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Thank you

Good presentation looks like she got too excited at somepoint

I am chronically ill and started paying attention to my HRV. My doctor does not believe in it, but you can’t tell me there’s not a correlation. I’ve had tremors since my hospitalization for acute pancreatitis 6 months ago. My HRV can drop drastically overnight and wow…I know when to check. My pain in the hospital had them missing the pancreatitis because it wasn’t in the “normal” place. It travelled along the vagus nerve. That’s where I heard about it for the first time. My doctor was the one to mention that.

Weakness and stiffness my whole life...56 years old...2 years ago genetic testing showed autosomal dominate mutation in KBTBD13....finally something to hang my hat on....thanks for this webinar

Excellent presentation, thank you! After doing some searching, I didn't find any literature specifically dealing with neurometabolic tremors misdiagnosed as conversion/functional/psychogenic in origin. This would be an interesting study. That being said, as a patient who has tremor and was once diagnosed with conversion/functioanl/psychogenic disorder as the cause and then obtained an organic diagnosis of Paroxysmal Kinesigenic Dyskinesia with parkinsonian symptoms, started on levodopa which is helping with some of my symptoms, the literature extensively mentions that PKD is often misdiagnosed as psychogenic initially. According to the study below, see figure 1, PKD is mentioned in this discussion of neurometabolic conditions so perhaps this is to be expected. Thank you again for this presentation, speaking as a patient. Gorka Fernández-Eulate J et al. Diagnostic approach in adult-onset neurometabolic diseases. Neurol Neurosurg Psychiatry . 2022 Apr;93(4):413-421. doi: 10.1136/jnnp-2021-328045. Epub 2022 Feb 9. Pubmed PMID: 35140137

LDN and diet has worked for me for over a decade.

"Over the past several years" there was one infectious disease standing out: Covid. It would have been 'nice' if you would have included Neuro Covid

Treatment for cluster headaches can involve various approaches depending on the severity and frequency of the attacks. Here are some common treatments: 1. Acute attack relief: Oxygen therapy and triptan medications (such as sumatriptan) can help alleviate the intense pain during a cluster headache attack. 2. Preventive medications: Your doctor may prescribe medications to prevent cluster headache episodes, including calcium channel blockers, corticosteroids, or antiseizure medications like verapamil, prednisone, or topiramate. 3. Nerve block injections: Injecting a local anesthetic around the nerves associated with cluster headaches can provide temporary relief. 4. Deep brain stimulation: In severe cases, where other treatments have failed, deep brain stimulation may be considered. This involves implanting electrodes in the brain to modulate pain signals. 5. Lifestyle adjustments: Avoiding triggers such as alcohol, strong smells, and high altitude can help reduce the frequency and severity of cluster headaches. It's essential to work closely with a healthcare provider to develop a treatment plan tailored to your specific needs and medical history.

Diagnosing cluster headaches typically involves a thorough medical history review and physical examination by a healthcare professional. They may also order additional tests such as imaging studies (like MRI or CT scans) to rule out other possible causes for your symptoms. Additionally, keeping a headache diary can help track the frequency, duration, and severity of your headaches, which can assist in making an accurate diagnosis. Once diagnosed, your healthcare provider can work with you to develop a personalized treatment plan to manage your symptoms effectively.

Cluster headaches are severe headaches that occur in clusters, often striking suddenly and causing intense pain around one eye. They're known for their cyclical pattern, typically happening at the same time each day or even during specific seasons. These headaches can be incredibly debilitating and may last anywhere from 15 minutes to three hours. Treatment options vary but may include medication to prevent attacks or alleviate symptoms during an episode. If you're experiencing cluster headaches, it's essential to consult with a healthcare professional for proper diagnosis and management.

What if you have positive biopsy but negative antibody?

Shiitake mushrooms

How to Reverse Tardive Dyskinesia?

I was a chronic cluster head plus chronic migraines, tensions and a 4th undiagnosable headache that was a result of one of my brain surgeries ( ONS/DBS). Now left with a lifetime TBI. Then I was the first in the world to treat CCH with stem cell. I’m almost 5 years remission from all 4 headaches. ( video of stem cell procedure. on my page)

Thank You 👍🏻

I appreciated this video very much. At the end of the presentation, when the question about adult patients arose, Dr. Brown described my family perfectly - still ambulatory, met the usual milestones, but always weak, never star athletes, never sure what was wrong. Respiratory compromise, bulbar weakness, and neck weakness are "normal" for them. Everyone says, "What's wrong with you? Can't you stay awake/keep up/ act normal?" And we want to, but we can't. Invitae may finally have pointed us in the right direction, although even Invitae were very unwilling to acknowledge that the VUS might mean something, and they strongly recommended "no further testing." My mother died without any genetic testing whatsoever. I wish I could have given her the answers she was seeking, but hopefully my generation and those that follow will have the help she missed.

ಲಿಂಗರಾಜು ಆರ್

Thanks

VERY WELL DONE !!!! SUCH A COMPREHENSIVE PRESENTATION !!!! EXCELLENT PHYSICIAN !!!!

ruclips.net/video/k8TawjY73S0/видео.html I believe it is supposed to be Oyster Mushrooms and Red Yeast Rice that are of concern.

Is glycerol injection good for pain relif with botox

Are you familiar with Dr. Alan Macdonald study in which he performed autopsies on 10 MS patients. All 10 MS patients had filarial worms in their cerebral spinal fluid. The presentation is available on RUclips, where he talks about multiple sclerosis as a parasitosis. Parasites are the likely source for possibly many diseases . They’ve been here as long a we have. Why do we deworm our pets and ignore ourselves?

Great presentation

From about 57:25, it is stated that it is "impossible" to have low 24-hour urine copper and Wilson's disease. 1. What about when it is Wilson's disease and it's being treated? Then may urine copper be low? 2. This article describes a WD patient who presented WITHOUT "increased copper in urine, Kayser-Fleischer rings [...] and deposits of copper in liver. Introduction of penicillamine treatment [improved the] mental and general health of the patient. Molecular genetic analysis definitely confirmed the diagnosis of Wilson's disease." pubmed.ncbi.nlm.nih.gov/25639007/ 3. In this article, something not discussed in the body paragraphs is shown in Table 1: although the MEAN basal concentration of copper in the urine of the presymptomatic WD patients was 208 mcg/24hr (SD 151), the MEDIAN was just 17. Even in the CNS patients in that table, it takes less than 2 SD to hit 0 mcg/24hr. academic.oup.com/qjmed/article/104/9/775/1591553 4. "Because ceruloplasmin is an acute phase reactant, urine copper is elevated during acute inflammation. During the recovery phase, urine copper is usually below normal, reflecting the expected physiologic response to replace the copper that was depleted during inflammation." healthmatters.io/understand-blood-test-results/copper-24-hour-urine How does this work in a WD patient? 5. This article (gut.bmj.com/content/46/3/415) discusses 30 WD patients, including 2 who shared these traits: age over 40, normal ceruloplasmin, low urine copper, no Kayser-Fleischer rings. A critical letter in response, and the study authors' reply to it (confirming details about the 2 patients), are here (www.ncbi.nlm.nih.gov/pmc/articles/PMC1773089/). The critical letter points out that, "if the patient has been treated with a chelating agent, even briefly, and then the drug stopped, there is often a rebound period when urine copper will drop below 100 [mcg/24hr]". "Sometimes a therapeutic trial is the only way to come to a decision in these patients."

This article presents a chelation regimen that the authors say reduces the risk of adverse effects significantly from that of the usual protocol. It's the same drugs, but with different patterns of titration. www.ncbi.nlm.nih.gov/pmc/articles/PMC8584493/

Hello friends, I am Eduardo Lemos from Portugal and I am 47 years old. I have had HIV since 1995 and in 2010 I suffered a PML that completely paralyzed my left arm and leg and I need a crutch to move around.

The BEST HOSPITAL in the WORLD! In 2009 my mom was hit by a truck, I THANK GOD for this hospital! She lived! I'm very GRATEFUL still for them saving her life!!! 2022

hello sir I am MG patient my problem 10 years

Excellent review

𝚙𝚛𝚘𝚖𝚘𝚜𝚖

Great insights..Wonderful presentation..

Could the natural astaxanthin that is a strong immuna improvment help to fight the virus?

Any thought on Ocrelizumab?

Dronuwa cured me from Herpes virus using his herbal supplement and cleanser #Dronuwa

Dronuwa cured me from Herpes virus using his herbal supplement and cleanser #Dronuwa

You are such a delightful and a dedicated traditional doctor Emuakhe on RUclips.you are truly an inspiration.i cannot thank you much enough for introducing your herbs that completely cure my HIV parmanently...

Is dry skin common in patients?

I am a grateful former patient of Dr. Hedera’s. He treated my neurological presentation of Wilson’s Disease in 1999 and again from 2012-2017ish. He treated me with great respect and even when my tremors were so bad that my arms were flailing around, destroying everything in their path, he gave me hope. His bedside manner is kind and businesslike. I am so fortunate to have been treated by him. If you have Wilson’s Disease and have the chance to be treated by Dr. Hedera, or someone who trained under his supervision, you can rest easy that you will be excellently cared for.

Hi, great video. When will the Remyelination be available for ms patients please.

The antibodies for TN is it different from the antibodies they do for COVID19? I had antibodies for covid and it has not helped my TN at all. Is Baclafin a good medication for TN? I tried the Topamax for my TN and it didn't help and had a side effect from the medication. Also what new medications have came out in 2021for treatment for TN?

You all are good keep me a live in my room as the all that you see you all i n the car Rick in

The hospital

I'm now working on the nutritional aspect of this...

Why don't you doc's in the US use STEM CELLS! For those who can afford it obtain the unbiblical stem cells procedure overseas. I realize that pharma are in the pockets of the docs 😡 Yes and I have RRPMS😡

Anti-Histamines work on allergies namely H1, Anti-acids work on the stomach and gut namely H2. Could it be that MS is caused by a Histamine? Namely H3 and H4. H3 involves the CNS and H4 involves the white blood cells. Which both are involved in MS. There is no medicine or cure for H3 or H4. There is no cure for MS. Coincidence?? Just lately they have devolved a medicine that blocks H3. This is currently being used on a MS trial. Where there is smoke there is fire!! Your thoughts Dr. Feng.

I see MS as being a problem with the B-cells. The B-cells tell the T-cells what to do which causes the damage in MS. The B-cells are the Generals and T-cells are the soldiers of the immune system. It's good to kill the soldiers or make them retreat. But Generals are going to keep telling the soldiers what to do. We need to tell the Generals to retire. lol!!

SECRET MAFIA'S CRUEL CORRUPCIÓN CRIMINAL COCAIN PANDILLAS ABUSERS..TORTURAS 1997 TO 2021.ABUSER SECUESTRO TIRTURAS.MORENOS SECURITY'S COCAIN.PANDILLAS.87 YEAR OLD REEVA,ROBIN AND SCOTT J.SILVERMAN.TRAFFING CHILDREN AND FAMILY'S HUMAN ORGANOS TRAFFING DR.J.ESIN JACKSON HOSPITAL.MASACRES.CANAL 7TV MANIPULADO IN.ISLA BAY HARBOR

Opicinumab is DOA. The fact that it barely benefitted mostly RRMS patients earlier than 8 years disease duration suggests that possibly the body's own reserve capacity for remyelination contributed to these weak results. The fact is, that remyelination, to paraphrase this summary, may only help the "correct" MS population, read RRMS, leaving the progressive population without hope. When axons are injured or dying, remyelination will NOT improve function. Until we focus on neurogenesis, growing new axons, disability will not be reversed by just remyelination. It is just one small piece of the puzzle.

This talk should be played fast; 1.75 at least. the EMG clips have audio here: drive.google.com/file/d/1OTQiiMPqc_CQ5LwoeR8Ooz_d69l3g4km/view?usp=sharing ...and here: drive.google.com/file/d/1OV7Q0Ncjgn_M7kUo3O1zMlPqlPN-KX8P/view?usp=sharing