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Kan dåke legge inn den siste foredraget på kanalen ble dårlig lyd her. 😊

Når man er dverg og innfører lover for og sende elever som har jukset til høyeste rett. Men har selv jukset..😂 (hun bør bare måtte møte i høyesterett selv nå.) Fy faen for en dobbeltmoralsk klovn hun er..

Ingen av landbruksministrene vil ha god dyrevelferd- kun intensiv avl, oppdrett og slakt for minst mulig kroner og ører. Det går alltid på bekostning av dyrevelferden. Dessuten, det går nesten ikke en uke lengre uten at man leser i nyheter om fjøs som brenner ned og ingen dyr kunne reddes. Hva gjøres på den fronten? Lovpålagt brannvarslingsanlegg, lovpålagt og påbudt sprinkelanlegg?? Grusomme skjebner innen kjøttindustrien på den ene eller andre måten.

Super

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Since science has proven that infected animals shed prions into the landscape that are taken up by the soil and plants and can be passed on to other animals when these plants are consumed, then why is it if CWD is so bad that there are no hay embargos in place to prevent hay from being harvested, specifically in a high prevalence areas that is certainly infected, from being shipped all over the country? I have seen large herds of whitetails feeding in hay fields in high the highest prevalence areas and then be harvested and shipped all over to continue the spread.

Alt for mange fjøsbranner og dyr som drukner i gjødselskjellere...bør ikke det ryddes opp i like mye som dyretragediene i grisehold og husdyrhold generelt. Dette er en av næringene som bør ilegges det absolutt strengeste tilsyn what so ever. Kun da kan det avdekkes de grusomste feil og mangler Uanmeldte sådan

Nefrokalsinose har jeg løst , men kommer ikke gjennom byråkratiet .

wowie

Listen the goverment man made this disease to see if it will spread to humans because it's a man made prion that cause ot not a fungus

Reinsdyr hjerneprøve

Does anybody know how CWD GOT TO NORWAY in the first place??? Did it come from the small population of (long-ago) imported whitetails? CWD is hellishly contagious to other cervids and the long-term implications are horrendous. Here in North America it continues to spread inexorably. Willi B. in Canada.

nothing to do with the disease, but they all look like they need a glass water, some chapstick and a good moisturizer

Wow we even have this gene in our body it just hasn't mutated yet.

Rudolphs red nose was flesh from CWD.Change my mind

No there's sick animals cuz us as humans don't know how to quit fucking with things that we shouldn't be fucking with wouldnt doubt if the humans didnt create the nasty-ass brain worm

Deer need a serious buff

So, basically the Flare virus but in animals

Fun fact The fungus that's in the last of us Is a real parasite that just hasent quite mutated yet :)

na.to/sexy14378date

Porque russo, saiu abrindo o bicho que orra em. Doidera

Oh jeez i vomit when i saw that brain yucky jesus its gross

It's cuased by a bacterial infection

*Yall prepare your AC130's and AK'S to blow that shit up*

Haha the brain looks like an peanut

I wonder what peta is thinking about this

0:49

Brain of a reindeer 😨 Santa's gonna be pissed

Rudolph the red whoa reindeer

Whoever is the one that starts it first has big gay

if someone eat the meat they will get the cwd virus

So their like zombie reindeer

Subject: PRION 2017 CONFERENCE ABSTRACT First evidence of intracranial and peroral transmission of Chronic Wasting Disease (CWD) into Cynomolgus macaques: a work in progress First evidence of intracranial and peroral transmission of Chronic Wasting Disease (CWD) into Cynomolgus macaques: a work in progress Stefanie Czub1, Walter Schulz-Schaeffer2, Christiane Stahl-Hennig3, Michael Beekes4, Hermann Schaetzl5 and Dirk Motzkus6 1 University of Calgary Faculty of Veterinary Medicine/Canadian Food Inspection Agency; 2Universitatsklinikum des Saarlandes und Medizinische Fakultat der Universitat des Saarlandes; 3 Deutsches Primaten Zentrum/Goettingen; 4 Robert-Koch-Institut Berlin; 5 University of Calgary Faculty of Veterinary Medicine; 6 presently: Boehringer Ingelheim Veterinary Research Center; previously: Deutsches Primaten Zentrum/Goettingen This is a progress report of a project which started in 2009. 21 cynomolgus macaques were challenged with characterized CWD material from white-tailed deer (WTD) or elk by intracerebral (ic), oral, and skin exposure routes. Additional blood transfusion experiments are supposed to assess the CWD contamination risk of human blood product. Challenge materials originated from symptomatic cervids for ic, skin scarification and partially per oral routes (WTD brain). Challenge material for feeding of muscle derived from preclinical WTD and from preclinical macaques for blood transfusion experiments. We have confirmed that the CWD challenge material contained at least two different CWD agents (brain material) as well as CWD prions in muscle-associated nerves. Here we present first data on a group of animals either challenged ic with steel wires or per orally and sacrificed with incubation times ranging from 4.5 to 6.9 years at postmortem. Three animals displayed signs of mild clinical disease, including anxiety, apathy, ataxia and/or tremor. In four animals wasting was observed, two of those had confirmed diabetes. All animals have variable signs of prion neuropathology in spinal cords and brains and by supersensitive IHC, reaction was detected in spinal cord segments of all animals. Protein misfolding cyclic amplification (PMCA), real-time quaking-induced conversion (RT-QuiC) and PET-blot assays to further substantiate these findings are on the way, as well as bioassays in bank voles and transgenic mice. At present, a total of 10 animals are sacrificed and read-outs are ongoing. Preclinical incubation of the remaining macaques covers a range from 6.4 to 7.10 years. Based on the species barrier and an incubation time of > 5 years for BSE in macaques and about 10 years for scrapie in macaques, we expected an onset of clinical disease beyond 6 years post inoculation. PRION 2017 DECIPHERING NEURODEGENERATIVE DISORDERS TUESDAY, JUNE 13, 2017 PRION 2017 CONFERENCE ABSTRACT First evidence of intracranial and peroral transmission of Chronic Wasting Disease (CWD) into Cynomolgus macaques: a work in progress chronic-wasting-disease.blogspot.com/2017/06/prion-2017-conference-abstract-first.html FRIDAY, JUNE 16, 2017 P55 Susceptibility of human prion protein to in vitro conversion by chronic wasting disease prions chronic-wasting-disease.blogspot.com/2017/06/p55-susceptibility-of-human-prion.html SATURDAY, JUNE 10, 2017 Chronic Wasting Disease CWD TSE Prion to Humans, who makes that final call, when, or, has it already happened? chronic-wasting-disease.blogspot.com/2017/06/chronic-wasting-disease-cwd-tse-prion_10.html MONDAY, JUNE 12, 2017 Rethinking Major grain organizations opposition to CFIA's control zone approach to Chronic Wasting CWD TSE Prion Mad Deer Type Disease 2017? chronic-wasting-disease.blogspot.com/2017/06/rethinking-major-grain-organizations.html TUESDAY, JUNE 13, 2017 PRION 2017 CONFERENCE ABSTRACT Chronic Wasting Disease in European moose is associated with PrPSc features different from North American CWD chronic-wasting-disease.blogspot.com/2017/06/prion-2017-conference-abstract-chronic.html TUESDAY, JUNE 06, 2017 CHRONIC WASTING DISEASE CWD TSE PRION ZOONOSIS ZOONOTIC INSIDIOUS AND DIRE CONSEQUENCES AHEAD chronic-wasting-disease.blogspot.com/2017/06/chronic-wasting-disease-cwd-tse-prion.html