Thank you - I lost my husband recently, and they suspected a shunt. This is the best explanation of how it works. It also explains why continuing to raise the Oxygen levels did not improve his saturation levels. In the end, they had him on 12 liters. They never did find the shunt, and in his case, it was NOT in the heart. They did find cancer and suspected that was the cause of the shunt.
Great video with really clear explanations of shunting and describing why they don't respond effectively to O2 therapy! The videos would be even better without the incredible amount of advertisements populating half of the page throughout the video. I counted 5 different advertisements in 10 minutes. Please take it easy on the ads! Your content speaks well enough for itself!
could be dependent on patient age. PDA is common in children, especially newborns and premies. left untreated, possible severe complications in adults.
Shunting is rom partial or complete collapse of alveoli- which occurs because of one of two things (or both) 1- pulmonary edema or fluid in alveoli which prevents gas exchange 2- lack of pulmonary surfactant which reduces lung compliance, thus alveoli are floppy
This is a great video, thank you! Could pressure during a free dive cause a pulmonary shunt? And how is a pulmonary shunt different from or related to alveolar collapse?
Is a "pulmonary shunt" the same thing as an "intrapulmonary shunt"? Is is possible to have a "right to left intrapulmonary shunt"? I keep reading this phrase "right to left intrapulmonary shunt" and I don't know what it is talking about. Does it really mean venous to arterial? Because "right to left" makes no sense in the intrapulmonary context.
But i can’t understand something isn’t the blood with 85% saturation working its way through all tissues will reach the right side of the heart with a saturation which is less that 70% let’s say 60 % and ...it’s like the saturation will go down after every cycle i don’t understand Thank you in advance
Great lecture. Why is giving 100% O2 correlate to 760 mmgh? I know atmospheric pressure is 760 mmgh with an Fi02 of 20.9%. How is 100% of O2 create 760mmgh?
Thank you - I lost my husband recently, and they suspected a shunt. This is the best explanation of how it works. It also explains why continuing to raise the Oxygen levels did not improve his saturation levels. In the end, they had him on 12 liters. They never did find the shunt, and in his case, it was NOT in the heart. They did find cancer and suspected that was the cause of the shunt.
Sorry for your loss. Hope you're feeling better.
Great video with really clear explanations of shunting and describing why they don't respond effectively to O2 therapy!
The videos would be even better without the incredible amount of advertisements populating half of the page throughout the video. I counted 5 different advertisements in 10 minutes. Please take it easy on the ads! Your content speaks well enough for itself!
First video ive watched on this subject that has actually explained it well enough to understand, thanks!
I’m an anesthesiology resident and sometimes I just need a quick refresher on concepts and these videos are perfect. Thank you!
Good to hear! Thank you for the comment
Fantastic lecture as always! Thank you, sir!
Thank you sir you're a legend 🌼
gratitude
That makes so much sense. Thank you!
that was a great brief lecture!! thanks
Really nice explanation of HOW a shunt works, but you never explained how a shunt actually forms in the first place?
i had been waiting he will explain why shunting happened. i guess his knowledge is limited only from the book he read. wasting my 10 minutes.
Shunting occurs when alveoli are not ventilated, as with atelectasis, or when alveoli are filled with fluid, as with pneumonia or pulmonary edema.
could be dependent on patient age. PDA is common in children, especially newborns and premies. left untreated, possible severe complications in adults.
Shunting is rom partial or complete collapse of alveoli- which occurs because of one of two things (or both)
1- pulmonary edema or fluid in alveoli which prevents gas exchange
2- lack of pulmonary surfactant which reduces lung compliance, thus alveoli are floppy
This was helpful.
Thank you ao much.
Lovely explained!
This is a great video, thank you! Could pressure during a free dive cause a pulmonary shunt? And how is a pulmonary shunt different from or related to alveolar collapse?
Thanks for the video! Examples mentioned of R to L shunting are ASD, VSD, PDA but those are L to R shunts??
Thanks, very helpful
Pulmonary artery is blood going back to the heart? Not to the lungs? (3:40)
Great Job
Excellent sir..thanq very much
I got the idea of shutting Thank you sm ,,, but is it normal and where this happens
why is ARDS considered as shunting instead of pulmonary diffusion??
Helpful!
As explained in 1:19 , that 50% shunting happens normally?
Is a "pulmonary shunt" the same thing as an "intrapulmonary shunt"?
Is is possible to have a "right to left intrapulmonary shunt"? I keep reading this phrase "right to left intrapulmonary shunt" and I don't know what it is talking about. Does it really mean venous to arterial? Because "right to left" makes no sense in the intrapulmonary context.
An intrapulmonary shunt is a right to left shunt.
But i can’t understand something isn’t the blood with 85% saturation working its way through all tissues will reach the right side of the heart with a saturation which is less that 70% let’s say 60 % and ...it’s like the saturation will go down after every cycle i don’t understand
Thank you in advance
THANK YOU SO MUCH
👍👍👍👍👍💯
Great lecture. Why is giving 100% O2 correlate to 760 mmgh? I know atmospheric pressure is 760 mmgh with an Fi02 of 20.9%. How is 100% of O2 create 760mmgh?
Some people just wake up one day I start saving lives! You’re the best! Thank you for the amazing video!
Is a shunt a type of VQ mismatch? Or does it cause VQ mismatch ?
Thank you
Great learning material, good job!