honestly i love u bro... ur voice is so easy to listen to it feels like im listening to a friend talking to me. thank u so much for these super helpful vids :)
Thank you! what youre doing is extremely helpful to individuals who are visual learners/slow readers, like myself. I have a long way to go and I know that I will need your help during this difficult journey. Your videos are invaluable! please keep them coming! God bless you!! :)
Very helpfyl introductory lecture, thank you. Regarding the basic pathways I would have enjoyed a few sentences on how the fat is transported from adipose to fat burning tissue.
Hi, I am APO E 2 heterozygous, 57 almost 58. I really understand this ., and will copy it down because WHEN I go to any doctor with this information and ask to be referred to a Cardiologist/re Lipid specialist I get treated like a moron and ignored. Fats affect me BIG TIME! And my entire metabolic structure is so different to my Husbands who is an APO E 3/3. I do not know my other gene as I was only tested for the 2 Factor. I hope its a 3. Whatever I EAT affects me drastically and so its no processed or refined foods.. I had all my amalgams removed but not by a biological dentist so have been left quite ill from the toxic effects of this and also 2 Root canals left which as we know are a dead gangrenous bone inside a living organism and that doesnt work! I do need to get these 2 out as its affecting my health badly now. I would say that there is Familial Dsylipidemia but what class I have no idea. Mother had heart problems for all her life., died Heart failure at 70, diabetic late onset. Father massive coronary at age 67. Brother suicided but they did autopsy and he had atherosclerosis starting of at age 35. Dads brother died massive coronary. Mums dad died massive coronary. Mums half sister died burst Aorta. Mums brother has had a burst Aorta, still alive. Quite a history of heart in family. So what can I do any help be appreciated. Thank you :) email is desireeaquarian@yahoo.com.au
Okay, i am very confused about type 1 Hyperlipidemia now, what you said about it seems logical , however Fredrickson classification of hyperlipidemias states that only Chylomicrons are elevated in type 1 and that Serum has creamy top layer without being turbid. If VLDL are incresed serum should be turbid and if they aren't incresed how come they aren't, it's illogical. This is literally killing me.
Cheese I like, but refined flour and macaroni ... puts you into the "Idiot" category ... and statins are death ... complete waste of time here ... look at Armando Hasudungan's - Physiology of Lipoproteins Cholesterol 70 Going On 100
The world could use more non PhD teachers. Great Video.
"I covered the high yield stuff" - and I'm sooo grateful for that. Fantastic and very encouraging! You are awesome.
Trust me, 1 whole year, and today's first day I understand and probably even memorize it! Thank God for you! Thank you :)
honestly i love u bro... ur voice is so easy to listen to it feels like im listening to a friend talking to me. thank u so much for these super helpful vids :)
Thank you! what youre doing is extremely helpful to individuals who are visual learners/slow readers, like myself. I have a long way to go and I know that I will need your help during this difficult journey. Your videos are invaluable! please keep them coming! God bless you!! :)
Yay!!! This video made Apolipoproteins/ Familial Dyslipidemias sooo clear!! My love for Biochemistry has been restored!!! Lol!!! Thanks!!
Thank you! Helping me grasp this in time for Biochem final in 1st year dental school. Very appreciative!
Very helpfyl introductory lecture, thank you. Regarding the basic pathways I would have enjoyed a few sentences on how the fat is transported from adipose to fat burning tissue.
you are awesome....... thank you so much for the lecture you made it sound so easy......
Absolutely wonderfully explained. Now I clearly know what pizza's do inside me.
Thank you so much a medical student from Belgium
Absolutely great Video !!!!!
Thanks for such kind of lecture......great teaching of such tough topic......now it is very easy to make out.....
SO helpful! Studying for Step 1 and this was great. Thank you!
you're a life savior thank youuuuu
Man you are awesome, luckily my classmate recommended me this channel
you are seriously the best!
Hi, I am APO E 2 heterozygous, 57 almost 58. I really understand this ., and will copy it down because WHEN I go to any doctor with this information and ask to be referred to a Cardiologist/re Lipid specialist I get treated like a moron and ignored. Fats affect me BIG TIME! And my entire metabolic structure is so different to my Husbands who is an APO E 3/3. I do not know my other gene as I was only tested for the 2 Factor. I hope its a 3. Whatever I EAT affects me drastically and so its no processed or refined foods.. I had all my amalgams removed but not by a biological dentist so have been left quite ill from the toxic effects of this and also 2 Root canals left which as we know are a dead gangrenous bone inside a living organism and that doesnt work! I do need to get these 2 out as its affecting my health badly now. I would say that there is Familial Dsylipidemia but what class I have no idea. Mother had heart problems for all her life., died Heart failure at 70, diabetic late onset. Father massive coronary at age 67. Brother suicided but they did autopsy and he had atherosclerosis starting of at age 35. Dads brother died massive coronary. Mums dad died massive coronary. Mums half sister died burst Aorta. Mums brother has had a burst Aorta, still alive. Quite a history of heart in family. So what can I do any help be appreciated. Thank you :) email is desireeaquarian@yahoo.com.au
Great lecture, thanks!
You Are Awesome.. Couldn't be more easier!
Thank you soo much .you made it soo simple....❤💞
Couldn’t resist, had to subscribe. Amazing work 👍🏻
Amazing video!! Thank you soo much
Great lecture. Just to clarify, does VLDL have apolipoprotein A or no? At 21:16 VLDL is not listed under A, but A is listed with VLDL.
Absolutely fantastic 💯
i really enjoyed the lecture .. thanks a lot
and thank you for saying that we're awesome many times lol
THANK YOU!!
you are fantastic and so helpful!
is like you are explaining the First Aid to me, you should be part of Doctors in Training !
thanks to you so much,beautiful explanation
Thanks for the video.. really amazing.
Thanks for the great effort! Keep up the good work!
I stayed with you for 3 hours now. I love your voice. Why didn't you pursue med? Weee, you're awesome too!!
i didnt understand type 4 n 5 -> y increased chylomicrons with increased VLDL....does VLDL inhibits LPL activity?
Have same doubt.
You made it understandable ! Thank you
Thank you. If you were profesor you would be my favourite.
YOU ARE AWESOME!
Thanq sir you cleared our doubt!!!😊
thank you so much !!!
thank you so much..that was great!!!
Thank you very much!
You are amazing! Tks a lot!
you are amazing thank you very much !!
Thankyou so much :) Stay blessed
thank u
thank you so much for sharing!
You are great
Great lecture!
Excellent video! Thank you (:
You are a lifesaver , thanks :)
SUPERB PRESENTATIONS THANKS A LOT
brilliant video!!!
omg i love this video! made me understand it so clearly muah
Thank you for this!
You are awesome! Thanks so much:):)
this was amazing thank you !
Okay, i am very confused about type 1 Hyperlipidemia now, what you said about it seems logical , however Fredrickson classification of hyperlipidemias states that only Chylomicrons are elevated in type 1 and that Serum has creamy top layer without being turbid. If VLDL are incresed serum should be turbid and if they aren't incresed how come they aren't, it's illogical. This is literally killing me.
Thank you so much
thanks for nice explanation
Thank u very much sir...
Lovely!
great job.
Thanks! It really helped a lot. :)
thanks man! really useful!
thank you so much sir
I wish I had found this back when I first started studying for boards. -___-
Thank you!!!!
so clear.......
Thank you really helpful
I don't have any questions. Wow.
thank you!
Thanks for a great effort. I have one question though. Among the 5 defects that you have described, where does the increased TG in blood fall into?
+Sandeep Paudel Familial Hyperlipidemia (Type 4)
amazing
non-phd's are the Best teachers. I'm currently being taught by nothing but "experts" in their fields, but that doesn't make them good educators.
great video :D
Why do we need to convert cholesterol to cholesterol esters?
Because the cholesterol esters are able to pack together more tightly, hence more cholesterol in less space
Why no more videos
VLDL doesn't have apoA
Please tell me in bengali or hindi
Cheese I like, but refined flour and macaroni ... puts you into the "Idiot" category ... and statins are death ... complete waste of time here ... look at Armando Hasudungan's - Physiology of Lipoproteins Cholesterol
70 Going On 100
Carroll Hoagland ruclips.net/video/PkKH8lTxvzA/видео.html Good call!
It makes sense in first aid now lol
I love you... lol
great lecture!