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Hector orgullosa muñeco de lo gran q has llegado

Dlb

Rip juice wrld

Apob is not causal but downstream of inflammation

The graphics tell the story better than the transcript :) So looking at the history (which adds context) it does seem the advances in instrumentation are what must proceed better hypothesis. I did bill harris lipo [precursor] profile ... data data everywhere but not an oil droplet of water to drink !!. messin with HMG-COA while interesting in 100 yrs may look like a left turn in the field.

Thanks for this.

Thank you Dr. Rubin for sharing your presentation, and for all of your hard work. Remicade/Inflectra saved me. Finding a medicine that 'puts out the fire' is the first step, and the most crucial step. I think the next step is to rebuild the healthy gut bacteria. I drink a kefir smoothie from raw unpasteurized whole milk each day. I also avoid alcohol and caffeine. And I think these actions complement the medicines. You want to give the medicines the highest chance to work. In addition, I exercise, hike, and eat healthy food. -I was very skinny, just skin and bones, before I received Remicade. Within two months, I regained my weight and strength. I wish your patients, and all UC patients good luck!

from a suffering patient this is a very good presentation.

Excellent talk.

The psychogenic is just one factor. And it was only debunked by old studies.

If all cells can produce cholesterol themselves, what's the objective of liver to secret cholesterol wrapped in lipoprotein to the blood?

Use a better microphone next time. I can barely understand the guy. The speaker was also rude to the person who tried to point it out.

Lipoproteins have infiltrated the mic walls & cables

can i get the refernce ?

Nurse here.. He is spectacularly great with the knowledge of lipids.

Hi. Thanks for the presentation. Could there be discordance between ApoB number and the risk for atherogenic particles? For example, could the ApoB be in mid 90s (say, 94) but the actual risk be very very low because most of the ApoB LDL-carriers will be not atherogenic ? From this lecture I understand that the number of particles is what's matters, but can't the ApoBs be further divided to risky ApoB and non-risky ApoB ? (meaning, the total ApoB number might not be the final number to base treatment...)

please, as all pathologists apparently have failed to find even one particle in mid traverse , will you please show evidence of at least one microscopic slide in human existence ? , instead of only an illustration portraying only an infant's like , simple thin epithelium ... ...the microscopic image of LDL in the proggress of transversing across the thirty or more layers thick epithelium from the inner arterial lining before it arrives at the outside of that very dense & thick lining , accumulating in a cholesterol mass of a swelling plaque thrombosis,, in and around the vicinity of all the smooth muscle and capillaries surrounding the lining . for all the plaques in the world .not every LDL particle could have magically and instantaneously been transported across thirty + bound together layers of epithelium . please help Dr. Subbotin find the first example in medical history of a proof that a magic transport of LDL outward from within the very thick layers exists , before accumulation and burst after has resulted to allow it's passage , prior to build up of the plaque pressure causes the rupture. . cholesterol transport via enterocytes in the gut , does not suffice for an explanation of lipoproteins never showing up, in act of traversing a vastly thick barrier by comparison in arteries .

So glad Dr Dayspring teamed up with Dr Attia. I’ve followed both for many years.

is this lesson 20 years old?

Seems a passionate energetic young faculty. That is what other programs should encourage to have.

Great talk, learned a lot.

Very interesting program

Dr. Osterholm is the man

Why cant we use Ivermectin for now until everyone is vaccinated? It's readily available, cheap and proven to work against the severity of Covid.

This is wonderful in its frankness. The truth hard to face and take is the only way we get through this. Thank you Dr Osterholm!

Thank you Dr Osterholm

To gain the public's trust, authorities must always tell the truth, the whole truth, and nothing but the truth. This has not happened with covid (or with AIDS for that matter), and this is like fuel on the fire for conspiracy theorists.

The lipoproteins are there because of inflamation. The inflammation is there from injury. The endothelial injury is from chronically high glucose and insulin. The lipoproteins are trying to help, but it doesnt work that way and things go wrong. Need to lower glucose and insulin with diet. If you do, your triglycerides will go below 80 as he says is optimum. High LDL and Apo B is a red herring. That is why half or more MI patients have low or normal LDL. Tim Russerts LDL was below 100 at the time of his fatal MI. Risk ratio of high LDL is 1.2 at best. Then there is the argument that this stuff doesnt come in through the endothelial wall, but from the backside via the vaso vasorum. The endothelial wall is really 30 cells thick around the heart, not one cell as typically modeled. Ask a good pathologist who really gets to see all this. Good point that the liver can make LDL directly.

This is where juice died😪 dont go here