- Видео 16
- Просмотров 110 087
CVPhysiologist
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Добавлен 26 июл 2012
Humoral Regulation of Blood Pressure
The role of catecholamines, renin-angiotensin-aldosterone system, natriuretic peptides and vasopressin in regulating arterial pressure is described in this video, which is an edited and revised version of a lecture given to medical students in 2021.
Просмотров: 1 015
Видео
Autonomic Control of Blood Pressure
Просмотров 1,8 тыс.2 года назад
This lecture describes the afferent and efferent pathways for sympathetic and parasympathetic innervation of the heart and blood vessel, along the medullary regions that control autonomic function. Neurotransmitters and receptors are also discussed. Finally, baroreflexes originating from arterial and cardiopulmonary receptors are described.
Cardiac Muscle Length-Tension Relationship
Просмотров 2 тыс.2 года назад
This video describes the experimental method of how the length-tension relationship is generated for cardiac muscle. Passive, active and total tension development as a function of muscle length is described.
Biophysical principles of the circulation
Просмотров 1,3 тыс.2 года назад
This lecture describes vascular compliance and volumes determine mean arterial and venous pressures. Additionally, the factors that determine arterial pulse pressure are presented.
Ventricular compliance: normal and abnormal
Просмотров 5 тыс.3 года назад
Ventricular compliance is described under normal conditions, and in heart failure that is related to structural changes (hypertrophy and dilation) in the left ventricle.
Compliance: illustrated definition
Просмотров 2 тыс.3 года назад
The concept of compliance is defined, illustrated and briefly discussed in the context of ventricular compliance
Cardiac Length-Tension Relationship
Просмотров 12 тыс.3 года назад
The relationship between muscle length and tension development in cardiac muscle is described.
Mitral regurgitation pathophysiology
Просмотров 15 тыс.3 года назад
Cardiac pressure and volume changes in the presence of acute and chronic mitral valve regurgitation.
Mitral stenosis pathophysiology
Просмотров 9 тыс.3 года назад
Cardiac pressure and volume changes in the presence of mitral valve stenosis.
Aortic stenosis pathophysiology
Просмотров 10 тыс.3 года назад
Cardiac pressure and volume changes in response to aortic valve stenosis.
Aortic regurgitation pathophysiology
Просмотров 8 тыс.3 года назад
Cardiac pressure and volume changes in the presence of aortic regurgitation.
Coronary Steal Hemodynamics
Просмотров 1,3 тыс.3 года назад
Pressure and flow changes within normal and stenotic coronary arteries
Hemorrhagic Shock
Просмотров 11 тыс.3 года назад
Cardiovascular responses to hemorrhage with a focus on compensatory and decompensatory mechanisms.
Hypotension and Shock
Просмотров 3 тыс.3 года назад
Introductory lecture to hypotension and shock. Definitions, causes, and general characteristics are described
Principles of capillary fluid exchange
Просмотров 1,9 тыс.5 лет назад
Physical forces and membrane characteristics that determine the movement of fluid across capillaries.
Finally i found someone explained this
Why does increasing SVR increase blood pressure? And is this a automatic effect (the heart is not involved) or secondary to the heart working harder? If for example in a electrical circuit you increased resistance that would not change the voltage of the battery it would only change the current. So why does increasing SVR not just decrease CO and MAP be the same. I understand that if this were true it would be devastating for the human body so the real question is what physiologic mechanism increases blood pressure to defend the CO?
Wow ❤
Excellent explanation
thank you
Thank you for this explanation. You made it so much easier to understand!
wonderful explanation!
Wow, it’s pretty clear for me to understand the pathophysiology
Tnx a lot sir,well understood
😲 wow
great video, thorough information. much appreciated!
Great video
Thank you so much! Do you know if there is an optimal L1-L2 range in humans per unit of L0? Would that ratio be constant throughout the myocardium?
its very great, hope to see more videos
CCB effect on arterioles leads to increased flow through the venous system, right? Would that be the reason why some patients get ankle edema as a collateral for such drugs? I mean, increased central venous pressure?
Who came because of cells at work
Wonderfully explained, thank you so much sir!
Great video. Thank you!
You're welcome Taidhgin!
I loveeee your videos! Thank you so much for such useful information. I am waiting when video about hypertension will appear on your channel!
Great explanation
Very knowledgeable, congrats. Anyway I think you may have overestimated the systolic dysfunction on chronic MR, 48% is very low, should have surgery at 60% and at that point I think the forward stroke volume should be somehow conserved.
Thank you....
Thanks a lot
Thank you so much
Thanks for the easy yet highly conceptual explanation.......
im a medical student from turkey thanks to your videos now i have at least the double amount of understanding of cv physiology than i was lectured in school
make it triple
felt like a god after knowing the answer 21:55
great video again
makes me read more books about deep physiology stuff now that i can understand deeper into the subject
dude this is just insane wish you were my prof
dude i dont know what drives you to make these videos but i beg you to keep it up your content is amazing and highly appreciated great work all videos
thank you so much
Paramedic and in undergrad for biology. Great video, great format, and great explanation!
Thank you, that's really helpful!
Hi, plz a presentation on hypertension
Great, thank you verry much!
Thank you very much sir
How is o2 saturation affected by hemorrhagic shock ?
Arterial oxygen saturation is maintained during hemorrhagic shock as long as pulmonary perfusion and ventilation are still matched.
Thank you very much for your answer@@cvphysiologist1977
OXYGENATION SATURATION IS MAINTAINED. THE PROBLEM IS REDUCED CARDIAC OUT WHICH CONSEQUENTLY REDUCES OXYGEN DELIVERY!
Thank you for your valuable lectures Professor 🙏 ..
Hello Professor, if the pressure is influenced by the volume, shouldn't the volume be at the x-axis (manipulated variable), while the pressure be at the y-axis(responding variable)?
Because this lecture focuses on defining compliance, which is the ratio of volume to pressure, I plotted it in this manner (which is nearly always done for lung compliance). In my other lectures where I talk about ventricular pressure and volume relationships, I plot pressure against volume, which is by convention for heart chamber compliance. With those plots, the compliance is the reciprocal of the press-vol relationship slope. Either press or vol can be independent variables. All 3 variables, press-vol-compliance are therefore interdependent.
Thnx a loads
great topic very useful. thank you professor
There are very useful plssss
Add more videos pls
The moment I read on your website that wall stress is the expression helping to appreciate the afterload on individual muscle fibers. I was enlightened. Thank you Professor.
Thank you very much for your explanation! It helped me understand ventricular compliance more :)
Very helpful thank you so much
Thank you very much for providing this lecture. I perform Echocardiography, hence this knowledge is essential.
Thank you very much
Very informative, very concise, very clear. Thank you for making this