
Awesome lesson! Exactly what I needed for my complex adult health class!! The light is at the end of the tunnel! Thank you for helping me better understand!
Love the way how you have organized this channel with various playlists. It is hard to find such useful topics in such a structured manner anywhere else. Many thanks for making these. I think people get so immersed in listening to the lectures that they forget to tap the like buttons ether: Looking at the Views Vs Likes ratio!
Thank you so much Rahul. I have done my best to make it as easy as possible for people to find and get the information they are looking for so its great to hear this feedback. And yes, I agree, more likes have to be out there! :)
I was thinking when the stomach is perfused then digestion and stomach emptying takes place. So when blood flow is cut off to the stomach then there will be no digestion process taking place hence gastric residuals will be there. That would be evident from NG drainage. Hope that helped.
Thank you so much...video was very helpful and i will practice looking at patint, listening to them and feeling them along with looking at the numbers and monitors🫂 YOU ROCK🙌🏾✊🏾✊🏾👩🏾💻
True LV preload would be LVEDP, which is influenced by volume. In a lot of cases, in most patients, especially without SWAN/PA, we often refer to CVP as our surrogate for preload. Its essentially the volume and pressure to stretch and "preload" the heart prior to its contraction.
Amazing lecture as always!! Your content is really helping me in my final semester of nursing school. I just wanted to point out a potential correction in the renal signs. You stated elevated BUN and creatinine, as well as elevated K+. I believe potassium would be lowered due to RAAS activation of aldosterone and K+ excretion. Is this correct or am I mistaken?
Thank you very much for the comment and I'm really glad I have been helpful for you in nursing school! Almost done!!! To your point, in healthy, well perfused kidneys, you are correct that we would see the excretion of K+. In our case here, we are under perfused, leading to failure of various organ systems. In the case of the kidneys, we would be exhibiting AKI/ARF, which would mean they are not working optimally. This is what leads to the increase in Cr, BUN, and potassium levels. When entering renal failure, a big indicator on the urgency of starting dialysis is the potassium and other electrolyte levels. If your levels are rising, then you need to get some form of dialysis going sooner than later.
These are all secondary noninvasive measures of Haemodynamic function, not true non invasive direct monitoring - NICOM devices and the like are direct measures with a lot better reliability
🎓‼ Earn CE Credits for this lesson at ICU Advantage Academy: 👉🏼 adv.icu/academy
🤩🆕 NEW Beautifully Designed Hemodynamics Notes 👉🏼 adv.icu/notes-hemodynamics
📝 Older Style NOTES available to members! ► RUclips: adv.icu/ym | ► Patreon: adv.icu/pm
💲 10% off Critical Care Academy (CCRN Review): 👉🏼adv.icu/cca (USE CODE "icuadv10")
U r simply terrific. No student will listen to u and still feels empty as most of us feel after listening to our instructors. God bless u pls and keep up the good work 🙏👍👌👏👏
Thank you so much! Glad to hear you found it helpful!
thank you so much, it is a very good channel for new ICU nurses like me
Awesome lesson! Exactly what I needed for my complex adult health class!! The light is at the end of the tunnel! Thank you for helping me better understand!
You're almost there! You can do it! Thanks for this comment, and so glad you are finding these videos helpful.
Love the way how you have organized this channel with various playlists. It is hard to find such useful topics in such a structured manner anywhere else. Many thanks for making these.
I think people get so immersed in listening to the lectures that they forget to tap the like buttons ether: Looking at the Views Vs Likes ratio!
Thank you so much Rahul. I have done my best to make it as easy as possible for people to find and get the information they are looking for so its great to hear this feedback.
And yes, I agree, more likes have to be out there! :)
S
I love this, thank you!
Woohoo! Youre def welcome
Thank you so much for the amazing lecture
Hi these videos are so helpful! I have a question though, why would we see increased NG output with decreased circulation to gut? thanks in advance!
I was thinking when the stomach is perfused then digestion and stomach emptying takes place. So when blood flow is cut off to the stomach then there will be no digestion process taking place hence gastric residuals will be there. That would be evident from NG drainage.
Hope that helped.
Very good job man, thank you.
Thank you so much!
Superb , amazing lecture
I need the reference
I have to make presentation
If you could share please
Why exactly would you see an increase in NG output? If the GI tract was shutting down wouldn't you see decreased output? thanks!
I believe gastric secretions are largely unaffeted while gastric motility is significantly reduced.
Thank you so much...video was very helpful and i will practice looking at patint, listening to them and feeling them along with looking at the numbers and monitors🫂 YOU ROCK🙌🏾✊🏾✊🏾👩🏾💻
Please help me understand. Preload is left ventricular end diastole volune
True LV preload would be LVEDP, which is influenced by volume.
In a lot of cases, in most patients, especially without SWAN/PA, we often refer to CVP as our surrogate for preload. Its essentially the volume and pressure to stretch and "preload" the heart prior to its contraction.
Thanks, for the breakdown of Hemodynamic Monitoring! Your explanations really helped.
Glad you found them helpful!
Thanks !!!
I am very sorry to ask but can I know why patients with organ failure you will see ST elevation in them? Thank you!
One of the of the first organs that fail earlier in the kidney…this leads to Hyperkelemia which will in turn lead to ST elevation.
Amazing lecture as always!! Your content is really helping me in my final semester of nursing school. I just wanted to point out a potential correction in the renal signs. You stated elevated BUN and creatinine, as well as elevated K+. I believe potassium would be lowered due to RAAS activation of aldosterone and K+ excretion. Is this correct or am I mistaken?
Thank you very much for the comment and I'm really glad I have been helpful for you in nursing school! Almost done!!!
To your point, in healthy, well perfused kidneys, you are correct that we would see the excretion of K+. In our case here, we are under perfused, leading to failure of various organ systems. In the case of the kidneys, we would be exhibiting AKI/ARF, which would mean they are not working optimally. This is what leads to the increase in Cr, BUN, and potassium levels. When entering renal failure, a big indicator on the urgency of starting dialysis is the potassium and other electrolyte levels. If your levels are rising, then you need to get some form of dialysis going sooner than later.
@@ICUAdvantage That makes sense.... Thank you for the clarification!! I look forward to more videos!!
Perfect! And I'll keep em coming.
On my part 5. This is better than Barrons!!
Thanks so much for that! Glad you are finding it helpful
What is NG output?
NG (nasogastric) tube output.
These are all secondary noninvasive measures of Haemodynamic function, not true non invasive direct monitoring - NICOM devices and the like are direct measures with a lot better reliability