Metabolism of alcohol produces so much nadh and as a priority, that it ends up blocking other metabolic pathways that also reduce nad+. Just an extra note on why ethanol is not a good source of atp.
Thank you for sharing!. I still got a question, how is this related to alcohol tolerance or alcohol dependence? Is it because these people have too much alcohol dehydrogenase so they have to drink more to start feeling the minimum effect? if so, how come they can produce more alcohol dehydrogenase if their liver is damaged?. Thanks in advance!
Metabolism of alcohol produces so much nadh and as a priority, that it ends up blocking other metabolic pathways that also reduce nad+.
Just an extra note on why ethanol is not a good source of atp.
Great video, btw
So, the thing i don't understand is what exactly is the alcohol dehydrogenase in this reaction. Is it both serine and hystidine residues?
Thank you for sharing!. I still got a question, how is this related to alcohol tolerance or alcohol dependence? Is it because these people have too much alcohol dehydrogenase so they have to drink more to start feeling the minimum effect? if so, how come they can produce more alcohol dehydrogenase if their liver is damaged?. Thanks in advance!
Hello Alex.
Maybe the liver damaged dosen't mean that the enzyme is not present.
Great video!
Thank you!